Edwin Verlind

974 total citations
28 papers, 652 citations indexed

About

Edwin Verlind is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Edwin Verlind has authored 28 papers receiving a total of 652 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 5 papers in Oncology and 5 papers in Genetics. Recurrent topics in Edwin Verlind's work include Glycogen Storage Diseases and Myoclonus (3 papers), Genetic factors in colorectal cancer (3 papers) and Genomic variations and chromosomal abnormalities (3 papers). Edwin Verlind is often cited by papers focused on Glycogen Storage Diseases and Myoclonus (3 papers), Genetic factors in colorectal cancer (3 papers) and Genomic variations and chromosomal abnormalities (3 papers). Edwin Verlind collaborates with scholars based in Netherlands, United States and Germany. Edwin Verlind's co-authors include Charles H.C.M. Buys, Robert M.W. Hofstra, Hans Scheffer, Tineke van der Sluis, Harry Hollema, Ying Wu, Rob G.J. Mensink, Jan H. Kleibeuker, Rolf H. Sijmons and Maran J.W. Berends and has published in prestigious journals such as The Lancet, Nature Genetics and Gastroenterology.

In The Last Decade

Edwin Verlind

28 papers receiving 635 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Edwin Verlind Netherlands 15 293 268 196 135 115 28 652
Stephen C. Jefcoat United States 11 484 1.7× 176 0.7× 177 0.9× 181 1.3× 78 0.7× 11 760
Maurizio Trubia Italy 16 303 1.0× 104 0.4× 91 0.5× 80 0.6× 107 0.9× 18 615
Elisabeth Steichen‐Gersdorf Austria 15 319 1.1× 75 0.3× 86 0.4× 176 1.3× 64 0.6× 27 793
Margaret Huskey United States 12 439 1.5× 115 0.4× 136 0.7× 152 1.1× 25 0.2× 17 685
Sonia Pagliardini Italy 17 610 2.1× 109 0.4× 312 1.6× 79 0.6× 133 1.2× 25 988
Sheng‐Ben Liang Japan 14 421 1.4× 92 0.3× 185 0.9× 62 0.5× 78 0.7× 21 691
Suàrez Hg France 5 245 0.8× 102 0.4× 267 1.4× 115 0.9× 48 0.4× 8 602
Alessia Bottos Switzerland 12 238 0.8× 142 0.5× 284 1.4× 45 0.3× 89 0.8× 37 602
Niklas Ekman Finland 11 597 2.0× 141 0.5× 285 1.5× 52 0.4× 174 1.5× 14 888
Eunice Lee United States 7 369 1.3× 67 0.3× 106 0.5× 79 0.6× 50 0.4× 10 858

Countries citing papers authored by Edwin Verlind

Since Specialization
Citations

This map shows the geographic impact of Edwin Verlind's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edwin Verlind with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edwin Verlind more than expected).

Fields of papers citing papers by Edwin Verlind

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edwin Verlind. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edwin Verlind. The network helps show where Edwin Verlind may publish in the future.

Co-authorship network of co-authors of Edwin Verlind

This figure shows the co-authorship network connecting the top 25 collaborators of Edwin Verlind. A scholar is included among the top collaborators of Edwin Verlind based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edwin Verlind. Edwin Verlind is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Baranova, Ancha, Chantal Lummen, Maaike W.G. Looman, et al.. (2003). RFP2, c13ORF1, and FAM10A4 are the most likely tumor suppressor gene candidates for B-cell chronic lymphocytic leukemia. Cancer Genetics and Cytogenetics. 146(1). 48–57. 21 indexed citations
2.
Martı́nez, Francisco, et al.. (2003). X-linked hydrocephalus: another two families with an L1 mutation.. PubMed. 14(1). 57–65. 4 indexed citations
3.
Hout, Annemarie H. van der, Edwin Verlind, Frits A. Beemer, et al.. (2002). Occurrence of deletion of a COL2A1 allele as the mutation in Stickler syndrome shows that a collagen type II dosage effect underlies this syndrome. Human Mutation. 20(3). 236–236. 14 indexed citations
4.
Verlind, Edwin, et al.. (2001). A new family with a mutation in exon 2 of COL2A1 and Stickler syndrome without systemic manifestations.. Data Archiving and Networked Services (DANS). 1 indexed citations
5.
Wu, Ying, Maran J.W. Berends, Rolf H. Sijmons, et al.. (2001). A role for MLH3 in hereditary nonpolyposis colorectal cancer. Nature Genetics. 29(2). 137–138. 121 indexed citations
6.
Wu, Ying, Rob G.J. Mensink, Edwin Verlind, et al.. (2001). Germline mutations of EXO1 gene in patients with hereditary nonpolyposis colorectal cancer (HNPCC) and atypical HNPCC forms. Gastroenterology. 120(7). 1580–1587. 120 indexed citations
7.
Wu, Yige, et al.. (2000). Germline hMLH3 mutations in patients with suspected HNPCC.. Data Archiving and Networked Services (DANS). 3 indexed citations
8.
Vlies, Pieter van der, Edwin Verlind, Charles H.C.M. Buys, et al.. (2000). Exempting Homologous Pseudogene Sequences from Polymerase Chain Reaction Amplification Allows Genomic Keratin 14 Hotspot Mutation Analysis. Journal of Investigative Dermatology. 114(4). 616–619. 32 indexed citations
9.
Rake, Jan Peter, Gepke Visser, Edwin Verlind, et al.. (2000). Identification of a novel mutation (867delA) in the glucose-6-phosphatase gene in two siblings with glycogen storage disease type Ia with different phenotypes. Human Mutation. 15(4). 381–381. 10 indexed citations
10.
Rake, Jan Peter, Gepke Visser, Edwin Verlind, et al.. (2000). Glycogen storage disease type Ia: recent experience with mutation analysis, a summary of mutations reported in the literature and a newly developed diagnostic flowchart. European Journal of Pediatrics. 159(5). 322–330. 52 indexed citations
11.
Hayes, Vanessa M., Clemens M.F. Dirven, Anke Dam, et al.. (1999). High Frequency of TP53 Mutations in Juvenile Pilocytic Astrocytomas Indicates Role of TP53 in the Development of These Tumors. Brain Pathology. 9(3). 463–467. 38 indexed citations
12.
Hayes, Vanessa M., Wim Bleeker, Edwin Verlind, et al.. (1999). Comprehensive TP53-Denaturing Gradient Gel Electrophoresis Mutation Detection Assay Also Applicable to Archival Paraffin-Embedded Tissue. Diagnostic Molecular Pathology. 8(1). 2–10. 25 indexed citations
13.
Rake, Jan Peter, Edwin Verlind, Gepke Visser, et al.. (1999). Glycogen storage disease type Ia: Four novel mutations (175delGG, R170X, G266V and V338F) identified. Human Mutation. 13(2). 173–173. 10 indexed citations
14.
Scheffer, Hans, E. R. Brunt, Pieter van der Vlies, et al.. (1998). Three novel KCNA1 mutations in episodic ataxia type I families. Human Genetics. 102(4). 464–466. 48 indexed citations
17.
Kooy, R. Frank, et al.. (1995). An Integrated Map of Human Chromosome 13 Allowing Regional Localization of Genetic Markers. European Journal of Human Genetics. 3(3). 180–187. 2 indexed citations
18.
Kooy, R. Frank, Edwin Verlind, Roderick H.J. Houwen, et al.. (1994). A Deletion Hybrid Breakpoint Mapof the Chromosomal Region13q14-q21 Orders 19 GeneticMarkers in 10 Intervals. European Journal of Human Genetics. 2(1). 59–65. 7 indexed citations
19.
Scheffer, Hans, et al.. (1992). Identification of crossovers in Wilson disease families as reference points for a genetic localization of the gene. Human Genetics. 89(6). 607–11. 6 indexed citations
20.
Scheffer, Hans, David J. Bruinvels, Gerard J. te Meerman, et al.. (1990). Frequency of the ΔF508 mutation and XV2c,KM19 haplotypes in cystic fibrosis families from The Netherlands: haplotypes without ΔF508 still in disequilibrium. Human Genetics. 85(4). 425–427. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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