Edward G. Coles

828 total citations
9 papers, 686 citations indexed

About

Edward G. Coles is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Edward G. Coles has authored 9 papers receiving a total of 686 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Genetics and 1 paper in Cellular and Molecular Neuroscience. Recurrent topics in Edward G. Coles's work include Developmental Biology and Gene Regulation (6 papers), Congenital heart defects research (4 papers) and Wnt/β-catenin signaling in development and cancer (3 papers). Edward G. Coles is often cited by papers focused on Developmental Biology and Gene Regulation (6 papers), Congenital heart defects research (4 papers) and Wnt/β-catenin signaling in development and cancer (3 papers). Edward G. Coles collaborates with scholars based in United States, United Kingdom and Switzerland. Edward G. Coles's co-authors include Marianne Bronner‐Fraser, Lisa A. Taneyhill, David G. Wilkinson, Jeffrey H. Christiansen, Greg Elgar, Veronica van Heyningen, Nicholas D. Hastie, Dirk‐Jan Kleinjan, Laura S. Gammill and Jeffrey H. Miner and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Development and Current Opinion in Cell Biology.

In The Last Decade

Edward G. Coles

9 papers receiving 672 citations

Peers

Edward G. Coles
Elise Lamar United States
Jen‐Chih Hsieh United States
Micheline N. Laurent United States
Souichi Ogata United States
Olivia Orozco United States
Alexander B. Zhadanov United States
Edward G. Coles
Citations per year, relative to Edward G. Coles Edward G. Coles (= 1×) peers Heather P. Ostendorff

Countries citing papers authored by Edward G. Coles

Since Specialization
Citations

This map shows the geographic impact of Edward G. Coles's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward G. Coles with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward G. Coles more than expected).

Fields of papers citing papers by Edward G. Coles

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edward G. Coles. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward G. Coles. The network helps show where Edward G. Coles may publish in the future.

Co-authorship network of co-authors of Edward G. Coles

This figure shows the co-authorship network connecting the top 25 collaborators of Edward G. Coles. A scholar is included among the top collaborators of Edward G. Coles based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edward G. Coles. Edward G. Coles is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Coles, Edward G., Elizabeth R. Lawlor, & Marianne Bronner‐Fraser. (2008). EWS-FLI1 Causes Neuroepithelial Defects and Abrogates Emigration of Neural Crest Stem Cells. Stem Cells. 26(9). 2237–2244. 14 indexed citations
2.
Coles, Edward G., Lisa A. Taneyhill, & Marianne Bronner‐Fraser. (2007). A critical role for Cadherin6B in regulating avian neural crest emigration. Developmental Biology. 312(2). 533–544. 105 indexed citations
3.
Taneyhill, Lisa A., Edward G. Coles, & Marianne Bronner‐Fraser. (2007). Snail2 directly represses cadherin6B during epithelial-to-mesenchymal transitions of the neural crest. Development. 134(8). 1481–1490. 161 indexed citations
4.
Coles, Edward G., Laura S. Gammill, Jeffrey H. Miner, & Marianne Bronner‐Fraser. (2005). Abnormalities in neural crest cell migration in laminin α5 mutant mice. Developmental Biology. 289(1). 218–228. 57 indexed citations
5.
Coles, Edward G., Jeffrey H. Christiansen, Androulla Economou, Marianne Bronner‐Fraser, & David G. Wilkinson. (2004). A vertebrate crossveinless 2 homologue modulates BMP activity and neural crest cell migration. Development. 131(21). 5309–5317. 56 indexed citations
6.
Golding, Jon P., et al.. (2003). Roles of erbB4, rhombomere-specific, and rhombomere-independent cues in maintaining neural crest-free zones in the embryonic head. Developmental Biology. 266(2). 361–372. 34 indexed citations
7.
Christiansen, Jeffrey H., Edward G. Coles, Vicky Robinson, Andrea Pasini, & David G. Wilkinson. (2001). Screening from a subtracted embryonic chick hindbrain cDNA library: identification of genes expressed during hindbrain, midbrain and cranial neural crest development. Mechanisms of Development. 102(1-2). 119–133. 24 indexed citations
8.
Christiansen, Jeffrey H., Edward G. Coles, & David G. Wilkinson. (2000). Molecular control of neural crest formation, migration and differentiation. Current Opinion in Cell Biology. 12(6). 719–724. 136 indexed citations
9.
Elgar, Greg, et al.. (1998). Complete sequencing of the Fugu WAGR region from WT1 to PAX6: Dramatic compaction and conservation of synteny with human chromosome 11p13. Proceedings of the National Academy of Sciences. 95(22). 13068–13072. 99 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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