Edith Hümmler

11.5k total citations · 3 hit papers
133 papers, 8.9k citations indexed

About

Edith Hümmler is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Edith Hümmler has authored 133 papers receiving a total of 8.9k indexed citations (citations by other indexed papers that have themselves been cited), including 111 papers in Molecular Biology, 48 papers in Pulmonary and Respiratory Medicine and 45 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Edith Hümmler's work include Ion Transport and Channel Regulation (78 papers), Electrolyte and hormonal disorders (42 papers) and Hormonal Regulation and Hypertension (42 papers). Edith Hümmler is often cited by papers focused on Ion Transport and Channel Regulation (78 papers), Electrolyte and hormonal disorders (42 papers) and Hormonal Regulation and Hypertension (42 papers). Edith Hümmler collaborates with scholars based in Switzerland, United States and Germany. Edith Hümmler's co-authors include Bernard C. Rossier, Friedrich Beermann, Pierre Barker, Sylvain Pradervand, Wolfgang Schmid, Adriano Aguzzi, Julie A. Blendy, John T. Gatzy, Chantal Verdumo and G. Schütz and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Edith Hümmler

133 papers receiving 8.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Targeted disruption of the glucocorticoid receptor gene blocks adrenergic chromaffin cell development and severely retards lung maturation.

1995 748 citations Edith Hümmler et al. profile →
Early death due to defective neonatal lung liquid clearance in αENaC-deficient mice

1996 727 citations Edith Hümmler, Pierre Barker et al. profile →
The cells and peripheral representation of sodium taste in mice

2010 494 citations Edith Hümmler et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Edith Hümmler Switzerland	48	5.6k	2.1k	1.6k	1.6k	1.0k	133	8.9k
Cecilia M. Canessa United States	37	6.3k 1.1×	2.6k 1.2×	1.5k 0.9×	800 0.5×	350 0.3×	67	7.8k
Siegfried Waldegger Germany	47	5.1k 0.9×	918 0.4×	467 0.3×	1.7k 1.1×	366 0.4×	97	8.6k
Nicolette Farman France	50	4.3k 0.8×	1.5k 0.7×	3.4k 2.1×	509 0.3×	733 0.7×	143	8.3k
Itaru Kojima Japan	54	5.3k 0.9×	346 0.2×	1.7k 1.0×	1.1k 0.7×	1.1k 1.1×	226	10.1k
Jonathan Lytton Canada	43	7.5k 1.3×	586 0.3×	366 0.2×	1.1k 0.7×	650 0.6×	91	10.5k
Maria Svelto Italy	55	5.9k 1.0×	1.8k 0.9×	409 0.3×	478 0.3×	325 0.3×	219	8.2k
Richard Warth Germany	46	4.1k 0.7×	728 0.3×	1.2k 0.7×	290 0.2×	257 0.3×	108	5.4k
Giorgio Casari Italy	55	5.6k 1.0×	653 0.3×	761 0.5×	578 0.4×	1.1k 1.1×	144	10.5k
Xinmin Zhang United States	39	4.1k 0.7×	285 0.1×	322 0.2×	953 0.6×	630 0.6×	140	7.2k
Roberto Ravazzolo Italy	39	3.6k 0.6×	1.4k 0.6×	233 0.1×	215 0.1×	936 0.9×	206	6.5k

Countries citing papers authored by Edith Hümmler

Since Specialization

Citations

This map shows the geographic impact of Edith Hümmler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edith Hümmler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edith Hümmler more than expected).

Fields of papers citing papers by Edith Hümmler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edith Hümmler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edith Hümmler. The network helps show where Edith Hümmler may publish in the future.

Co-authorship network of co-authors of Edith Hümmler

This figure shows the co-authorship network connecting the top 25 collaborators of Edith Hümmler. A scholar is included among the top collaborators of Edith Hümmler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edith Hümmler. Edith Hümmler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Auberson, Muriel, et al.. (2024). Tmprss2 maintains epithelial barrier integrity and transepithelial sodium transport. Life Science Alliance. 7(3). e202302304–e202302304. 5 indexed citations

Maillard, Marc, et al.. (2022). Kidney-Specific CAP1/Prss8-Deficient Mice Maintain ENaC-Mediated Sodium Balance Through an Aldosterone Independent Pathway. Journal of the American Society of Nephrology. 33(11S). 812–812. 1 indexed citations

Mérillat, Anne‐Marie, Qing Wang, Marc Maillard, et al.. (2022). Kidney-Specific CAP1/Prss8-Deficient Mice Maintain ENaC-Mediated Sodium Balance through an Aldosterone Independent Pathway. International Journal of Molecular Sciences. 23(12). 6745–6745. 9 indexed citations

Nesterov, Viatcheslav, Marko Bertog, Jérémie Canonica, et al.. (2021). Critical role of the mineralocorticoid receptor in aldosterone-dependent and aldosterone-independent regulation of ENaC in the distal nephron. American Journal of Physiology-Renal Physiology. 321(3). F257–F268. 28 indexed citations

Dizin, Éva, Valérie Olivier, Isabelle Roth, et al.. (2021). Activation of the Hypoxia-Inducible Factor Pathway Inhibits Epithelial Sodium Channel–Mediated Sodium Transport in Collecting Duct Principal Cells. Journal of the American Society of Nephrology. 32(12). 3130–3145. 13 indexed citations

Wang, Yubao, Isabelle Roth, Valérie Olivier, et al.. (2020). Interaction between Epithelial Sodium Channel γ-Subunit and Claudin-8 Modulates Paracellular Sodium Permeability in Renal Collecting Duct. Journal of the American Society of Nephrology. 31(5). 1009–1023. 24 indexed citations

Maric, Darko, Camille Ansermet, Damien De Bellis, et al.. (2019). Deletion of the serine protease CAP2/Tmprss4 leads to dysregulated renal water handling upon dietary potassium depletion. Scientific Reports. 9(1). 19540–19540. 11 indexed citations

Koppes, Sjors A., Kristiane A. Engebretsen, Tove Agner, et al.. (2017). Current knowledge on biomarkers for contact sensitization and allergic contact dermatitis. Contact Dermatitis. 77(1). 1–16. 54 indexed citations

Bergmann, Silke, Judith M. A. van den Brand, Stefan Pöhlmann, et al.. (2016). The Proteolytic Activation of (H3N2) Influenza A Virus Hemagglutinin Is Facilitated by Different Type II Transmembrane Serine Proteases. Journal of Virology. 90(9). 4298–4307. 37 indexed citations

10.

Sun, Chengsan, Edith Hümmler, & David L. Hill. (2016). Selective Deletion of Sodium Salt Taste during Development Leads to Expanded Terminal Fields of Gustatory Nerves in the Adult Mouse Nucleus of the Solitary Tract. Journal of Neuroscience. 37(3). 660–672. 11 indexed citations

11.

Allaman-Pillet, Nathalie, et al.. (2015). Tgfbi/Bigh3 silencing activates ERK in mouse retina. Experimental Eye Research. 140. 159–170. 4 indexed citations

12.

Fu, Yiling, Maria Gerasimova, Paul W. Sanders, et al.. (2014). PPARγ Agonist-Induced Fluid Retention Depends on αENaC Expression in Connecting Tubules. The Nephron journals/Nephron journals. 129(1). 68–74. 14 indexed citations

13.

Pech, Vladimír, Monika Thumová, Young Hee Kim, et al.. (2012). ENaC inhibition stimulates Cl ⁻ secretion in the mouse cortical collecting duct through an NKCC1-dependent mechanism. American Journal of Physiology-Renal Physiology. 303(1). F45–F55. 26 indexed citations

14.

Hümmler, Edith & Carole Planès. (2009). Importance of ENaC-Mediated Sodium Transport in Alveolar Fluid Clearance Using Genetically-Engineered Mice. Cellular Physiology and Biochemistry. 25(1). 63–70. 48 indexed citations

15.

Planès, Carole, Céline Leyvraz, Tokujiro Uchida, et al.. (2005). In vitro and in vivo regulation of transepithelial lung alveolar sodium transport by serine proteases. American Journal of Physiology-Lung Cellular and Molecular Physiology. 288(6). L1099–L1109. 65 indexed citations

16.

Mauro, Theodora M., Martin J. Behne, Yuko Oda, et al.. (2002). The ENaC Channel is Required for Normal Epidermal Differentiation. Journal of Investigative Dermatology. 118(4). 589–594. 53 indexed citations

17.

Rubera, Isabelle, Grégoire Vuagniaux, Anne‐Marie Mérillat, et al.. (2002). A conditional allele at the mouse channel activating protease 1 (Prss8) gene locus. genesis. 32(2). 173–176. 20 indexed citations

18.

Hümmler, Edith, Anne‐Marie Mérillat, Isabelle Rubera, Bernard C. Rossier, & Friedrich Beermann. (2002). Conditional gene targeting of the Scnn1a (αENaC) gene locus. genesis. 32(2). 169–172. 36 indexed citations

19.

Hümmler, Edith. (1999). Implication of ENaC in salt-sensitive hypertension. The Journal of Steroid Biochemistry and Molecular Biology. 69(1-6). 385–390. 26 indexed citations

20.

Barker, Pierre, J. T. Gatzy, Barbara R. Grubb, et al.. (1998). Role of gammaENaC subunit in lung liquid clearance and electrolyte balance in newborn mice. Insights into perinatal adaptation and pseudohypoaldosteronism.. Journal of Clinical Investigation. 102(8). 1634–1640. 200 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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