Elizabeth M. Fozo
- Molecular Medicine top 2%
- Antibiotic Resistance in Bacteria 5
- Periodontics top 2%
- Endocrinology top 2%
- Vibrio bacteria research studies 4
- Genetics top 5%
- Bacterial Genetics and Biotechnology 14
- Ecology top 5%
- Bacteriophages and microbial interactions 15
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- RNA and protein synthesis mechanisms 9
- Genomics and Phylogenetic Studies 4
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- Antimicrobial Resistance in Staphylococcus 7
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- Probiotics and Fermented Foods 3
- Co-authors
- Robert G. QuiveyGisela StorzMatthew R. HemmJessica K. KajfaszSvetlana A. ShabalinaKira S. MakarovaNatalya YutinEugene V. Koonin
- Partner nations
- United StatesUnited KingdomGermany
In The Last Decade
Elizabeth M. Fozo
32 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 98
- Molecular Medicine 213
- Periodontics 184
- Endocrinology 198
- Genetics 612
- Ecology 527
Countries citing papers authored by Elizabeth M. Fozo
This map shows the geographic impact of Elizabeth M. Fozo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elizabeth M. Fozo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elizabeth M. Fozo more than expected).
Fields of papers citing papers by Elizabeth M. Fozo
This network shows the impact of papers produced by Elizabeth M. Fozo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elizabeth M. Fozo. The network helps show where Elizabeth M. Fozo may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Elizabeth M. Fozo, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 1 | |
| 2 | 2024 | 8 | |
| 3 | 2023 | 2 | |
| 4 | 2023 | 6 | |
| 5 | 2022 | 5 | |
| 6 | 2021 | 18 | |
| 7 | 2021 | 6 | |
| 8 | 2019 | 23 | |
| 9 | 2019 | 21 | |
| 10 | 2017 | 5 | |
| 11 | 2016 | 34 | |
| 12 | 2016 | 22 | |
| 13 | 2013 | 31 | |
| 14 | 2012 | 41 | |
| 15 | 2010 | 86 | |
| 16 | 2010 | 212 | |
| 17 | 2008 | 134 | |
| 18 | 2004 | 171 | |
| 19 | 2004 | 100 | |
| 20 | 2004 | 101 |
About Elizabeth M. Fozo
Elizabeth M. Fozo is a scholar working on Molecular Medicine, Endocrinology and Ecology, having authored 33 papers that have together received 1.7k indexed citations. Recurring topics across this work include Bacteriophages and microbial interactions (15 papers), Bacterial Genetics and Biotechnology (14 papers), RNA and protein synthesis mechanisms (9 papers), Antimicrobial Resistance in Staphylococcus (7 papers), Antibiotic Resistance in Bacteria (5 papers), Vibrio bacteria research studies (4 papers), Genomics and Phylogenetic Studies (4 papers) and Probiotics and Fermented Foods (3 papers). The work is most often cited by research in Molecular Medicine (213 citations), Periodontics (184 citations) and Endocrinology (198 citations). Elizabeth M. Fozo has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Robert G. Quivey, Gisela Storz, Matthew R. Hemm, Jessica K. Kajfasz, Svetlana A. Shabalina, Kira S. Makarova, Natalya Yutin, Eugene V. Koonin, Jason A. Opdyke and Virginia L. Clark. Their work appears in journals such as Nucleic Acids Research, PLoS ONE and Journal of Molecular Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.