E Leiberman

618 total citations
18 papers, 462 citations indexed

About

E Leiberman is a scholar working on Endocrinology, Diabetes and Metabolism, Molecular Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, E Leiberman has authored 18 papers receiving a total of 462 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Endocrinology, Diabetes and Metabolism, 10 papers in Molecular Biology and 4 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in E Leiberman's work include Growth Hormone and Insulin-like Growth Factors (10 papers), Sexual Differentiation and Disorders (7 papers) and Lipid metabolism and disorders (4 papers). E Leiberman is often cited by papers focused on Growth Hormone and Insulin-like Growth Factors (10 papers), Sexual Differentiation and Disorders (7 papers) and Lipid metabolism and disorders (4 papers). E Leiberman collaborates with scholars based in Israel, Netherlands and United States. E Leiberman's co-authors include Ze’ev Hochberg, Ariel Rösler, M I New, Perrin C. White, Jakob Dupont, M. Zachmann, Anna Biason‐Lauber, Eli Hershkovitz, Moshe Phillip and O Butenandt and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Clinical Endocrinology & Metabolism and The Journal of Pediatrics.

In The Last Decade

E Leiberman

18 papers receiving 444 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
E Leiberman Israel 11 350 282 98 60 59 18 462
Aya T Nanba United States 14 746 2.1× 257 0.9× 95 1.0× 390 6.5× 46 0.8× 16 816
R. �sterby Sweden 6 125 0.4× 79 0.3× 78 0.8× 62 1.0× 50 0.8× 8 522
Christine Dos Santos France 8 296 0.8× 231 0.8× 231 2.4× 44 0.7× 78 1.3× 10 518
James H. Mersey United States 12 298 0.9× 121 0.4× 36 0.4× 93 1.6× 88 1.5× 24 453
R. Roßkamp Germany 14 413 1.2× 205 0.7× 136 1.4× 193 3.2× 29 0.5× 32 654
Matilde Marchetta Italy 8 112 0.3× 82 0.3× 104 1.1× 36 0.6× 48 0.8× 9 346
Takashi Hamajima Japan 12 118 0.3× 137 0.5× 80 0.8× 41 0.7× 8 0.1× 33 317
Rolf P. Zurbrügg Switzerland 12 108 0.3× 122 0.4× 40 0.4× 33 0.6× 14 0.2× 25 300
María Luisa Ordóñez-Sánchez Mexico 11 142 0.4× 201 0.7× 153 1.6× 140 2.3× 67 1.1× 18 426
Hisanobu Adachi Japan 6 63 0.2× 52 0.2× 25 0.3× 30 0.5× 9 0.2× 20 278

Countries citing papers authored by E Leiberman

Since Specialization
Citations

This map shows the geographic impact of E Leiberman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E Leiberman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E Leiberman more than expected).

Fields of papers citing papers by E Leiberman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E Leiberman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E Leiberman. The network helps show where E Leiberman may publish in the future.

Co-authorship network of co-authors of E Leiberman

This figure shows the co-authorship network connecting the top 25 collaborators of E Leiberman. A scholar is included among the top collaborators of E Leiberman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E Leiberman. E Leiberman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Leiberman, E, et al.. (2004). The Childhood Component of the ICP Model Is Appropriate for Growth Analysis of Short Israeli Children. Hormone Research in Paediatrics. 62(3). 119–123. 3 indexed citations
2.
Abdullatif, Hussein, E Leiberman, Milton R. Brown, Rivka Carmi, & John S. Parks. (2000). Growth Hormone Deficiency Type IB Caused by Cryptic Splicing of the GH-1 Gene. Journal of Pediatric Endocrinology and Metabolism. 13(1). 21–8. 6 indexed citations
3.
Rekers-Mombarg, L.T.M., G. Massa, J.M. Wit, et al.. (1998). Growth hormone therapy with three dosage regimens in children with idiopathic short stature. The Journal of Pediatrics. 132(3). 455–460. 32 indexed citations
4.
Hershkovitz, Eli, et al.. (1998). Increase of serum lipoprotein (a) levels during growth hormone therapy in normal short children. European Journal of Pediatrics. 157(1). 4–7. 10 indexed citations
5.
Phillip, Moshe, et al.. (1998). Once versus twice daily injections of growth hormone in children with idiopathic short stature. Acta Paediatrica. 87(5). 518–520. 7 indexed citations
6.
Phillip, Moshe, et al.. (1998). Once versus twice daily injections of growth hormone in children with idiopathic short stature. Acta Paediatrica. 87(5). 518–520. 10 indexed citations
7.
Leiberman, E, et al.. (1997). Subnormal Cortisol Response to Adrenocorticotropin in Isolated Partial 17,20-Lyase Deficiency. Journal of Pediatric Endocrinology and Metabolism. 10(4). 387–90. 8 indexed citations
8.
Biason‐Lauber, Anna, E Leiberman, & M. Zachmann. (1997). A Single Amino Acid Substitution in the Putative Redox Partner-Binding Site of P450c17 as Cause of Isolated 17,20-Lyase Deficiency1. The Journal of Clinical Endocrinology & Metabolism. 82(11). 3807–3812. 52 indexed citations
9.
Hershkovitz, Eli, et al.. (1996). Short-Term Growth Hormone Therapy Increases Serum Lipoprotein (a) Levels in Normal Short Children without Growth Hormone Deficiency. Hormone Research. 46(1). 38–40. 11 indexed citations
10.
Phillip, Moshe, et al.. (1996). An Early Rise in Urine N-Telopeptide Predicts the Growth Response of Normal Prepubertal Short Children to Growth Hormone Therapy. Journal of Pediatric Endocrinology and Metabolism. 9(5). 519–21. 16 indexed citations
11.
Rekers-Mombarg, L.T.M., J.M. Wit, G. Massa, et al.. (1996). Spontaneous growth in idiopathic short stature. European Study Group.. Archives of Disease in Childhood. 75(3). 175–180. 49 indexed citations
12.
Phillip, Moshe, et al.. (1995). Thyrotropin Secreting Pituitary Adenoma Associated with Hypopituitarism and Diabetes Insipidus in an Adolescent Boy. Journal of Pediatric Endocrinology and Metabolism. 8(1). 47–50. 10 indexed citations
13.
Hershkovitz, Eli, Shlomit Shalitin, Jonathan M. Levy, et al.. (1995). The new syndrome of congenital hypoparathyroidism associated with dysmorphism, growth retardation, and developmental delay--a report of six patients.. PubMed. 31(5). 293–7. 13 indexed citations
14.
Hershkovitz, Eli, et al.. (1995). High prevalence of thyroxine-binding globulin deficiency among Bedouin infants in southern Israel.. PubMed. 31(8). 500–2. 3 indexed citations
15.
Zadik, Zvi, Stuart A. Chalew, Amnon Zung, et al.. (1994). Effect of long-term growth hormone therapy on bone age and pubertal maturation in boys with and without classic growth hormone deficiency. The Journal of Pediatrics. 125(2). 189–195. 41 indexed citations
16.
Gedalia, Abraham, E Leiberman, Matitiahu Lifshitz, et al.. (1993). Thyroid Involvement in Children With Familial Mediterranean Fever. Clinical Pediatrics. 32(6). 357–359. 4 indexed citations
17.
White, Perrin C., Jakob Dupont, M I New, et al.. (1991). A mutation in CYP11B1 (Arg-448----His) associated with steroid 11 beta-hydroxylase deficiency in Jews of Moroccan origin.. Journal of Clinical Investigation. 87(5). 1664–1667. 183 indexed citations
18.
Leiberman, E, et al.. (1985). Steroid receptors in a granulosa cell tumor (GCT) in an infant with precocious puberty. Medical and Pediatric Oncology. 13(6). 370–374. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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