Douglas W. Allan

2.0k total citations
43 papers, 1.4k citations indexed

About

Douglas W. Allan is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Surgery. According to data from OpenAlex, Douglas W. Allan has authored 43 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Cellular and Molecular Neuroscience, 19 papers in Molecular Biology and 9 papers in Surgery. Recurrent topics in Douglas W. Allan's work include Neurobiology and Insect Physiology Research (17 papers), Developmental Biology and Gene Regulation (9 papers) and Congenital Diaphragmatic Hernia Studies (8 papers). Douglas W. Allan is often cited by papers focused on Neurobiology and Insect Physiology Research (17 papers), Developmental Biology and Gene Regulation (9 papers) and Congenital Diaphragmatic Hernia Studies (8 papers). Douglas W. Allan collaborates with scholars based in Canada, United States and United Kingdom. Douglas W. Allan's co-authors include John J. Greer, Stefan Thor, Irene Miguel‐Aliaga, Susan E. St. Pierre, Randal P. Babiuk, Robert P. Lemke, Miguel Martin‐Caraballo, Wei Zhang, Robin D. Clugston and Kevin Eade and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Douglas W. Allan

40 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Douglas W. Allan Canada 22 659 456 398 344 172 43 1.4k
J. Hikke van Doorninck Netherlands 15 971 1.5× 328 0.7× 95 0.2× 333 1.0× 65 0.4× 17 1.8k
Laurent Soustelle France 20 420 0.6× 467 1.0× 121 0.3× 117 0.3× 28 0.2× 42 1.0k
Jordane Malaterre Australia 24 835 1.3× 318 0.7× 120 0.3× 78 0.2× 40 0.2× 41 1.5k
Shanru Li United States 20 812 1.2× 80 0.2× 192 0.5× 258 0.8× 45 0.3× 26 1.4k
Jennifer Skidmore United States 20 651 1.0× 198 0.4× 130 0.3× 122 0.4× 37 0.2× 30 1.1k
Felicitas Pröls Germany 23 860 1.3× 223 0.5× 120 0.3× 54 0.2× 36 0.2× 55 1.5k
Yuki Sato Japan 23 947 1.4× 506 1.1× 124 0.3× 43 0.1× 61 0.4× 65 1.8k
Yoko Kameda Japan 27 872 1.3× 500 1.1× 253 0.6× 126 0.4× 481 2.8× 118 2.0k
Aı̈da Metzenberg United States 16 660 1.0× 230 0.5× 70 0.2× 105 0.3× 25 0.1× 19 1.2k
Annie Meiniel France 24 753 1.1× 675 1.5× 66 0.2× 52 0.2× 159 0.9× 74 1.4k

Countries citing papers authored by Douglas W. Allan

Since Specialization
Citations

This map shows the geographic impact of Douglas W. Allan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Douglas W. Allan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Douglas W. Allan more than expected).

Fields of papers citing papers by Douglas W. Allan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Douglas W. Allan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Douglas W. Allan. The network helps show where Douglas W. Allan may publish in the future.

Co-authorship network of co-authors of Douglas W. Allan

This figure shows the co-authorship network connecting the top 25 collaborators of Douglas W. Allan. A scholar is included among the top collaborators of Douglas W. Allan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Douglas W. Allan. Douglas W. Allan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
McIntosh, Graeme H., Barry P. Young, Tianshun Lian, et al.. (2025). Revealing function-altering MECP2 mutations in individuals with autism spectrum disorder using yeast and Drosophila. Genetics. 231(1).
2.
Allan, Douglas W., et al.. (2023). Adult expression of Semaphorins and Plexins is essential for motor neuron survival. Scientific Reports. 13(1). 5894–5894.
5.
Losada‐Pérez, María, et al.. (2018). Segmentally homologous neurons acquire two different terminal neuropeptidergic fates in the Drosophila nervous system. PLoS ONE. 13(4). e0194281–e0194281. 7 indexed citations
6.
Tang, Jonathan C. Y., et al.. (2015). Gene Regulatory Mechanisms Underlying the Spatial and Temporal Regulation of Target-Dependent Gene Expression in Drosophila Neurons. PLoS Genetics. 11(12). e1005754–e1005754. 8 indexed citations
7.
Cowan, Catherine M., Shmma Quraishe, Sarah Hands, et al.. (2015). Rescue from tau-induced neuronal dysfunction produces insoluble tau oligomers. Scientific Reports. 5(1). 17191–17191. 32 indexed citations
8.
Bejarano, Fernando, Piero Sanfilippo, David M. Tyler, et al.. (2014). Homeotic Function of Drosophila Bithorax-Complex miRNAs Mediates Fertility by Restricting Multiple Hox Genes and TALE Cofactors in the CNS. Developmental Cell. 29(6). 635–648. 36 indexed citations
9.
Tang, Jonathan C. Y., et al.. (2013). Female-biased dimorphism underlies a female-specific role for post-embryonic Ilp7 neurons inDrosophilafertility. Development. 140(18). 3915–3926. 36 indexed citations
10.
Allan, Douglas W., et al.. (2013). Subtype-specific neuronal remodeling duringDrosophilametamorphosis. Fly. 7(2). 78–86. 18 indexed citations
11.
Cowan, Catherine M., et al.. (2011). Modelling Tauopathies in Drosophila: Insights from the Fruit Fly. International Journal of Alzheimer s Disease. 2011(1). 598157–598157. 17 indexed citations
12.
Eade, Kevin & Douglas W. Allan. (2009). Neuronal Phenotype in the Mature Nervous System Is Maintained by Persistent Retrograde Bone Morphogenetic Protein Signaling. Journal of Neuroscience. 29(12). 3852–3864. 24 indexed citations
13.
Greer, John J., Douglas W. Allan, Randal P. Babiuk, & Robin D. Clugston. (2005). INSIGHTS INTO THE PATHOGENESIS AND AETIOLOGY OF CONGENITAL DIAPHRAGMATIC HERNIA FROM RODENT MODELS. Fetal and Maternal Medicine Review. 16(3). 211–220. 2 indexed citations
14.
Allan, Douglas W., Susan E. St. Pierre, Irene Miguel‐Aliaga, & Stefan Thor. (2003). Specification of Neuropeptide Cell Identity by the Integration of Retrograde BMP Signaling and a Combinatorial Transcription Factor Code. Cell. 113(1). 73–86. 140 indexed citations
15.
Allan, Douglas W. & Stefan Thor. (2003). Together at Last. Neuron. 38(5). 675–677. 21 indexed citations
16.
Allan, Douglas W. & John J. Greer. (1997). Pathogenesis of nitrofen-induced congenital diaphragmatic hernia in fetal rats. Journal of Applied Physiology. 83(2). 338–347. 91 indexed citations
17.
Allan, Douglas W. & John J. Greer. (1997). Development of phrenic motoneuron morphology in the fetal rat. The Journal of Comparative Neurology. 382(4). 469–479. 44 indexed citations
18.
Allan, Douglas W. & John J. Greer. (1997). Embryogenesis of the phrenic nerve and diaphragm in the fetal rat. The Journal of Comparative Neurology. 382(4). 459–468. 83 indexed citations
19.
Greer, John J., et al.. (1996). Respiratory rhythm generation in a precocial rodent in vitro preparation. Respiration Physiology. 103(2). 105–112. 14 indexed citations
20.
Stewart, Randall R., Douglas W. Allan, & Colin McCaig. (1996). Lectins implicate specific carbohydrate domains in electric field stimulated nerve growth and guidance. Journal of Neurobiology. 30(3). 425–437. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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