Eduardo Chávez

545 total citations
31 papers, 438 citations indexed

About

Eduardo Chávez is a scholar working on Sensory Systems, Cellular and Molecular Neuroscience and Biomedical Engineering. According to data from OpenAlex, Eduardo Chávez has authored 31 papers receiving a total of 438 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Sensory Systems, 5 papers in Cellular and Molecular Neuroscience and 5 papers in Biomedical Engineering. Recurrent topics in Eduardo Chávez's work include Hearing, Cochlea, Tinnitus, Genetics (13 papers), Ear Surgery and Otitis Media (4 papers) and Neurogenesis and neuroplasticity mechanisms (4 papers). Eduardo Chávez is often cited by papers focused on Hearing, Cochlea, Tinnitus, Genetics (13 papers), Ear Surgery and Otitis Media (4 papers) and Neurogenesis and neuroplasticity mechanisms (4 papers). Eduardo Chávez collaborates with scholars based in United States, Peru and Switzerland. Eduardo Chávez's co-authors include Allen F. Ryan, Kwang Pak, Lina Mullen, Yves Brand, Sara Euteneuer, Masatsugu Masuda, Kenji Kondo, Keiji Tabuchi, Ryoukichi Ikeda and Arwa Kurabi and has published in prestigious journals such as Journal of Clinical Investigation, PLoS ONE and Scientific Reports.

In The Last Decade

Eduardo Chávez

30 papers receiving 435 citations

Peers

Eduardo Chávez
Dongguang Wei United States
Jessica M. Appler United States
Hiu-Tung Wong United States
Judith S. Kempfle United States
Eduardo Chávez
Citations per year, relative to Eduardo Chávez Eduardo Chávez (= 1×) peers Marja Boström

Countries citing papers authored by Eduardo Chávez

Since Specialization
Citations

This map shows the geographic impact of Eduardo Chávez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eduardo Chávez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eduardo Chávez more than expected).

Fields of papers citing papers by Eduardo Chávez

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eduardo Chávez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eduardo Chávez. The network helps show where Eduardo Chávez may publish in the future.

Co-authorship network of co-authors of Eduardo Chávez

This figure shows the co-authorship network connecting the top 25 collaborators of Eduardo Chávez. A scholar is included among the top collaborators of Eduardo Chávez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eduardo Chávez. Eduardo Chávez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chávez, Eduardo, et al.. (2024). Control of an aquaponic system to improve the yield of gray tilapia and lettuce cultivation. International Journal of Power Electronics and Drive Systems/International Journal of Electrical and Computer Engineering. 15(1). 505–505. 1 indexed citations
3.
Lee, Eun-Jin, et al.. (2024). Mutations in unfolded protein response regulator ATF6 cause hearing and vision loss syndrome. Journal of Clinical Investigation. 135(3). 3 indexed citations
4.
Chávez, Eduardo, et al.. (2023). Wearable with integrated piezoelectric energy harvester for geolocation of people with Alzheimer's. International Journal of Power Electronics and Drive Systems/International Journal of Electrical and Computer Engineering. 14(1). 497–497.
5.
Sakamoto, Takashi, Kwang Pak, Eduardo Chávez, Allen F. Ryan, & Arwa Kurabi. (2022). HB-EGF Plays a Pivotal Role in Mucosal Hyperplasia During Otitis Media Induced by a Viral Analog. Frontiers in Cellular and Infection Microbiology. 12. 823714–823714. 6 indexed citations
6.
Kurabi, Arwa, et al.. (2022). A transcytotic transport mechanism across the tympanic membrane. Scientific Reports. 12(1). 984–984. 9 indexed citations
7.
Chávez, Eduardo, Kwang Pak, Arwa Kurabi, et al.. (2021). A Screen of Autophagy Compounds Implicates the Proteasome in Mammalian Aminoglycoside-Induced Hair Cell Damage. Frontiers in Cell and Developmental Biology. 9. 762751–762751. 7 indexed citations
8.
Noel, Julia E., Keigo Suzukawa, Eduardo Chávez, et al.. (2020). A kinase inhibitor screen identifies signaling pathways regulating mucosal growth during otitis media. PLoS ONE. 15(8). e0235634–e0235634. 3 indexed citations
9.
Brand, Yves, Kwang Pak, Eduardo Chávez, et al.. (2014). Neural Cell Adhesion Molecule NrCAM Is Expressed in the Mammalian Inner Ear and Modulates Spiral Ganglion Neurite Outgrowth in an In Vitro Alternate Choice Assay. Journal of Molecular Neuroscience. 55(4). 836–844. 8 indexed citations
10.
Chávez, Eduardo, Neha Jain, Soledad Levano, et al.. (2014). Inhibition of MMP-2 but not MMP-9 Influences Inner Ear Spiral Ganglion Neurons In Vitro. Cellular and Molecular Neurobiology. 34(7). 1011–1021. 5 indexed citations
11.
Suzukawa, Keigo, Kwang Pak, Eduardo Chávez, et al.. (2014). A Mouse Model of Otitis Media Identifies HB-EGF as a Mediator of Inflammation-Induced Mucosal Proliferation. PLoS ONE. 9(7). e102739–e102739. 20 indexed citations
12.
Ikeda, Ryoukichi, Kwang Pak, Eduardo Chávez, & Allen F. Ryan. (2014). Transcription Factors with Conserved Binding Sites Near ATOH1 on the POU4F3 Gene Enhance the Induction of Cochlear Hair Cells. Molecular Neurobiology. 51(2). 672–684. 39 indexed citations
13.
Brand, Yves, et al.. (2013). Neural Cell Adhesion Molecule L1 Modulates Type I But Not Type II Inner Ear Spiral Ganglion Neurite Outgrowth in an In Vitro Alternate Choice Assay. Journal of Molecular Neuroscience. 51(3). 663–670. 7 indexed citations
14.
Kondo, Kenji, Kwang Pak, Eduardo Chávez, et al.. (2013). Changes in responsiveness of rat spiral ganglion neurons to neurotrophins across age: differential regulation of survival and neuritogenesis. International Journal of Neuroscience. 123(7). 465–475. 12 indexed citations
15.
Masuda, Masatsugu, Kwang Pak, Eduardo Chávez, & Allen F. Ryan. (2012). TFE2 and GATA3 enhance induction of POU4F3 and myosin VIIa positive cells in nonsensory cochlear epithelium by ATOH1. Developmental Biology. 372(1). 68–80. 41 indexed citations
16.
Euteneuer, Sara, Kuo H. Yang, Eduardo Chávez, et al.. (2012). Glial cell line-derived neurotrophic factor (GDNF) induces neuritogenesis in the cochlear spiral ganglion via neural cell adhesion molecule (NCAM). Molecular and Cellular Neuroscience. 54. 30–43. 41 indexed citations
17.
18.
Taura, Kojiro, Yun‐Hoon Choung, Masatsugu Masuda, et al.. (2010). Histone deacetylase inhibition enhances adenoviral vector transduction in inner ear tissue. Neuroscience. 166(4). 1185–1193. 8 indexed citations
19.
Euteneuer, Sara, Eduardo Chávez, Lina Mullen, et al.. (2007). Laminin and fibronectin modulate inner ear spiral ganglion neurite outgrowth in an in vitro alternate choice assay. Developmental Neurobiology. 67(13). 1721–1730. 49 indexed citations
20.
Neyts, Johan, Marisa Flores-Aguilar, Irene L. Smith, et al.. (1995). Absence of Infectious Retinitis after Injection of Human Cytomegalovirus into Rabbit Eyes. The Journal of Infectious Diseases. 171(4). 782–787. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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