Doris K. Wu

7.2k total citations
63 papers, 4.9k citations indexed

About

Doris K. Wu is a scholar working on Molecular Biology, Sensory Systems and Cellular and Molecular Neuroscience. According to data from OpenAlex, Doris K. Wu has authored 63 papers receiving a total of 4.9k indexed citations (citations by other indexed papers that have themselves been cited), including 50 papers in Molecular Biology, 30 papers in Sensory Systems and 12 papers in Cellular and Molecular Neuroscience. Recurrent topics in Doris K. Wu's work include Developmental Biology and Gene Regulation (38 papers), Hearing, Cochlea, Tinnitus, Genetics (30 papers) and Congenital heart defects research (18 papers). Doris K. Wu is often cited by papers focused on Developmental Biology and Gene Regulation (38 papers), Hearing, Cochlea, Tinnitus, Genetics (30 papers) and Congenital heart defects research (18 papers). Doris K. Wu collaborates with scholars based in United States, South Korea and Japan. Doris K. Wu's co-authors include Daniel Choo, Weise Chang, Donna M. Fekete, Seung Ha Oh, Randy L. Johnson, Jinwoong Bok, Allen F. Ryan, Matthew W. Kelley, Antonio Simeone and Fábio Daumas Nunes and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

In The Last Decade

Doris K. Wu

63 papers receiving 4.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Doris K. Wu United States 36 3.1k 2.8k 745 645 486 63 4.9k
Andrew K. Groves United States 44 4.3k 1.4× 3.0k 1.1× 679 0.9× 403 0.6× 879 1.8× 104 7.0k
Matthew W. Kelley United States 37 2.3k 0.7× 2.8k 1.0× 554 0.7× 314 0.5× 249 0.5× 70 4.6k
Matthew W. Kelley United States 36 3.2k 1.0× 1.9k 0.7× 342 0.5× 372 0.6× 624 1.3× 69 4.9k
Neil Segil United States 37 2.7k 0.9× 2.8k 1.0× 570 0.8× 226 0.4× 263 0.5× 59 5.0k
Douglas A. Cotanche United States 36 1.3k 0.4× 3.2k 1.2× 888 1.2× 502 0.8× 218 0.4× 75 4.2k
Andrew J. Griffith United States 41 3.6k 1.1× 4.3k 1.6× 240 0.3× 1.8k 2.7× 249 0.5× 117 6.8k
Karen P. Steel United Kingdom 52 6.7k 2.1× 5.9k 2.1× 459 0.6× 1.6k 2.4× 819 1.7× 183 11.2k
Fernando Giráldez Spain 34 2.1k 0.7× 1.5k 0.5× 405 0.5× 139 0.2× 684 1.4× 81 3.4k
Ruth Anne Eatock United States 31 1.8k 0.6× 2.9k 1.1× 505 0.7× 1.2k 1.9× 551 1.1× 57 4.1k
Richard J. Goodyear United Kingdom 41 1.9k 0.6× 3.3k 1.2× 321 0.4× 1.0k 1.6× 205 0.4× 72 4.5k

Countries citing papers authored by Doris K. Wu

Since Specialization
Citations

This map shows the geographic impact of Doris K. Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Doris K. Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Doris K. Wu more than expected).

Fields of papers citing papers by Doris K. Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Doris K. Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Doris K. Wu. The network helps show where Doris K. Wu may publish in the future.

Co-authorship network of co-authors of Doris K. Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Doris K. Wu. A scholar is included among the top collaborators of Doris K. Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Doris K. Wu. Doris K. Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wu, Doris K., et al.. (2016). Temporal coupling between specifications of neuronal and macular fates of the inner ear. Developmental Biology. 414(1). 21–33. 4 indexed citations
2.
Bok, Jinwoong, et al.. (2013). Auditory ganglion source of Sonic hedgehog regulates timing of cell cycle exit and differentiation of mammalian cochlear hair cells. Proceedings of the National Academy of Sciences. 110(34). 13869–13874. 78 indexed citations
3.
Uchikawa, Masanori, et al.. (2013). Progression of Neurogenesis in the Inner Ear Requires Inhibition ofSox2Transcription by Neurogenin1 and Neurod1. Journal of Neuroscience. 33(9). 3879–3890. 59 indexed citations
4.
Sachdev, Shrikesh, Trupti Joshi, Doris K. Wu, et al.. (2012). Elongation Factor 1 alpha1 and Genes Associated with Usher Syndromes Are Downstream Targets of GBX2. PLoS ONE. 7(11). e47366–e47366. 12 indexed citations
5.
Bok, Jinwoong, et al.. (2010). Transient retinoic acid signaling confers anterior-posterior polarity to the inner ear. Proceedings of the National Academy of Sciences. 108(1). 161–166. 71 indexed citations
6.
Koo, Soo Kyung, et al.. (2009). Lmx1a maintains proper neurogenic, sensory, and non-sensory domains in the mammalian inner ear. Developmental Biology. 333(1). 14–25. 64 indexed citations
7.
Bok, Jinwoong, et al.. (2009). Distinct contributions from the hindbrain and mesenchyme to inner ear morphogenesis. Developmental Biology. 337(2). 324–334. 19 indexed citations
8.
Hwang, Chan Ho, Antonio Simeone, Eseng Lai, & Doris K. Wu. (2009). Foxg1 is required for proper separation and formation of sensory cristae during inner ear development. Developmental Dynamics. 238(11). 2725–2734. 48 indexed citations
9.
Bok, Jinwoong, et al.. (2007). Opposing gradients of Gli repressor and activators mediate Shh signaling along the dorsoventral axis of the inner ear. Development. 134(9). 1713–1722. 90 indexed citations
10.
Bok, Jinwoong, et al.. (2007). Role of hindbrain in inner ear morphogenesis: Analysis of Noggin knockout mice. Developmental Biology. 311(1). 69–78. 19 indexed citations
11.
Cole, Laura, et al.. (2004). The role of Pax2 in mouse inner ear development. Developmental Biology. 272(1). 161–175. 137 indexed citations
12.
Pereira, Fred A., et al.. (2002). The Nuclear Receptor Nor-1 Is Essential for Proliferation of the Semicircular Canals of the Mouse Inner Ear. Molecular and Cellular Biology. 22(3). 935–945. 78 indexed citations
13.
Chang, Weise, Peter ten Dijke, & Doris K. Wu. (2002). BMP Pathways Are Involved in Otic Capsule Formation and Epithelial–Mesenchymal Signaling in the Developing Chicken Inner Ear. Developmental Biology. 251(2). 380–394. 68 indexed citations
14.
Wilcox, Edward R., Sadaf Naz, Saima Riazuddin, et al.. (2001). Mutations in the Gene Encoding Tight Junction Claudin-14 Cause Autosomal Recessive Deafness DFNB29. Cell. 104(1). 165–172. 344 indexed citations
15.
Chang, Weise, et al.. (1999). Ectopic Noggin Blocks Sensory and Nonsensory Organ Morphogenesis in the Chicken Inner Ear. Developmental Biology. 216(1). 369–381. 85 indexed citations
16.
Li, Hongzhen, Doris K. Wu, & Susan L. Sullivan. (1999). Characterization and expression of sema4g, a novel member of the semaphorin gene family. Mechanisms of Development. 87(1-2). 169–173. 10 indexed citations
17.
Choo, Daniel, et al.. (1998). The Differential Sensitivities of Inner Ear Structures to Retinoic Acid during Development. Developmental Biology. 204(1). 136–150. 36 indexed citations
18.
Kiernan, Amy E., Fábio Daumas Nunes, Doris K. Wu, & Donna M. Fekete. (1997). The Expression Domain of Two Related Homeobox Genes Defines a Compartment in the Chicken Inner Ear That May Be Involved in Semicircular Canal Formation. Developmental Biology. 191(2). 215–229. 42 indexed citations
19.
Wu, Doris K. & C. L. Cepko. (1993). Development of dopaminergic neurons is insensitive to optic nerve section in the neonatal rat retina. Developmental Brain Research. 74(2). 253–260. 30 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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