Douglas C. Weiser

1.1k total citations
17 papers, 869 citations indexed

About

Douglas C. Weiser is a scholar working on Molecular Biology, Cell Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Douglas C. Weiser has authored 17 papers receiving a total of 869 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 12 papers in Cell Biology and 3 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Douglas C. Weiser's work include Cellular Mechanics and Interactions (6 papers), Microtubule and mitosis dynamics (4 papers) and RNA regulation and disease (3 papers). Douglas C. Weiser is often cited by papers focused on Cellular Mechanics and Interactions (6 papers), Microtubule and mitosis dynamics (4 papers) and RNA regulation and disease (3 papers). Douglas C. Weiser collaborates with scholars based in United States. Douglas C. Weiser's co-authors include Shirish Shenolikar, Matthew Brush, Shi Li, David Kimelman, John M. Hallenbeck, John H. Connor, Richard H. Row, Ujwal J. Pyati, Ryan T. Terry-Lorenzo and Todd D. Prickett and has published in prestigious journals such as Journal of Biological Chemistry, Genes & Development and PLoS ONE.

In The Last Decade

Douglas C. Weiser

17 papers receiving 859 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Douglas C. Weiser United States	12	603	487	183	61	60	17	869
Canhong Cao United States	10	592 1.0×	403 0.8×	94 0.5×	79 1.3×	49 0.8×	11	876
Sara K. Young United States	16	607 1.0×	215 0.4×	134 0.7×	58 1.0×	73 1.2×	25	878
Stephen J. Terry United Kingdom	13	498 0.8×	292 0.6×	145 0.8×	55 0.9×	55 0.9×	16	894
Heide Plesken United States	9	701 1.2×	248 0.5×	143 0.8×	83 1.4×	90 1.5×	9	1.0k
Abel R. Alcázar-Román United States	13	772 1.3×	194 0.4×	129 0.7×	38 0.6×	56 0.9×	17	985
Verena Arndt Germany	8	764 1.3×	371 0.8×	269 1.5×	51 0.8×	20 0.3×	8	1.0k
Anna McGeachy United States	5	1.3k 2.1×	231 0.5×	113 0.6×	100 1.6×	32 0.5×	8	1.5k
Bertrand Kleizen Netherlands	13	652 1.1×	336 0.7×	112 0.6×	96 1.6×	44 0.7×	17	1.0k
Krupa Pattni United Kingdom	8	389 0.6×	453 0.9×	114 0.6×	85 1.4×	65 1.1×	9	764

Countries citing papers authored by Douglas C. Weiser

Since Specialization

Citations

This map shows the geographic impact of Douglas C. Weiser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Douglas C. Weiser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Douglas C. Weiser more than expected).

Fields of papers citing papers by Douglas C. Weiser

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Douglas C. Weiser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Douglas C. Weiser. The network helps show where Douglas C. Weiser may publish in the future.

Co-authorship network of co-authors of Douglas C. Weiser

This figure shows the co-authorship network connecting the top 25 collaborators of Douglas C. Weiser. A scholar is included among the top collaborators of Douglas C. Weiser based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Douglas C. Weiser. Douglas C. Weiser is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Wrischnik, Lisa A., et al.. (2023). The PPP1R15 Family of eIF2-alpha Phosphatase Targeting Subunits (GADD34 and CReP). International Journal of Molecular Sciences. 24(24). 17321–17321. 18 indexed citations

Lang, Iréne, et al.. (2020). The Evolution of Duplicated Genes of the Cpi-17/Phi-1 (ppp1r14) Family of Protein Phosphatase 1 Inhibitors in Teleosts. International Journal of Molecular Sciences. 21(16). 5709–5709. 12 indexed citations

Lang, Iréne, et al.. (2018). Alternative splicing of (ppp1r12a/mypt1) in zebrafish produces a novel myosin phosphatase targeting subunit. Gene. 675. 15–26. 2 indexed citations

Weiser, Douglas C., et al.. (2015). Characterization of the three zebrafish orthologs of the mitochondrial GTPase Miro/Rhot. Comparative Biochemistry and Physiology Part B Biochemistry and Molecular Biology. 191. 126–134. 6 indexed citations

Jayashankar, Vaishali, et al.. (2013). Protein Phosphatase 1 β Paralogs Encode the Zebrafish Myosin Phosphatase Catalytic Subunit. PLoS ONE. 8(9). e75766–e75766. 15 indexed citations

Weiser, Douglas C. & David Kimelman. (2011). Analysis of Cell Shape and Polarity During Zebrafish Gastrulation. Methods in molecular biology. 839. 53–68. 9 indexed citations

Landsverk, Megan, Douglas C. Weiser, Mark Hannibal, & David Kimelman. (2010). Alternative Splicing of sept9a and sept9b in Zebrafish Produces Multiple mRNA Transcripts Expressed Throughout Development. PLoS ONE. 5(5). e10712–e10712. 11 indexed citations

Weiser, Douglas C., Richard H. Row, & David Kimelman. (2009). Rho-regulated Myosin phosphatase establishes the level of protrusive activity required for cell movements during zebrafish gastrulation. Development. 136(14). 2375–2384. 61 indexed citations

Weiser, Douglas C., et al.. (2008). Cell shape regulation by Gravin requires N-terminal membrane effector domains. Biochemical and Biophysical Research Communications. 375(4). 512–516. 5 indexed citations

10.

Weiser, Douglas C., Ujwal J. Pyati, & David Kimelman. (2007). Gravin regulates mesodermal cell behavior changes required for axis elongation during zebrafish gastrulation. Genes & Development. 21(12). 1559–1571. 50 indexed citations

11.

Weiser, Douglas C., et al.. (2005). Importance of a Surface Hydrophobic Pocket on Protein Phosphatase-1 Catalytic Subunit in Recognizing Cellular Regulators. Journal of Biological Chemistry. 280(16). 15903–15911. 30 indexed citations

12.

Weiser, Douglas C., et al.. (2004). The Inhibitor-1 C Terminus Facilitates Hormonal Regulation of Cellular Protein Phosphatase-1. Journal of Biological Chemistry. 279(47). 48904–48914. 16 indexed citations

13.

Weiser, Douglas C. & Shirish Shenolikar. (2003). Use of Protein Phosphatase Inhibitors. Current Protocols in Protein Science. 31(1). Unit 13.10–Unit 13.10. 16 indexed citations

14.

Brush, Matthew, Douglas C. Weiser, & Shirish Shenolikar. (2003). Growth Arrest and DNA Damage-Inducible Protein GADD34 Targets Protein Phosphatase 1α to the Endoplasmic Reticulum and Promotes Dephosphorylation of the α Subunit of Eukaryotic Translation Initiation Factor 2. Molecular and Cellular Biology. 23(4). 1292–1303. 315 indexed citations

15.

Weiser, Douglas C. & Shirish Shenolikar. (2003). Use of Protein Phosphatase Inhibitors. Current Protocols in Molecular Biology. 62(1). 9 indexed citations

16.

Terry-Lorenzo, Ryan T., et al.. (2002). Neurabins Recruit Protein Phosphatase-1 and Inhibitor-2 to the Actin Cytoskeleton. Journal of Biological Chemistry. 277(48). 46535–46543. 67 indexed citations

17.

Connor, John H., Douglas C. Weiser, Shi Li, John M. Hallenbeck, & Shirish Shenolikar. (2001). Growth Arrest and DNA Damage-Inducible Protein GADD34 Assembles a Novel Signaling Complex Containing Protein Phosphatase 1 and Inhibitor 1. Molecular and Cellular Biology. 21(20). 6841–6850. 227 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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