E Schneider

2.1k total citations
21 papers, 1.8k citations indexed

About

E Schneider is a scholar working on Molecular Biology, Oncology and Infectious Diseases. According to data from OpenAlex, E Schneider has authored 21 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 12 papers in Oncology and 3 papers in Infectious Diseases. Recurrent topics in E Schneider's work include Drug Transport and Resistance Mechanisms (7 papers), Cancer therapeutics and mechanisms (6 papers) and Lung Cancer Research Studies (4 papers). E Schneider is often cited by papers focused on Drug Transport and Resistance Mechanisms (7 papers), Cancer therapeutics and mechanisms (6 papers) and Lung Cancer Research Studies (4 papers). E Schneider collaborates with scholars based in United States, Switzerland and Germany. E Schneider's co-authors include Masayuki Nakagawa, Kenneth H. Cowan, Alexey Khodjakov, José A. Sánchez‐Alcázar, Markus Borner, D D Ross, L. Austin Doyle, Julie K. Horton, James Chih‐Hsin Yang and Jeffrey G. Ault and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Blood and The Journal of Immunology.

In The Last Decade

E Schneider

21 papers receiving 1.7k citations

Peers

E Schneider
Erasmus Schneider United States
Eugene Mechetner United States
D D Ross United States
C.M. Kuiper Netherlands
H J Broxterman Netherlands
Erasmus Schneider United States
E Schneider
Citations per year, relative to E Schneider E Schneider (= 1×) peers Erasmus Schneider

Countries citing papers authored by E Schneider

Since Specialization
Citations

This map shows the geographic impact of E Schneider's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E Schneider with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E Schneider more than expected).

Fields of papers citing papers by E Schneider

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E Schneider. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E Schneider. The network helps show where E Schneider may publish in the future.

Co-authorship network of co-authors of E Schneider

This figure shows the co-authorship network connecting the top 25 collaborators of E Schneider. A scholar is included among the top collaborators of E Schneider based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E Schneider. E Schneider is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pirnia, Farzaneh, E Schneider, Daniel Betticher, & Markus Borner. (2002). Mitomycin C induces apoptosis and caspase-8 and -9 processing through a caspase-3 and Fas-independent pathway. Cell Death and Differentiation. 9(9). 905–914. 128 indexed citations
2.
Smitherman, Pamela K., Julie Aldridge, Erin L. Volk, et al.. (2001). Resistance to mitoxantrone in multidrug-resistant MCF7 breast cancer cells: evaluation of mitoxantrone transport and the role of multidrug resistance protein family proteins.. PubMed. 61(14). 5461–7. 53 indexed citations
3.
Sánchez‐Alcázar, José A., Jeffrey G. Ault, Alexey Khodjakov, & E Schneider. (2000). Increased mitochondrial cytochrome c levels and mitochondrial hyperpolarization precede camptothecin-induced apoptosis in Jurkat cells. Cell Death and Differentiation. 7(11). 1090–1100. 146 indexed citations
4.
Volk, Erin L., Kyle H. Rohde, John J. McGuire, et al.. (2000). Methotrexate cross-resistance in a mitoxantrone-selected multidrug-resistant MCF7 breast cancer cell line is attributable to enhanced energy-dependent drug efflux.. PubMed. 60(13). 3514–21. 107 indexed citations
5.
Schneider, E, et al.. (1999). Multidrug resistance.. PubMed. 18. 152–77. 4 indexed citations
6.
Ross, D D, Weidong Yang, Lynne V. Abruzzo, et al.. (1999). Atypical Multidrug Resistance: Breast Cancer Resistance Protein Messenger RNA Expression in Mitoxantrone-Selected Cell Lines. JNCI Journal of the National Cancer Institute. 91(5). 429–433. 284 indexed citations
7.
Schneider, E, M. Lambermont, Walter Feremans, et al.. (1997). Autologous stem cell infusion for acute myeloblastic leukemia in an HIV-1 carrier. Bone Marrow Transplantation. 20(7). 611–612. 11 indexed citations
8.
Tolcher, Anthony W., Joyce O’Shaughnessy, R B Weiss, et al.. (1997). A phase I study of topotecan followed sequentially by doxorubicin in patients with advanced malignancies.. PubMed. 3(5). 755–60. 22 indexed citations
9.
Yao, Rong, E Schneider, Thomas J. Ryan, & John Galivan. (1996). Human gamma-glutamyl hydrolase: cloning and characterization of the enzyme expressed in vitro.. Proceedings of the National Academy of Sciences. 93(19). 10134–10138. 70 indexed citations
10.
Schneider, E, et al.. (1995). Increased expression of the multidrug resistance-associated protein gene in relapsed acute leukemia. Blood. 85(1). 186–193. 155 indexed citations
12.
Borner, Markus, Charles E. Myers, Oliver Sartor, et al.. (1995). Drug-induced apoptosis is not necessarily dependent on macromolecular synthesis or proliferation in the p53-negative human prostate cancer cell line PC-3.. PubMed. 55(10). 2122–8. 106 indexed citations
13.
Schneider, E, et al.. (1994). Mechanisms of resistance to ansamycin antibiotics in human breast cancer cell lines.. Molecular Pharmacology. 46(4). 677–684. 29 indexed citations
14.
Kaufmann, Scott H., JE Karp, R. G. Wyn Jones, et al.. (1994). Topoisomerase II levels and drug sensitivity in adult acute myelogenous leukemia. Blood. 83(2). 517–530. 9 indexed citations
15.
Schneider, E, Julie K. Horton, James Chih‐Hsin Yang, Masayuki Nakagawa, & Kenneth H. Cowan. (1994). Multidrug resistance-associated protein gene overexpression and reduced drug sensitivity of topoisomerase II in a human breast carcinoma MCF7 cell line selected for etoposide resistance.. PubMed. 54(1). 152–8. 216 indexed citations
16.
Kaufmann, Scott H., JE Karp, R. G. Wyn Jones, et al.. (1994). Topoisomerase II levels and drug sensitivity in adult acute myelogenous leukemia. Blood. 83(2). 517–530. 99 indexed citations
17.
Nakagawa, Masayuki, E Schneider, Katharine H. Dixon, et al.. (1992). Reduced intracellular drug accumulation in the absence of P-glycoprotein (mdr1) overexpression in mitoxantrone-resistant human MCF-7 breast cancer cells.. PubMed. 52(22). 6175–81. 119 indexed citations
18.
Laeng, Hubert, E Schneider, Reinhard Bolli, et al.. (1988). Participation of mitochondrial proliferation in morphological differentiation of murine mastocytoma cells. Experimental Cell Research. 179(1). 222–232. 18 indexed citations
19.
Mann, Douglas L., Elizabeth Read-Connole, L O Arthur, et al.. (1988). HLA-DR is involved in the HIV-1 binding site on cells expressing MHC class II antigens.. The Journal of Immunology. 141(4). 1131–1136. 34 indexed citations
20.
Schneider, E, et al.. (1984). DNA Polymerase Activities of Mammalian Cell-Cycle Mutants and “Wild-Type” Cells in Different States of Proliferative Activity and Quiescence. Advances in experimental medicine and biology. 179. 331–335. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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