Elda Cannavò

2.8k total citations
30 papers, 2.1k citations indexed

About

Elda Cannavò is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Elda Cannavò has authored 30 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 8 papers in Oncology and 8 papers in Cancer Research. Recurrent topics in Elda Cannavò's work include DNA Repair Mechanisms (20 papers), Genetic factors in colorectal cancer (7 papers) and CRISPR and Genetic Engineering (7 papers). Elda Cannavò is often cited by papers focused on DNA Repair Mechanisms (20 papers), Genetic factors in colorectal cancer (7 papers) and CRISPR and Genetic Engineering (7 papers). Elda Cannavò collaborates with scholars based in Switzerland, United States and Italy. Elda Cannavò's co-authors include Petr Ćejka, Stephen C. Kowalczykowski, Roopesh Anand, Lepakshi Ranjha, Josef Jiricny, Giordano Reginato, Subhash Pokharel, Judith L. Campbell, Piotr Polaczek and Giancarlo Marra and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Elda Cannavò

29 papers receiving 2.1k citations

Peers

Elda Cannavò

ONCOL

PFM

Irina I. Dianova United Kingdom

Federico Lazzaro Italy

Eun Yong Shim United States

Anneke van Hoffen Netherlands

Agnès Tissier France

Hiroyuki Sasanuma Japan

Jean-Marie Buerstedde Japan

E C Friedberg United States

Joshua C. Saldivar United States

Ayako Ui Japan

Irina I. Dianova United Kingdom

Elda Cannavò

1.7k ×0.9

653 ×1.4

ONCOL

375 ×1.0

75 ×0.4

PFM

133 ×0.7

Citations per year, relative to Elda Cannavò Elda Cannavò (= 1×) peers Irina I. Dianova

Countries citing papers authored by Elda Cannavò

Since Specialization

Citations

This map shows the geographic impact of Elda Cannavò's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elda Cannavò with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elda Cannavò more than expected).

Fields of papers citing papers by Elda Cannavò

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Elda Cannavò. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elda Cannavò. The network helps show where Elda Cannavò may publish in the future.

Co-authorship network of co-authors of Elda Cannavò

This figure shows the co-authorship network connecting the top 25 collaborators of Elda Cannavò. A scholar is included among the top collaborators of Elda Cannavò based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elda Cannavò. Elda Cannavò is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Rinaldi, Andrea C., Giordano Reginato, Ananya Acharya, et al.. (2025). Mechanism of trinucleotide repeat expansion by MutSβ-MutLγ and contraction by FAN1. Nature Communications. 16(1). 9445–9445. 1 indexed citations

Allison, Rachal M., et al.. (2024). Exploring the removal of Spo11 and topoisomerases from DNA breaks in S. cerevisiae by human Tyrosyl DNA Phosphodiesterase 2. DNA repair. 142. 103757–103757.

Reginato, Giordano, Yanbo Wang, Jingzhou Hao, et al.. (2024). HLTF disrupts Cas9-DNA post-cleavage complexes to allow DNA break processing. Nature Communications. 15(1). 5789–5789. 17 indexed citations

Ceppi, Ilaria, Elda Cannavò, Roshan Singh Thakur, et al.. (2023). PLK1 regulates CtIP and DNA2 interplay in long-range DNA end resection. Genes & Development. 37(3-4). 119–135. 13 indexed citations

Reginato, Giordano, et al.. (2023). Xrs2/NBS1 promote end-bridging activity of the MRE11-RAD50 complex. Biochemical and Biophysical Research Communications. 695. 149464–149464. 5 indexed citations

Ceppi, Ilaria, Aurore Sanchez, Elda Cannavò, et al.. (2022). WRN helicase and mismatch repair complexes independently and synergistically disrupt cruciform DNA structures. The EMBO Journal. 42(3). e111998–e111998. 24 indexed citations

Gobbini, Elisa, Corinne Cassani, Renata Tisi, et al.. (2021). Sae2 and Rif2 regulate MRX endonuclease activity at DNA double-strand breaks in opposite manners. Cell Reports. 34(13). 108906–108906. 18 indexed citations

Cannavò, Elda, Charly Chahwan, Christopher D. Smith, et al.. (2021). Inhibition of MRN activity by a telomere protein motif. Nature Communications. 12(1). 3856–3856. 21 indexed citations

Cannavò, Elda, Sara N. Andres, Vera M. Kissling, et al.. (2018). Regulatory control of DNA end resection by Sae2 phosphorylation. Nature Communications. 9(1). 4016–4016. 60 indexed citations

10.

Reginato, Giordano, Elda Cannavò, & Petr Ćejka. (2017). Physiological protein blocks direct the Mre11–Rad50–Xrs2 and Sae2 nuclease complex to initiate DNA end resection. Genes & Development. 31(23-24). 2325–2330. 105 indexed citations

11.

Oh, Julyun, Amr Al-Zain, Elda Cannavò, Petr Ćejka, & Lorraine S. Symington. (2016). Xrs2 Dependent and Independent Functions of the Mre11-Rad50 Complex. Molecular Cell. 64(2). 405–415. 60 indexed citations

12.

Anand, Roopesh, Lepakshi Ranjha, Elda Cannavò, & Petr Ćejka. (2016). Phosphorylated CtIP Functions as a Co-factor of the MRE11-RAD50-NBS1 Endonuclease in DNA End Resection. Molecular Cell. 64(5). 940–950. 248 indexed citations

13.

Deere, Jesse D., Robert C. Kauffman, Elda Cannavò, et al.. (2014). Analysis of Multiply Spliced Transcripts in Lymphoid Tissue Reservoirs of Rhesus Macaques Infected with RT-SHIV during HAART. PLoS ONE. 9(2). e87914–e87914. 15 indexed citations

14.

Cannavò, Elda & Petr Ćejka. (2014). Sae2 promotes dsDNA endonuclease activity within Mre11–Rad50–Xrs2 to resect DNA breaks. Nature. 514(7520). 122–125. 338 indexed citations

15.

Ćejka, Petr, Elda Cannavò, Piotr Polaczek, et al.. (2010). DNA end resection by Dna2–Sgs1–RPA and its stimulation by Top3–Rmi1 and Mre11–Rad50–Xrs2. Nature. 467(7311). 112–116. 366 indexed citations

16.

Deere, Jesse D., Joanne Higgins, Elda Cannavò, et al.. (2010). Viral Decay Kinetics in the Highly Active Antiretroviral Therapy-Treated Rhesus Macaque Model of AIDS. PLoS ONE. 5(7). e11640–e11640. 12 indexed citations

17.

Kratz, Katja, Ataman Sendoel, Ralf Eberhard, et al.. (2010). Deficiency of FANCD2-Associated Nuclease KIAA1018/FAN1 Sensitizes Cells to Interstrand Crosslinking Agents. Cell. 142(1). 77–88. 211 indexed citations

18.

Cannavò, Elda, Bertran Gerrits, Giancarlo Marra, Ralph Schlapbach, & Josef Jiricny. (2006). Characterization of the Interactome of the Human MutL Homologues MLH1, PMS1, and PMS2. Journal of Biological Chemistry. 282(5). 2976–2986. 115 indexed citations

19.

Fuggetta, Maria Pia, Stefania D’Atri, Giulia Lanzilli, et al.. (2004). In vitro antitumour activity of resveratrol in human melanoma cells sensitive or resistant to temozolomide. Melanoma Research. 14(3). 189–196. 38 indexed citations

20.

Franzese, Ornella, et al.. (2001). Effect of Saquinavir on proliferation and telomerase activity of human peripheral blood mononuclear cells. Life Sciences. 69(13). 1509–1520. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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