Douglas E. Brash

14.6k total citations · 6 hit papers
96 papers, 10.3k citations indexed

About

Douglas E. Brash is a scholar working on Molecular Biology, Oncology and Dermatology. According to data from OpenAlex, Douglas E. Brash has authored 96 papers receiving a total of 10.3k indexed citations (citations by other indexed papers that have themselves been cited), including 61 papers in Molecular Biology, 27 papers in Oncology and 19 papers in Dermatology. Recurrent topics in Douglas E. Brash's work include Cancer-related Molecular Pathways (22 papers), DNA Repair Mechanisms (20 papers) and Skin Protection and Aging (16 papers). Douglas E. Brash is often cited by papers focused on Cancer-related Molecular Pathways (22 papers), DNA Repair Mechanisms (20 papers) and Skin Protection and Aging (16 papers). Douglas E. Brash collaborates with scholars based in United States, United Kingdom and Sweden. Douglas E. Brash's co-authors include William A. Haseltine, Alan S. Jonason, Annemarie Ziegler, David J. Leffell, Howard P. Baden, Amy Lin, Greg J. McKenna, Jeffrey A. Rudolph, Alan J. Halperin and Jeffrey A. Simon and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Douglas E. Brash

92 papers receiving 10.0k citations

Hit Papers

A role for sunlight in skin cancer: UV-induced p53 mutati... 1988 2026 2000 2013 1991 1994 1988 1996 1996 500 1000 1.5k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Douglas E. Brash United States 44 5.7k 2.9k 2.9k 1.6k 1.5k 96 10.3k
Kenneth H. Kraemer United States 57 9.4k 1.7× 2.1k 0.7× 2.7k 0.9× 3.3k 2.0× 1.2k 0.8× 196 12.9k
Peter G. Parsons Australia 51 4.4k 0.8× 1.6k 0.5× 2.3k 0.8× 1.0k 0.6× 851 0.6× 268 9.0k
Alain Sarasin France 61 9.5k 1.7× 1.0k 0.4× 2.9k 1.0× 3.0k 1.8× 842 0.6× 282 12.0k
Margaret L. Kripke United States 59 2.7k 0.5× 5.0k 1.7× 3.1k 1.1× 1.5k 0.9× 1.5k 1.0× 210 11.6k
Frans C. S. Ramaekers Netherlands 58 7.1k 1.3× 507 0.2× 2.4k 0.8× 1.1k 0.7× 1.6k 1.1× 240 13.4k
Wim Declercq Belgium 65 10.3k 1.8× 1.1k 0.4× 1.9k 0.7× 2.1k 1.3× 2.7k 1.8× 133 16.0k
Glenn Merlino United States 73 10.9k 1.9× 914 0.3× 6.1k 2.1× 2.0k 1.2× 1.0k 0.7× 215 17.9k
James E. Cleaver United States 63 11.8k 2.1× 921 0.3× 2.7k 0.9× 4.8k 2.9× 523 0.3× 257 14.5k
Toshio Mori Japan 44 4.3k 0.8× 867 0.3× 1.2k 0.4× 950 0.6× 393 0.3× 238 7.6k
Stuart H. Yuspa United States 83 13.6k 2.4× 2.5k 0.9× 4.5k 1.6× 3.1k 1.9× 1.1k 0.7× 328 23.4k

Countries citing papers authored by Douglas E. Brash

Since Specialization
Citations

This map shows the geographic impact of Douglas E. Brash's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Douglas E. Brash with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Douglas E. Brash more than expected).

Fields of papers citing papers by Douglas E. Brash

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Douglas E. Brash. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Douglas E. Brash. The network helps show where Douglas E. Brash may publish in the future.

Co-authorship network of co-authors of Douglas E. Brash

This figure shows the co-authorship network connecting the top 25 collaborators of Douglas E. Brash. A scholar is included among the top collaborators of Douglas E. Brash based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Douglas E. Brash. Douglas E. Brash is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Brash, Douglas E. & Elizabeth R. Gaillard. (2024). Chemiexcitation in preventing macular degeneration. SHILAP Revista de lepidopterología. 5.
2.
Menon, Vijay & Douglas E. Brash. (2023). Next-generation sequencing methodologies to detect low-frequency mutations: “Catch me if you can”. Mutation Research/Reviews in Mutation Research. 792. 108471–108471. 18 indexed citations
3.
Brash, Douglas E. & Letícia Christina Pires Gonçalves. (2023). Chemiexcitation: Mammalian Photochemistry in the Dark. Photochemistry and Photobiology. 99(2). 251–276. 9 indexed citations
4.
Leachman, Sancy A., Thomas J. Hornyak, Gregory S. Barsh, et al.. (2019). Melanoma to Vitiligo: The Melanocyte in Biology & Medicine–Joint Montagna Symposium on the Biology of Skin/PanAmerican Society for Pigment Cell Research Annual Meeting. Journal of Investigative Dermatology. 140(2). 269–274. 4 indexed citations
5.
Brash, Douglas E., Letícia Christina Pires Gonçalves, & Etelvino José Henriques Bechara. (2018). Chemiexcitation and Its Implications for Disease. Trends in Molecular Medicine. 24(6). 527–541. 25 indexed citations
6.
7.
Rochette, Patrick J., Sandrine Lacoste, Jean‐Philippe Therrien, et al.. (2009). Influence of cytosine methylation on ultraviolet-induced cyclobutane pyrimidine dimer formation in genomic DNA. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 665(1-2). 7–13. 48 indexed citations
8.
Chao, Dennis L., et al.. (2008). Preneoplastic lesion growth driven by the death of adjacent normal stem cells. Proceedings of the National Academy of Sciences. 105(39). 15034–15039. 28 indexed citations
9.
Asplund, Anna, Anna Gustafsson, Norbert Wikonkál, et al.. (2005). PTCH codon 1315 polymorphism and risk for nonmelanoma skin cancer. British Journal of Dermatology. 152(5). 868–873. 25 indexed citations
10.
Chakraborty, Ashok K., Rossitza Lazova, Stella M. Davies, et al.. (2004). Genetic evidence for tumor-hematopoietic cell hybrids in a human renal cell carcinoma metastasis. Cancer Research. 64. 407–407. 1 indexed citations
11.
Brash, Douglas E., et al.. (1998). Skin precancer.. PubMed. 32. 69–113. 13 indexed citations
12.
Brash, Douglas E.. (1997). Sunlight and the onset of skin cancer. Trends in Genetics. 13(10). 410–414. 246 indexed citations
13.
Gailani, Mae R., Mona Ståhle‐Bäckdahl, David J. Leffell, et al.. (1996). The role of the human homologue of Drosophila patched in sporadic basal cell carcinomas. Nature Genetics. 14(1). 78–81. 593 indexed citations breakdown →
14.
Gailani, Mae R., David J. Leffell, Annemarie Ziegler, et al.. (1996). Relationship Between Sunlight Exposure and a Key Genetic Alteration in Basal Cell Carcinoma. JNCI Journal of the National Cancer Institute. 88(6). 349–354. 145 indexed citations
15.
Jonason, Alan S., Subrahmanyam Kunala, Richard J. Restifo, et al.. (1996). Frequent clones of p53-mutated keratinocytes in normal human skin. Proceedings of the National Academy of Sciences. 93(24). 14025–14029. 504 indexed citations breakdown →
16.
Mitchell, David L., Douglas E. Brash, & Rodney S. Nairn. (1990). Rapid repair kinetics of pyrimidine(6–4)pyrimidone photoproducts in human cells are due to excision rather than conformational change. Nucleic Acids Research. 18(4). 963–971. 74 indexed citations
17.
Brash, Douglas E.. (1988). UV MUTAGENIC PHOTOPRODUCTS IN Escherichia coli AND HUMAN CELLS: A MOLECULAR GENETICS PERSPECTIVE ON HUMAN SKIN CANCER*. Photochemistry and Photobiology. 48(1). 59–66. 198 indexed citations
18.
Brash, Douglas E., George E. Mark, Michael Farrell, & Curtis C. Harris. (1987). Overview of human cells in genetic research: Altered phenotypes in human cells caused by transferred genes. Somatic Cell and Molecular Genetics. 13(4). 429–440. 9 indexed citations
20.
Brash, Douglas E.. (1979). A method for quantitative measurement of DNA damage and repair in vivo /. OhioLink ETD Center (Ohio Library and Information Network). 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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