Edward J. Sanderlin

633 total citations
11 papers, 380 citations indexed

About

Edward J. Sanderlin is a scholar working on Molecular Biology, Surgery and Oncology. According to data from OpenAlex, Edward J. Sanderlin has authored 11 papers receiving a total of 380 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Surgery and 4 papers in Oncology. Recurrent topics in Edward J. Sanderlin's work include Drug Transport and Resistance Mechanisms (4 papers), Sphingolipid Metabolism and Signaling (3 papers) and Helicobacter pylori-related gastroenterology studies (3 papers). Edward J. Sanderlin is often cited by papers focused on Drug Transport and Resistance Mechanisms (4 papers), Sphingolipid Metabolism and Signaling (3 papers) and Helicobacter pylori-related gastroenterology studies (3 papers). Edward J. Sanderlin collaborates with scholars based in United States and Switzerland. Edward J. Sanderlin's co-authors include Li V. Yang, Mona A. Marie, Calvin R. Justus, Pius Loetscher, Juraj Velcicky, Kvin Lertpiriyapong, Heng Hong, Swati Satturwar, James G. Fox and Qi Cai and has published in prestigious journals such as Nature Communications, Cancer Research and International Journal of Molecular Sciences.

In The Last Decade

Edward J. Sanderlin

10 papers receiving 379 citations

Peers

Edward J. Sanderlin
Wen-Cheng Chung United States
Miao He China
Shuliang Shi China
Shruti Pandita United States
Chetan K. Rane United States
Saúl Álvarez–Teijeiro Spain
Julian Musa Germany
Ewa Stypulkowski United States
Wen-Cheng Chung United States
Edward J. Sanderlin
Citations per year, relative to Edward J. Sanderlin Edward J. Sanderlin (= 1×) peers Wen-Cheng Chung

Countries citing papers authored by Edward J. Sanderlin

Since Specialization
Citations

This map shows the geographic impact of Edward J. Sanderlin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward J. Sanderlin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward J. Sanderlin more than expected).

Fields of papers citing papers by Edward J. Sanderlin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edward J. Sanderlin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward J. Sanderlin. The network helps show where Edward J. Sanderlin may publish in the future.

Co-authorship network of co-authors of Edward J. Sanderlin

This figure shows the co-authorship network connecting the top 25 collaborators of Edward J. Sanderlin. A scholar is included among the top collaborators of Edward J. Sanderlin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edward J. Sanderlin. Edward J. Sanderlin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Justus, Calvin R., Mona A. Marie, Edward J. Sanderlin, & Li V. Yang. (2024). The Roles of Proton-Sensing G-Protein-Coupled Receptors in Inflammation and Cancer. Genes. 15(9). 1151–1151. 8 indexed citations
2.
Justus, Calvin R., Mona A. Marie, Edward J. Sanderlin, & Li V. Yang. (2023). Transwell In Vitro Cell Migration and Invasion Assays. Methods in molecular biology. 2644. 349–359. 76 indexed citations
3.
Marie, Mona A., Edward J. Sanderlin, Swati Satturwar, et al.. (2023). GPR4 Knockout Attenuates Intestinal Inflammation and Forestalls the Development of Colitis-Associated Colorectal Cancer in Murine Models. Cancers. 15(20). 4974–4974. 6 indexed citations
4.
Sanderlin, Edward J., Melissa M. Keenan, Martin Mense, et al.. (2022). CFTR mRNAs with nonsense codons are degraded by the SMG6-mediated endonucleolytic decay pathway. Nature Communications. 13(1). 2344–2344. 19 indexed citations
5.
Marie, Mona A., Edward J. Sanderlin, Alexander F. Hoffman, & Li V. Yang. (2022). Abstract 6139: GPR4 promotes the development of colitis-associated colorectal cancer in an experimental mouse model. Cancer Research. 82(12_Supplement). 6139–6139.
6.
Marie, Mona A., Edward J. Sanderlin, Swati Satturwar, et al.. (2021). GPR65 (TDAG8) inhibits intestinal inflammation and colitis-associated colorectal cancer development in experimental mouse models. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1868(1). 166288–166288. 29 indexed citations
7.
Sanderlin, Edward J., et al.. (2020). The Proton-Sensing GPR4 Receptor Regulates Paracellular Gap Formation and Permeability of Vascular Endothelial Cells. iScience. 23(2). 100848–100848. 38 indexed citations
8.
Sanderlin, Edward J., Mona A. Marie, Juraj Velcicky, Pius Loetscher, & Li V. Yang. (2019). Pharmacological inhibition of GPR4 remediates intestinal inflammation in a mouse colitis model. European Journal of Pharmacology. 852. 218–230. 34 indexed citations
9.
Justus, Calvin R., Edward J. Sanderlin, Lixue Dong, et al.. (2017). Contextual tumor suppressor function of T cell death-associated gene 8 (TDAG8) in hematological malignancies. Journal of Translational Medicine. 15(1). 204–204. 25 indexed citations
10.
Sanderlin, Edward J., Nancy R. Leffler, Kvin Lertpiriyapong, et al.. (2016). GPR4 deficiency alleviates intestinal inflammation in a mouse model of acute experimental colitis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1863(2). 569–584. 44 indexed citations
11.
Sanderlin, Edward J., et al.. (2015). Molecular Connections between Cancer Cell Metabolism and the Tumor Microenvironment. International Journal of Molecular Sciences. 16(5). 11055–11086. 101 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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