E. Ogawa

2.6k total citations · 1 hit paper
20 papers, 2.3k citations indexed

About

E. Ogawa is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, E. Ogawa has authored 20 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Oncology and 5 papers in Pulmonary and Respiratory Medicine. Recurrent topics in E. Ogawa's work include Acute Myeloid Leukemia Research (5 papers), Retinoids in leukemia and cellular processes (5 papers) and RNA Interference and Gene Delivery (4 papers). E. Ogawa is often cited by papers focused on Acute Myeloid Leukemia Research (5 papers), Retinoids in leukemia and cellular processes (5 papers) and RNA Interference and Gene Delivery (4 papers). E. Ogawa collaborates with scholars based in Japan, Canada and United States. E. Ogawa's co-authors include Masanobu Satake, Katsuya Shigesada, Yoshiaki Ito, Manabu Inuzuka, Mitsuo Maruyama, Hiroshi Kagoshima, Jie Lu, Masato Maruyama, Yuki Ito and Suk-Chul Bae and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Oncology and Molecular and Cellular Biology.

In The Last Decade

E. Ogawa

19 papers receiving 2.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

PEBP2/PEA2 represents a family of transcription factors homologous to the products of the Drosophila runt gene and the human AML1 gene.

1993 548 citations E. Ogawa, Masato Maruyama et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
E. Ogawa Japan	12	1.8k	877	620	369	306	20	2.3k
A. Thomas Look United States	27	1.2k 0.7×	515 0.6×	596 1.0×	322 0.9×	312 1.0×	40	2.3k
Christoph Lutz Germany	16	971 0.5×	1.1k 1.2×	515 0.8×	497 1.3×	476 1.6×	28	2.2k
D E Zhang United States	9	1.2k 0.6×	942 1.1×	228 0.4×	466 1.3×	151 0.5×	9	1.7k
I Miyoshi Japan	17	1.2k 0.7×	594 0.7×	643 1.0×	283 0.8×	100 0.3×	77	2.1k
Shin-ichi Mizuno Japan	14	1.1k 0.6×	774 0.9×	276 0.4×	1.2k 3.1×	140 0.5×	16	2.4k
Li Chong Chan Hong Kong	20	1.1k 0.6×	735 0.8×	335 0.5×	229 0.6×	247 0.8×	39	2.2k
Yumi Yashiro–Ohtani United States	20	1.7k 0.9×	391 0.4×	545 0.9×	1.3k 3.5×	335 1.1×	28	3.0k
Douglas Tkachuk United States	14	1.1k 0.6×	734 0.8×	245 0.4×	337 0.9×	123 0.4×	17	2.0k
Niklas Feldhahn United States	21	1.2k 0.6×	256 0.3×	522 0.8×	642 1.7×	179 0.6×	41	2.1k
Yuka Harada Japan	23	1.0k 0.6×	1.2k 1.4×	203 0.3×	267 0.7×	229 0.7×	106	2.0k

Countries citing papers authored by E. Ogawa

Since Specialization

Citations

This map shows the geographic impact of E. Ogawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. Ogawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. Ogawa more than expected).

Fields of papers citing papers by E. Ogawa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. Ogawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. Ogawa. The network helps show where E. Ogawa may publish in the future.

Co-authorship network of co-authors of E. Ogawa

This figure shows the co-authorship network connecting the top 25 collaborators of E. Ogawa. A scholar is included among the top collaborators of E. Ogawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E. Ogawa. E. Ogawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Ogata, Tsuyoshi, et al.. (2021). Secondary Attack Rate among Non-Spousal Household Contacts of Coronavirus Disease 2019 in Tsuchiura, Japan, August 2020–February 2021. International Journal of Environmental Research and Public Health. 18(17). 8921–8921. 11 indexed citations

2.

Takenaka, Kazumasa, et al.. (2012). [Lung cancer accompanied by active pulmonary tuberculosis].. PubMed. 65(5). 401–4.

3.

Tanaka, Fumihiro, E. Ogawa, Hiromi Wada, et al.. (2006). Clinical significance of HuR expression, an mRNA-binding protein in non-small cell lung cancer (NSCLC). Journal of Clinical Oncology. 24(18_suppl). 10098–10098. 1 indexed citations

4.

Ogawa, E., Fumihiro Tanaka, Kazuyoshi Yanagihara, & Hiroshi Wada. (2005). A phase II trial of adjuvant chemotherapy with weekly paclitaxel (Pac) and carboplatin (Cb) followed by UFT for completely resected pathologic (p-) stage II (N1) and IIIA (N2) non-small cell lung cancer (NSCLC). Journal of Clinical Oncology. 23(16_suppl). 7315–7315. 1 indexed citations

5.

Ogawa, E., Kazumasa Takenaka, Kazuhiro Yanagihara, et al.. (2004). Clinical significance of VEGF-C status in tumour cells and stromal macrophages in non-small cell lung cancer patients. British Journal of Cancer. 91(3). 498–503. 77 indexed citations

6.

Hirata, T., et al.. (2004). Suppression of Postoperative Systemic Inflammatory Response Syndrome with Clarithromycin following Lung Cancer Surgery. European Surgical Research. 36(1). 13–19. 7 indexed citations

7.

Hirata, T., et al.. (2004). Phase I/II study of weekly docetaxel in patients with previously treated non-small cell lung cancer (NSCLC). Journal of Clinical Oncology. 22(14_suppl). 7243–7243. 2 indexed citations

8.

Hirata, T., et al.. (2004). Phase I/II study of weekly docetaxel in patients with previously treated non-small cell lung cancer (NSCLC). Journal of Clinical Oncology. 22(14_suppl). 7243–7243. 2 indexed citations

9.

Kanno, Tomohiko, Yuka Kanno, Lin‐Feng Chen, et al.. (1998). Intrinsic Transcriptional Activation-Inhibition Domains of the Polyomavirus Enhancer Binding Protein 2/Core Binding Factor α Subunit Revealed in the Presence of the β Subunit. Molecular and Cellular Biology. 18(5). 2444–2454. 176 indexed citations

10.

Bae, Suk-Chul, Ei-ichi Takahashi, E. Ogawa, et al.. (1995). Cloning, mapping and expression of PEBP2αC, a third gene encoding the mammalian Runt domain. Gene. 159(2). 245–248. 146 indexed citations

11.

Lu, Jie, Mitsuo Maruyama, Masanobu Satake, et al.. (1995). Subcellular Localization of the α and β Subunits of the Acute Myeloid Leukemia-Linked Transcription Factor PEBP2/CBF. Molecular and Cellular Biology. 15(3). 1651–1661. 96 indexed citations

12.

Ogawa, E., et al.. (1994). Temperature dependence of the field-stimulated exoelectron emission. Applied Surface Science. 76-77. 21–25. 1 indexed citations

13.

Sakakura, Chohei, Yuko Yamaguchi‐Iwai, Masanobu Satake, et al.. (1994). Growth inhibition and induction of differentiation of t(8;21) acute myeloid leukemia cells by the DNA-binding domain of PEBP2 and the AML1/MTG8(ETO)-specific antisense oligonucleotide.. Proceedings of the National Academy of Sciences. 91(24). 11723–11727. 83 indexed citations

14.

Bae, Suk‐Chul, E. Ogawa, Mitsuo Maruyama, et al.. (1994). PEBP2 alpha B/mouse AML1 consists of multiple isoforms that possess differential transactivation potentials.. Molecular and Cellular Biology. 14(5). 3242–3252. 187 indexed citations

15.

Bae, Suk‐Chul, E. Ogawa, Mitsuo Maruyama, et al.. (1994). PEBP2αB/Mouse AML1 Consists of Multiple Isoforms That Possess Differential Transactivation Potentials. Molecular and Cellular Biology. 14(5). 3242–3252. 51 indexed citations

16.

Ogawa, E., Manabu Inuzuka, Mitsuo Maruyama, et al.. (1993). Molecular Cloning and Characterization of PEBP2β, the Heterodimeric Partner of a Novel Drosophila runt-Related DNA Binding Protein PEBP2α. Virology. 194(1). 314–331. 450 indexed citations

17.

Yamaguchi‐Iwai, Yuko, E. Ogawa, Mitsuo Maruyama, et al.. (1993). Isolation of PEBP2 alpha B cDNA representing the mouse homolog of human acute myeloid leukemia gene, AML1.. PubMed. 8(3). 809–14. 216 indexed citations

18.

Ogawa, E., Masato Maruyama, Hiroshi Kagoshima, et al.. (1993). PEBP2/PEA2 represents a family of transcription factors homologous to the products of the Drosophila runt gene and the human AML1 gene.. Proceedings of the National Academy of Sciences. 90(14). 6859–6863. 548 indexed citations breakdown →

19.

Furukawa, Kazuhiro, Yohei Yamaguchi, E. Ogawa, et al.. (1990). A ubiquitous repressor interacting with an F9 cell-specific silencer and its functional suppression by differentiated cell-specific positive factors.. PubMed. 1(3). 135–47. 42 indexed citations

20.

Kamachi, Yusuke, E. Ogawa, Maki Asano, et al.. (1990). Purification of a mouse nuclear factor that binds to both the A and B cores of the polyomavirus enhancer. Journal of Virology. 64(10). 4808–4819. 195 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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