Elisa Ave

606 total citations
8 papers, 436 citations indexed

About

Elisa Ave is a scholar working on Genetics, Immunology and Molecular Biology. According to data from OpenAlex, Elisa Ave has authored 8 papers receiving a total of 436 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Genetics, 4 papers in Immunology and 3 papers in Molecular Biology. Recurrent topics in Elisa Ave's work include Chronic Lymphocytic Leukemia Research (6 papers), Lymphoma Diagnosis and Treatment (3 papers) and Immunodeficiency and Autoimmune Disorders (2 papers). Elisa Ave is often cited by papers focused on Chronic Lymphocytic Leukemia Research (6 papers), Lymphoma Diagnosis and Treatment (3 papers) and Immunodeficiency and Autoimmune Disorders (2 papers). Elisa Ave collaborates with scholars based in Italy. Elisa Ave's co-authors include Gianpietro Semenzato, Anna Cabrelle, Cristina Gattazzo, Antonella Teramo, F. Calabrese, Gian Paolo Fadini, Sara Teolato, Marianna Gnoato, Carlo Agostini and Renato Zambello and has published in prestigious journals such as Blood, PLoS ONE and Clinical Cancer Research.

In The Last Decade

Elisa Ave

8 papers receiving 435 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Elisa Ave Italy	7	167	132	132	131	122	8	436
François Plassa France	10	64 0.4×	253 1.9×	35 0.3×	127 1.0×	178 1.5×	18	618
Michael Frost United States	8	86 0.5×	32 0.2×	78 0.6×	215 1.6×	35 0.3×	11	400
Catia Lo Pardo Italy	14	39 0.2×	45 0.3×	78 0.6×	266 2.0×	85 0.7×	18	507
H. Eugene Liu Taiwan	10	32 0.2×	69 0.5×	48 0.4×	260 2.0×	27 0.2×	16	436
Chiara Angiolilli Netherlands	11	24 0.1×	49 0.4×	172 1.3×	252 1.9×	25 0.2×	16	512
Frank El Ouriaghli United States	9	33 0.2×	34 0.3×	153 1.2×	133 1.0×	38 0.3×	10	390
Norihiro Awaya Japan	12	41 0.2×	27 0.2×	90 0.7×	203 1.5×	122 1.0×	26	474
James C.M. Wang Canada	4	48 0.3×	34 0.3×	262 2.0×	99 0.8×	27 0.2×	6	458
Jaroslav Žák United States	10	83 0.5×	112 0.8×	174 1.3×	123 0.9×	13 0.1×	17	446

Countries citing papers authored by Elisa Ave

Since Specialization

Citations

This map shows the geographic impact of Elisa Ave's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elisa Ave with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elisa Ave more than expected).

Fields of papers citing papers by Elisa Ave

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Elisa Ave. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elisa Ave. The network helps show where Elisa Ave may publish in the future.

Co-authorship network of co-authors of Elisa Ave

This figure shows the co-authorship network connecting the top 25 collaborators of Elisa Ave. A scholar is included among the top collaborators of Elisa Ave based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elisa Ave. Elisa Ave is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

Trimarco, Valentina, Elisa Ave, Monica Facco, et al.. (2015). Cross-talk between chronic lymphocytic leukemia (CLL) tumor B cells and mesenchymal stromal cells (MSCs): implications for neoplastic cell survival. Oncotarget. 6(39). 42130–42149. 38 indexed citations

Frezzato, Federica, Valentina Trimarco, Veronica Martini, et al.. (2014). Leukaemic cells from chronic lymphocytic leukaemia patients undergo apoptosis following microtubule depolymerization and Lyn inhibition by nocodazole. British Journal of Haematology. 165(5). 659–672. 21 indexed citations

Teramo, Antonella, Cristina Gattazzo, Giulia Tasca, et al.. (2013). Intrinsic and extrinsic mechanisms contribute to maintain the JAK/STAT pathway aberrantly activated in T-type large granular lymphocyte leukemia. Blood. 121(19). 3843–3854. 78 indexed citations

Visco, Carlo, Francesca Moretta, Erika Falisi, et al.. (2013). Double productive immunoglobulin sequence rearrangements in patients with chronic lymphocytic leukemia. American Journal of Hematology. 88(4). 277–282. 10 indexed citations

Manni, Sabrina, Laura Quotti Tubi, Anna Colpo, et al.. (2012). Protein Kinase CK2 Protects Multiple Myeloma Cells from ER Stress–Induced Apoptosis and from the Cytotoxic Effect of HSP90 Inhibition through Regulation of the Unfolded Protein Response. Clinical Cancer Research. 18(7). 1888–1900. 66 indexed citations

Frezzato, Federica, Cristina Gattazzo, Veronica Martini, et al.. (2012). HS1, a Lyn Kinase Substrate, Is Abnormally Expressed in B-Chronic Lymphocytic Leukemia and Correlates with Response to Fludarabine-Based Regimen. PLoS ONE. 7(6). e39902–e39902. 28 indexed citations

Gattazzo, Cristina, Veronica Martini, Federica Frezzato, et al.. (2012). Cortactin Expression Is Tightly Connected to B-Cell Chronic Lymphocytic Leukemia Aggressiveness. Blood. 120(21). 4561–4561. 1 indexed citations

Cabrelle, Anna, Antonella Teramo, Marianna Gnoato, et al.. (2010). Sarcoidosis is a Th1/Th17 multisystem disorder. Thorax. 66(2). 144–150. 194 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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