Edith Álvarez-León

670 total citations
29 papers, 522 citations indexed

About

Edith Álvarez-León is a scholar working on Immunology, Molecular Biology and Epidemiology. According to data from OpenAlex, Edith Álvarez-León has authored 29 papers receiving a total of 522 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Immunology, 9 papers in Molecular Biology and 8 papers in Epidemiology. Recurrent topics in Edith Álvarez-León's work include Atherosclerosis and Cardiovascular Diseases (9 papers), Adipokines, Inflammation, and Metabolic Diseases (6 papers) and Immune Cell Function and Interaction (4 papers). Edith Álvarez-León is often cited by papers focused on Atherosclerosis and Cardiovascular Diseases (9 papers), Adipokines, Inflammation, and Metabolic Diseases (6 papers) and Immune Cell Function and Interaction (4 papers). Edith Álvarez-León collaborates with scholars based in Mexico and Brazil. Edith Álvarez-León's co-authors include Gilberto Vargas‐Alarcón, José Manuel Fragoso, Maité Vallejo, Jesús K. Yamamoto‐Furusho, Rosalinda Posadas‐Sánchez, José Manuel Rodrı́guez-Pérez, Pedro A. Reyes, Carlos Posadas‐Romero, Nonanzit Pérez‐Hernández and Teresa Villarreal‐Molina and has published in prestigious journals such as PLoS ONE, International Journal of Molecular Sciences and BioMed Research International.

In The Last Decade

Edith Álvarez-León

28 papers receiving 514 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Edith Álvarez-León Mexico	16	212	135	98	70	70	29	522
Stefania Gaspari Italy	15	180 0.8×	218 1.6×	50 0.5×	67 1.0×	96 1.4×	46	659
Eduardo Gómez-Bañuelos Mexico	16	189 0.9×	91 0.7×	83 0.8×	30 0.4×	57 0.8×	33	556
Robert W. Siggins United States	20	176 0.8×	241 1.8×	169 1.7×	110 1.6×	52 0.7×	35	735
Marie‐France Belair France	14	198 0.9×	174 1.3×	67 0.7×	130 1.9×	54 0.8×	19	716
Scott E. Wenderfer United States	16	297 1.4×	144 1.1×	46 0.5×	35 0.5×	28 0.4×	60	780
Ruksana Huda United States	14	105 0.5×	90 0.7×	106 1.1×	53 0.8×	38 0.5×	29	586
P. Roux‐Lombard Switzerland	15	410 1.9×	111 0.8×	83 0.8×	62 0.9×	145 2.1×	18	814
Ismael Dale Cotrim Guerreiro da Silva Brazil	18	127 0.6×	185 1.4×	103 1.1×	90 1.3×	40 0.6×	53	668
Sajjad Rafiq United Kingdom	12	134 0.6×	157 1.2×	73 0.7×	35 0.5×	82 1.2×	16	511
Helen Williams Australia	14	211 1.0×	102 0.8×	113 1.2×	39 0.6×	70 1.0×	23	476

Countries citing papers authored by Edith Álvarez-León

Since Specialization

Citations

This map shows the geographic impact of Edith Álvarez-León's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edith Álvarez-León with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edith Álvarez-León more than expected).

Fields of papers citing papers by Edith Álvarez-León

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Edith Álvarez-León. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edith Álvarez-León. The network helps show where Edith Álvarez-León may publish in the future.

Co-authorship network of co-authors of Edith Álvarez-León

This figure shows the co-authorship network connecting the top 25 collaborators of Edith Álvarez-León. A scholar is included among the top collaborators of Edith Álvarez-León based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Edith Álvarez-León. Edith Álvarez-León is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Vargas‐Alarcón, Gilberto, et al.. (2025). Role of miRNAs in Regulating Ascending Aortic Dilation in Bicuspid Aortic Valve Patients Operated for Aortic Stenosis. International Journal of Molecular Sciences. 26(2). 779–779. 4 indexed citations

Silva‐Palacios, Alejandro, Elizabeth Soria‐Castro, Edith Álvarez-León, et al.. (2025). Cardioprotective effect of senotherapy in chronically obese middle-aged female rats may be mediated by a MERCSs/Nrf2 interaction. The Journal of Nutritional Biochemistry. 142. 109923–109923.

Soto, María Elena, Israel Pérez-Torres, Vicente Castrejón-Téllez, et al.. (2025). Analysis of FBN1, TGFβ2, TGFβR1 and TGFβR2 mRNA as Key Molecular Mechanisms in the Damage of Aortic Aneurysm and Dissection in Marfan Syndrome. International Journal of Molecular Sciences. 26(7). 3067–3067. 2 indexed citations

Jimenez, Roland, Edith Álvarez-León, Wylly Ramsés García‐Niño, et al.. (2024). Quercetin preserves mitochondria-endoplasmic reticulum contact sites improving mitochondrial dynamics in aged myocardial cells. Biogerontology. 26(1). 29–29. 6 indexed citations

Yamamoto‐Furusho, Jesús K., Rosalinda Posadas‐Sánchez, Edith Álvarez-León, & Gilberto Vargas‐Alarcón. (2016). Protective role of Interleukin 27 (IL-27) gene polymorphisms in patients with ulcerative colitis. Immunology Letters. 172. 79–83. 26 indexed citations

Monroy-Muñoz, Irma Eloísa, Javier Ángeles-Martínez, Rosalinda Posadas‐Sánchez, et al.. (2016). PLA2G2A polymorphisms are associated with metabolic syndrome and type 2 diabetes mellitus. Results from the genetics of atherosclerotic disease Mexican study. Immunobiology. 222(10). 967–972. 15 indexed citations

Ángeles-Martínez, Javier, Rosalinda Posadas‐Sánchez, Edith Álvarez-León, et al.. (2015). Monocyte chemoattractant protein-1 gene ( MCP-1 ) polymorphisms are associated with risk of premature coronary artery disease in Mexican patients from the Genetics of Atherosclerotic Disease (GEA) study. Immunology Letters. 167(2). 125–130. 12 indexed citations

Vargas‐Alarcón, Gilberto, Miguel Cruz, Adán Valladares, et al.. (2015). The interleukin-1β-511 T>C (rs16944) gene polymorphism is associated with risk of developing silent myocardial ischemia in diabetic patients. Immunology Letters. 168(1). 7–12. 12 indexed citations

López-Reyes, Alberto, José Manuel Rodrı́guez-Pérez, Javier Fernández‐Torres, et al.. (2014). The HIF1A rs2057482 polymorphism is associated with risk of developing premature coronary artery disease and with some metabolic and cardiovascular risk factors. The Genetics of Atherosclerotic Disease (GEA) Mexican Study. Experimental and Molecular Pathology. 96(3). 405–410. 21 indexed citations

10.

Vargas‐Alarcón, Gilberto, Carlos Posadas‐Romero, Teresa Villarreal‐Molina, et al.. (2014). IL-24 Gene Polymorphisms Are Associated with Cardiometabolic Parameters and Cardiovascular Risk Factors But Not with Premature Coronary Artery Disease: The Genetics of Atherosclerotic Disease Mexican Study. Journal of Interferon & Cytokine Research. 34(9). 659–666. 11 indexed citations

11.

Yamamoto‐Furusho, Jesús K., et al.. (2014). Association of the interleukin 15 (IL-15) gene polymorphisms with the risk of developing ulcerative colitis in Mexican individuals. Molecular Biology Reports. 41(4). 2171–2176. 11 indexed citations

12.

Martínez‐Ríos, Marco Antonio, Edith Álvarez-León, Javier Ángeles-Martínez, et al.. (2014). Haplotypes of the angiotensin-converting enzyme (ACE) gene are associated with coronary artery disease but not with restenosis after coronary stenting. Experimental and Molecular Pathology. 97(1). 166–170. 11 indexed citations

13.

Cruz, Miguel, José Manuel Fragoso, Edith Álvarez-León, et al.. (2013). The TGF-B1 and IL-10 gene polymorphisms are associated with risk of developing silent myocardial ischemia in the diabetic patients. Immunology Letters. 156(1-2). 18–22. 21 indexed citations

14.

Vargas‐Alarcón, Gilberto, Carlos Posadas‐Romero, Teresa Villarreal‐Molina, et al.. (2013). Single Nucleotide Polymorphisms within LIPA (Lysosomal Acid Lipase A) Gene Are Associated with Susceptibility to Premature Coronary Artery Disease. A Replication in the Genetic of Atherosclerotic Disease (GEA) Mexican Study. PLoS ONE. 8(9). e74703–e74703. 22 indexed citations

15.

Yamamoto‐Furusho, Jesús K., et al.. (2012). Genetic polymorphisms of interleukin 20 (IL-20) in patients with ulcerative colitis. Immunology Letters. 149(1-2). 50–53. 15 indexed citations

16.

Fragoso, José Manuel, Teresa Juárez‐Cedillo, José Manuel Rodrı́guez-Pérez, et al.. (2010). The Interleukin 6 - 572 G>C (rs1800796) Polymorphism Is Associated with the Risk of Developing Acute Coronary Syndrome. Genetic Testing and Molecular Biomarkers. 14(6). 759–763. 19 indexed citations

17.

Vargas‐Alarcón, Gilberto, et al.. (2008). Comparative study of the residues 63 and 67 on the HLA‐B molecule in patients with Takayasu's arteritis and tuberculosis. Cell Biochemistry and Function. 26(7). 820–823. 8 indexed citations

18.

Soto, María Elena, et al.. (2007). Comparison Distribution of HLA-B Alleles in Mexican Patients with Takayasu Arteritis and Tuberculosis. Human Immunology. 68(5). 449–453. 32 indexed citations

19.

Rodrı́guez-Pérez, José Manuel, José Manuel Fragoso, Edith Álvarez-León, et al.. (2006). MHC class II genes in Mexican patients with idiopathic dilated cardiomyopathy. Experimental and Molecular Pathology. 82(1). 49–52. 16 indexed citations

20.

Hernández‐Pacheco, Guadalupe, José Manuel Rodrı́guez-Pérez, Nonanzit Pérez‐Hernández, et al.. (2003). Tumor necrosis factor-alpha promoter polymorphisms in Mexican patients with rheumatic heart disease. Journal of Autoimmunity. 21(1). 59–63. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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