E.‐J. Speckmann

2.2k total citations
89 papers, 1.8k citations indexed

About

E.‐J. Speckmann is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Psychiatry and Mental health. According to data from OpenAlex, E.‐J. Speckmann has authored 89 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 67 papers in Cellular and Molecular Neuroscience, 35 papers in Molecular Biology and 23 papers in Psychiatry and Mental health. Recurrent topics in E.‐J. Speckmann's work include Neuroscience and Neuropharmacology Research (50 papers), Ion channel regulation and function (29 papers) and Epilepsy research and treatment (22 papers). E.‐J. Speckmann is often cited by papers focused on Neuroscience and Neuropharmacology Research (50 papers), Ion channel regulation and function (29 papers) and Epilepsy research and treatment (22 papers). E.‐J. Speckmann collaborates with scholars based in Germany, United States and Iran. E.‐J. Speckmann's co-authors include U. Altrup, D. Bingmann, J. Walden, Otto W. Witte, Christian E. Elger, J. Walden, H. Straub, H. Caspers, Rüdiger Köhling and Ali Gorji and has published in prestigious journals such as Brain, Brain Research and Neuroscience.

In The Last Decade

E.‐J. Speckmann

88 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
E.‐J. Speckmann Germany	26	1.2k	703	543	485	206	89	1.8k
E.‐J. Speckmann Germany	20	844 0.7×	547 0.8×	439 0.8×	295 0.6×	105 0.5×	48	1.3k
Erwin‐Josef Speckmann Germany	23	906 0.8×	604 0.9×	667 1.2×	357 0.7×	202 1.0×	79	1.7k
Robert F. Ackermann United States	27	1.6k 1.3×	657 0.9×	597 1.1×	479 1.0×	191 0.9×	54	2.4k
Adam C. Errington United Kingdom	21	1.2k 1.0×	691 1.0×	480 0.9×	594 1.2×	223 1.1×	37	1.8k
James O. McNamara United States	21	1.3k 1.1×	950 1.4×	584 1.1×	297 0.6×	255 1.2×	32	2.1k
B.M. Cohen United States	29	691 0.6×	662 0.9×	966 1.8×	601 1.2×	83 0.4×	48	2.5k
W. A. Wilson United States	27	2.0k 1.7×	913 1.3×	317 0.6×	858 1.8×	199 1.0×	46	2.4k
C. Marescaux France	25	1.6k 1.4×	659 0.9×	1.1k 1.9×	624 1.3×	482 2.3×	58	2.3k
Martin Vreugdenhil United Kingdom	29	1.7k 1.4×	978 1.4×	447 0.8×	891 1.8×	155 0.8×	53	2.5k
Joanna S. Fowler United States	15	1.5k 1.3×	466 0.7×	376 0.7×	552 1.1×	70 0.3×	18	2.2k

Countries citing papers authored by E.‐J. Speckmann

Since Specialization

Citations

This map shows the geographic impact of E.‐J. Speckmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E.‐J. Speckmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E.‐J. Speckmann more than expected).

Fields of papers citing papers by E.‐J. Speckmann

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E.‐J. Speckmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E.‐J. Speckmann. The network helps show where E.‐J. Speckmann may publish in the future.

Co-authorship network of co-authors of E.‐J. Speckmann

This figure shows the co-authorship network connecting the top 25 collaborators of E.‐J. Speckmann. A scholar is included among the top collaborators of E.‐J. Speckmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E.‐J. Speckmann. E.‐J. Speckmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ghadiri, Maryam Khaleghi, et al.. (2015). Altered inhibition in the hippocampal neural networks after spreading depression. Neuroscience. 304. 190–197. 9 indexed citations

Haghir, Hossein, Stjepana Kovac, E.‐J. Speckmann, Karl Zilles, & Ali Gorji. (2009). Patterns of neurotransmitter receptor distributions following cortical spreading depression. Neuroscience. 163(4). 1340–1352. 43 indexed citations

Wölfer, Johannes, et al.. (2008). Influences of pCO2 and bicarbonate concentration on bioelectric phenomena in ischemic hippocampal ex vivo tissue. Neuroscience. 158(2). 617–622. 1 indexed citations

Köhling, Rüdiger, et al.. (2002). Spatio-temporal patterns of neuronal activity: analysis of optical imaging data using geometric shape matching. Journal of Neuroscience Methods. 114(1). 17–23. 6 indexed citations

Bingmann, D., Martin Wiemann, E.‐J. Speckmann, et al.. (2000). Cutting of living hippocampal slices by a highly pressurised water jet (macromingotome). Journal of Neuroscience Methods. 102(1). 1–9. 4 indexed citations

Köhling, Rüdiger, M. Qü, Karl Zilles, & E.‐J. Speckmann. (1999). Current-source-density profiles associated with sharp waves in human epileptic neocortical tissue. Neuroscience. 94(4). 1039–1050. 33 indexed citations

Hülsmann, Swen, H. Wassmann, Dag Moskopp, et al.. (1998). Neuroprotection of mild hypothermia: differential effects. Brain Research. 786(1-2). 267–269. 11 indexed citations

Speckmann, E.‐J.. (1997). Generation of Field Potentials in the Brain. The Journal of Clinical Pharmacology. 37(S1). 8S–10S. 4 indexed citations

Köhling, Rüdiger, Swen Hülsmann, Sampsa Vanhatalo, et al.. (1996). Anoxic terminal negative DC-shift in human neocortical slices in vitro. Brain Research. 741(1-2). 174–179. 16 indexed citations

10.

Altrup, U., et al.. (1994). Attenuation of a voltage-dependent sodium current by GABA (identified neurons, buccal ganglia, Helix pomatia. Brain Research. 663(1). 131–139. 4 indexed citations

11.

Köhling, Rüdiger, et al.. (1994). Spontaneous and stimulustriggered epileptic discharges: Delayed antiepileptic effect with triggering. Experimental Brain Research. 100(3). 376–384. 11 indexed citations

12.

Lücke, A. & E.‐J. Speckmann. (1993). Influence of the organic calcium antagonist verapamil on (NMDA) induced cortical field potentials (neocortical slice, guinea pig). Neuroscience Letters. 161(2). 179–182. 8 indexed citations

13.

Altrup, U., et al.. (1992). Effects of Valproate in a Model Nervous System (Buccal Ganglia of Helix Pomatia): II. Epileptogenic Actions. Epilepsia. 33(4). 753–759. 10 indexed citations

14.

Straub, H., et al.. (1992). Low magnesium-induced epileptiform discharges in guinea pig hippocampal slices: depression by the organic calcium antagonist verapamil. Brain Research. 577(1). 29–35. 36 indexed citations

15.

Bingmann, D., et al.. (1991). Caffeine-induced epileptic discharges in CA3 neurons of hippocampal slices of the guinea pig. Neuroscience Letters. 129(1). 51–54. 27 indexed citations

16.

Altrup, U., A. Lehmenkühler, & E.‐J. Speckmann. (1991). Effects of the hypnotic drug etomidate in a model nervous system (buccal ganglia, helix pomatia). Comparative Biochemistry and Physiology Part C Comparative Pharmacology. 99(3). 579–587. 19 indexed citations

17.

Walden, J., E.‐J. Speckmann, D. Bingmann, & H. Straub. (1990). Augmentation ofN-methyl-d-aspartate induced depolarizations by GABA in neocortical and archicortical neurons. Brain Research. 510(1). 127–129. 9 indexed citations

18.

Walden, J., E.‐J. Speckmann, & D. Bingmann. (1989). Augmentation of glutamate responses by GABA in the rat's motorcortex in vivo. Neuroscience Letters. 101(2). 209–213. 13 indexed citations

19.

Altrup, U. & E.‐J. Speckmann. (1988). Epileptic discharges induced by pentylenetetrazol: changes of shape of dendrites. Brain Research. 456(2). 401–405. 23 indexed citations

20.

Walden, J. & E.‐J. Speckmann. (1988). Suppression of recurrent generalized tonic-clonic seizure discharges by intraventricular perfusion of a calcium antagonist. Electroencephalography and Clinical Neurophysiology. 69(4). 353–362. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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