Doranda Perdomo

861 total citations
11 papers, 542 citations indexed

About

Doranda Perdomo is a scholar working on Infectious Diseases, Molecular Biology and Parasitology. According to data from OpenAlex, Doranda Perdomo has authored 11 papers receiving a total of 542 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Infectious Diseases, 4 papers in Molecular Biology and 4 papers in Parasitology. Recurrent topics in Doranda Perdomo's work include Amoebic Infections and Treatments (6 papers), Parasitic Infections and Diagnostics (4 papers) and Cystic Fibrosis Research Advances (3 papers). Doranda Perdomo is often cited by papers focused on Amoebic Infections and Treatments (6 papers), Parasitic Infections and Diagnostics (4 papers) and Cystic Fibrosis Research Advances (3 papers). Doranda Perdomo collaborates with scholars based in France, Canada and India. Doranda Perdomo's co-authors include Gergely L. Lukács, A. S. Verkman, Walter E. Finkbeiner, Radu G. Avramescu, Guido Veit, Pirjo M. Apaja, Nancy Guillén, Dániel Szöllősi, Miklós Bagdány and Puay-Wah Phuan and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Scientific Reports.

In The Last Decade

Doranda Perdomo

11 papers receiving 540 citations

Peers

Doranda Perdomo
Drake C. Bouzek United States
Heather A. Hartman United States
Young‐Ran Ha South Korea
David Carey United States
Lijia Wu China
Drake C. Bouzek United States
Doranda Perdomo
Citations per year, relative to Doranda Perdomo Doranda Perdomo (= 1×) peers Drake C. Bouzek

Countries citing papers authored by Doranda Perdomo

Since Specialization
Citations

This map shows the geographic impact of Doranda Perdomo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Doranda Perdomo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Doranda Perdomo more than expected).

Fields of papers citing papers by Doranda Perdomo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Doranda Perdomo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Doranda Perdomo. The network helps show where Doranda Perdomo may publish in the future.

Co-authorship network of co-authors of Doranda Perdomo

This figure shows the co-authorship network connecting the top 25 collaborators of Doranda Perdomo. A scholar is included among the top collaborators of Doranda Perdomo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Doranda Perdomo. Doranda Perdomo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Perdomo, Doranda, Denis Dacheux, Nicolas Landrein, et al.. (2022). TbKINX1B: a novel BILBO1 partner and an essential protein in bloodstream form Trypanosoma brucei. Parasite. 29. 14–14. 4 indexed citations
2.
Pineda, Erika & Doranda Perdomo. (2017). Entamoeba histolytica under Oxidative Stress: What Countermeasure Mechanisms Are in Place?. Cells. 6(4). 44–44. 38 indexed citations
3.
Perdomo, Doranda, Mélanie Bonhivers, & Derrick R. Robinson. (2016). The Trypanosome Flagellar Pocket Collar and Its Ring Forming Protein—TbBILBO1. Cells. 5(1). 9–9. 14 indexed citations
4.
Veit, Guido, Kathryn E. Oliver, Pirjo M. Apaja, et al.. (2016). Ribosomal Stalk Protein Silencing Partially Corrects the ΔF508-CFTR Functional Expression Defect. PLoS Biology. 14(5). e1002462–e1002462. 46 indexed citations
5.
Haggie, Peter M., Puay‐Wah Phuan, Joseph‐Anthony Tan, et al.. (2016). Correctors and Potentiators Rescue Function of the Truncated W1282X-Cystic Fibrosis Transmembrane Regulator (CFTR) Translation Product. Journal of Biological Chemistry. 292(3). 771–785. 72 indexed citations
6.
Perdomo, Doranda, Maria Manich, Sylvie Syan, et al.. (2016). Intracellular traffic of the lysine and glutamic acid rich protein KERP1 reveals features of endomembrane organization inEntamoeba histolytica. Cellular Microbiology. 18(8). 1134–1152. 13 indexed citations
7.
Perdomo, Doranda, et al.. (2014). Cellular and proteomics analysis of the endomembrane system from the unicellular Entamoeba histolytica. Journal of Proteomics. 112. 125–140. 31 indexed citations
8.
Perdomo, Doranda, et al.. (2014). Data set for the proteomics analysis of the endomembrane system from the unicellular Entamoeba histolytica. Data in Brief. 1. 29–36. 6 indexed citations
9.
Veit, Guido, Radu G. Avramescu, Doranda Perdomo, et al.. (2014). Some gating potentiators, including VX-770, diminish ΔF508-CFTR functional expression. Science Translational Medicine. 6(246). 246ra97–246ra97. 244 indexed citations
10.
Perdomo, Doranda, Bruno Baron, Arturo Rojo-Domı́nguez, et al.. (2013). The α-helical regions of KERP1 are important in Entamoeba histolytica adherence to human cells. Scientific Reports. 3(1). 1171–1171. 15 indexed citations
11.
Santi‐Rocca, Julien, et al.. (2009). Use of Bacterially Expressed dsRNA to Downregulate Entamoeba histolytica Gene Expression. PLoS ONE. 4(12). e8424–e8424. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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