E. H. Weiss

2.1k total citations
34 papers, 1.8k citations indexed

About

E. H. Weiss is a scholar working on Immunology, Molecular Biology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, E. H. Weiss has authored 34 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Immunology, 8 papers in Molecular Biology and 6 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in E. H. Weiss's work include T-cell and B-cell Immunology (10 papers), Immune Cell Function and Interaction (7 papers) and Monoclonal and Polyclonal Antibodies Research (6 papers). E. H. Weiss is often cited by papers focused on T-cell and B-cell Immunology (10 papers), Immune Cell Function and Interaction (7 papers) and Monoclonal and Polyclonal Antibodies Research (6 papers). E. H. Weiss collaborates with scholars based in Germany, United States and United Kingdom. E. H. Weiss's co-authors include Richard A. Flavell, Andrew L. Mellor, L. Golden, Karen Fahrner, Gerald Messer, G. Riethmüller, Ulrich Spengler, M. Jung, G. R. Pape and Sune Kvist and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

E. H. Weiss

34 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
E. H. Weiss Germany 17 1.1k 535 284 194 176 34 1.8k
John P. Atkinson United States 20 2.3k 2.0× 447 0.8× 261 0.9× 209 1.1× 590 3.4× 23 3.1k
R. Philip Custer United States 16 969 0.9× 713 1.3× 390 1.4× 235 1.2× 278 1.6× 23 2.4k
Leo P. de Waal Netherlands 23 927 0.8× 385 0.7× 203 0.7× 125 0.6× 232 1.3× 66 1.9k
A. S. Daar Oman 15 754 0.7× 304 0.6× 113 0.4× 175 0.9× 188 1.1× 41 1.6k
David Stephany United States 21 1.7k 1.5× 404 0.8× 141 0.5× 400 2.1× 287 1.6× 35 2.6k
V. Keijsers Netherlands 14 1.1k 1.0× 276 0.5× 106 0.4× 96 0.5× 292 1.7× 16 1.7k
Anthony B. Troutt United States 23 1.2k 1.1× 705 1.3× 137 0.5× 53 0.3× 237 1.3× 33 2.3k
José R. Regueiro Spain 25 1.4k 1.3× 366 0.7× 251 0.9× 85 0.4× 291 1.7× 108 2.0k
Akemi Wakisaka Japan 26 1.0k 0.9× 972 1.8× 289 1.0× 482 2.5× 164 0.9× 97 2.6k
Giovanni Salvatori Italy 24 1.5k 1.3× 1.0k 1.9× 270 1.0× 61 0.3× 171 1.0× 32 2.5k

Countries citing papers authored by E. H. Weiss

Since Specialization
Citations

This map shows the geographic impact of E. H. Weiss's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by E. H. Weiss with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites E. H. Weiss more than expected).

Fields of papers citing papers by E. H. Weiss

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by E. H. Weiss. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by E. H. Weiss. The network helps show where E. H. Weiss may publish in the future.

Co-authorship network of co-authors of E. H. Weiss

This figure shows the co-authorship network connecting the top 25 collaborators of E. H. Weiss. A scholar is included among the top collaborators of E. H. Weiss based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with E. H. Weiss. E. H. Weiss is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zareie, Pirooz, E. H. Weiss, Daniel H. Kaplan, & Laura K. Mackay. (2025). Cutaneous T cell immunity. Nature Immunology. 26(7). 1014–1022. 2 indexed citations
2.
Kopp, R., et al.. (2009). Association of MICA-TM and MICB C1_2_A Microsatellite Polymorphisms with Tumor Progression in Patients with Colorectal Cancer. Journal of Clinical Immunology. 29(4). 545–554. 18 indexed citations
3.
Wetzke, Martin, et al.. (2008). MICA*055: a new allele with eight GCT repeats in the exon 5 microsatellite. Tissue Antigens. 72(4). 410–411. 2 indexed citations
4.
Török, Helga‐Paula, Martin Wetzke, Vanessa Beynon, et al.. (2007). The 14-bp deletion polymorphism in the HLA-G gene displays significant differences between ulcerative colitis and Crohn's disease and is associated with ileocecal resection in Crohn's disease. International Immunology. 19(5). 621–626. 41 indexed citations
5.
Glas, Jürgen, Kathleen A. Martin, R. Kopp, et al.. (2001). MICA, MICB and C1_4_1 polymorphism in Crohn’s disease and ulcerative colitis. Tissue Antigens. 58(4). 243–249. 45 indexed citations
6.
Witter, K., et al.. (2001). Linkage disequilibria between HLA‐B, C1_4_1, MICA and MICB. Tissue Antigens. 58(6). 411–418. 14 indexed citations
7.
Rebmann, Vera, Kerstin Pfeiffer, Soldano Ferrone, et al.. (1999). Detection of soluble HLA‐G molecules in plasma and amniotic fluid. Tissue Antigens. 53(1). 14–22. 165 indexed citations
8.
Kipar, Anja, et al.. (1998). Expression of major histocompatibility complex class II antigen in neoplastic cells of canine cutaneous histiocytoma. Veterinary Immunology and Immunopathology. 62(1). 1–13. 21 indexed citations
9.
Weiss, E. H. & Astrid Cannich. (1998). The genomic organization of the human factor H gene family. Molecular Immunology. 35(6-7). 411–411. 1 indexed citations
10.
Kirveskari, Juha, H. Kellner, Maarit Wuorela, et al.. (1997). False-negative serological HLA-B27 typing results may be due to altered antigenic epitopes and can be detected by polymerase chain reaction. Lara D. Veeken. 36(2). 185–189. 39 indexed citations
11.
Ulbrecht, Matthias, Matthias Wjst, Joachim Heinrich, et al.. (1997). High serum IgE concentrations: Association with HLA-DR and markers on chromosome 5q31 and chromosome 11q13. Journal of Allergy and Clinical Immunology. 99(6). 828–836. 27 indexed citations
12.
Frankenberger, Bernhard, Sabine Scholz, E. Keller, et al.. (1997). Routine molecular genotyping of HLA-B27 in spondyloarthropathies overcomes the obstacles of serological typing and reveals an increased B *2702 frequency in ankylosing spondylitis.. PubMed. 24(5). 899–903. 12 indexed citations
13.
Nagata, Toshi, E. H. Weiss, Kuniya Abe, et al.. (1995). Physical mapping of the retinoid X receptorB gene in mouse and human. Immunogenetics. 41(2-3). 83–90. 20 indexed citations
14.
Zwirner, Jörg, Winfríed Weissenhorn, Lars Karlsson, et al.. (1992). Expression of a functional chimeric Ig-MHC class II protein. The Journal of Immunology. 148(1). 272–276. 1 indexed citations
15.
Messer, Gerald, Jacqueline Zemmour, Harry T. Orr, et al.. (1992). HLA-J, a second inactivated class I HLA gene related to HLA-G and HLA-A. Implications for the evolution of the HLA-A-related genes. The Journal of Immunology. 148(12). 4043–4053. 55 indexed citations
18.
Viitanen, A M, et al.. (1988). Lack of hybridization between Yersinia enterocolitica and HLA-B27 DNA. PubMed. 15(7). 1123–5. 1 indexed citations
19.
Weiss, E. H., Andrew L. Mellor, L. Golden, et al.. (1983). Structure and Expression of Class I Genes of the Mouse Major Histocompatibility Complex. Hämatologie und Bluttransfusion. 28. 454–458. 1 indexed citations
20.
Bullman, H., Meinrad Busslinger, E. deBoer, et al.. (1982). The structure and expression of mammalian gene clusters.. PubMed. 103 Pt A. 37–55. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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