Dora Višnjić

6.0k total citations · 1 hit paper
40 papers, 1.4k citations indexed

About

Dora Višnjić is a scholar working on Molecular Biology, Hematology and Cell Biology. According to data from OpenAlex, Dora Višnjić has authored 40 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 14 papers in Hematology and 8 papers in Cell Biology. Recurrent topics in Dora Višnjić's work include Acute Myeloid Leukemia Research (14 papers), Protein Kinase Regulation and GTPase Signaling (8 papers) and Retinoids in leukemia and cellular processes (8 papers). Dora Višnjić is often cited by papers focused on Acute Myeloid Leukemia Research (14 papers), Protein Kinase Regulation and GTPase Signaling (8 papers) and Retinoids in leukemia and cellular processes (8 papers). Dora Višnjić collaborates with scholars based in Croatia, United States and Italy. Dora Višnjić's co-authors include David W. Rowe, Héctor L. Aguila, Hrvoje Banfíƈ, Žana Kalajzić, Vedran Katavić, Joseph Lorenzo, Hrvoje Lalić, Vilma Dembitz, Drago Batinić and Alex Lichtler and has published in prestigious journals such as Journal of Biological Chemistry, Blood and Scientific Reports.

In The Last Decade

Dora Višnjić

40 papers receiving 1.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Hematopoiesis is severely altered in mice with an induced osteoblast deficiency

2004 566 citations Dora Višnjić et al. Blood profile →

Peers

Dora Višnjić

HEMAT

GENET

IMMUN

ONCOL

B. Masella Italy

Sophia K. Khaldoyanidi United States

B Nico Italy

Yasuhiko Miyata Japan

Ryohichi Sugimura Hong Kong

Elena Tenedini Italy

Haruko Shima Japan

Susan Prohaska United States

R Schirò Italy

Berno Dankbar Germany

B. Masella Italy

Dora Višnjić

890 ×1.2

381 ×0.7

HEMAT

248 ×0.8

GENET

233 ×0.9

IMMUN

221 ×0.9

ONCOL

Citations per year, relative to Dora Višnjić Dora Višnjić (= 1×) peers B. Masella

Countries citing papers authored by Dora Višnjić

Since Specialization

Citations

This map shows the geographic impact of Dora Višnjić's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dora Višnjić with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dora Višnjić more than expected).

Fields of papers citing papers by Dora Višnjić

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dora Višnjić. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dora Višnjić. The network helps show where Dora Višnjić may publish in the future.

Co-authorship network of co-authors of Dora Višnjić

This figure shows the co-authorship network connecting the top 25 collaborators of Dora Višnjić. A scholar is included among the top collaborators of Dora Višnjić based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dora Višnjić. Dora Višnjić is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Lalić, Hrvoje, Vilma Dembitz, Josip Batinić, et al.. (2024). Bone marrow stromal cells enhance differentiation of acute myeloid leukemia induced by pyrimidine synthesis inhibitors. American Journal of Physiology-Cell Physiology. 327(5). C1202–C1218. 1 indexed citations

Lalić, Hrvoje, et al.. (2023). Bone marrow stromal cells reduce low-dose cytarabine-induced differentiation of acute myeloid leukemia. Frontiers in Pharmacology. 14. 1258151–1258151. 2 indexed citations

Višnjić, Dora, et al.. (2021). AICAr, a Widely Used AMPK Activator with Important AMPK-Independent Effects: A Systematic Review. Cells. 10(5). 1095–1095. 93 indexed citations

Dembitz, Vilma, Hrvoje Lalić, Josip Batinić, et al.. (2021). All-trans retinoic acid induces differentiation in primary acute myeloid leukemia blasts carrying an inversion of chromosome 16. International Journal of Hematology. 115(1). 43–53. 6 indexed citations

Dembitz, Vilma, Hrvoje Lalić, Ivan Kodvanj, et al.. (2020). 5-aminoimidazole-4-carboxamide ribonucleoside induces differentiation in a subset of primary acute myeloid leukemia blasts. BMC Cancer. 20(1). 1090–1090. 6 indexed citations

Dembitz, Vilma, et al.. (2019). The ribonucleoside AICAr induces differentiation of myeloid leukemia by activating the ATR/Chk1 via pyrimidine depletion. Journal of Biological Chemistry. 294(42). 15257–15270. 18 indexed citations

Višnjić, Dora, Vilma Dembitz, & Hrvoje Lalić. (2018). The Role of AMPK/mTOR Modulators in the Therapy of Acute Myeloid Leukemia. Current Medicinal Chemistry. 26(12). 2208–2229. 21 indexed citations

Banfíƈ, Hrvoje, et al.. (2015). Inositol pyrophosphates modulate cell cycle independently of alteration in telomere length. Advances in Biological Regulation. 60. 22–28. 12 indexed citations

Dembitz, Vilma, et al.. (2015). The mechanism of synergistic effects of arsenic trioxide and rapamycin in acute myeloid leukemia cell lines lacking typical t(15;17) translocation. International Journal of Hematology. 102(1). 12–24. 9 indexed citations

10.

Višnjić, Dora, Hrvoje Lalić, Vilma Dembitz, & Hrvoje Banfíƈ. (2014). Metabolism and differentiation. Periodicum Biologorum. 116(1). 37–43. 14 indexed citations

11.

Lalić, Hrvoje, et al.. (2013). 5-Aminoimidazole-4-carboxamide ribonucleoside induces differentiation of acute myeloid leukemia cells. Leukemia & lymphoma. 55(10). 2375–2383. 15 indexed citations

12.

Lalić, Hrvoje, et al.. (2012). Rapamycin enhances dimethyl sulfoxide-mediated growth arrest in human myelogenous leukemia cells. Leukemia & lymphoma. 53(11). 2253–2261. 8 indexed citations

13.

Banfíƈ, Hrvoje, Antonio Bedalov, John D. York, & Dora Višnjić. (2012). Inositol Pyrophosphates Modulate S Phase Progression after Pheromone-induced Arrest in Saccharomyces cerevisiae. Journal of Biological Chemistry. 288(3). 1717–1725. 19 indexed citations

14.

Banfíƈ, Hrvoje, et al.. (2011). Different effects of phosphatidylinositol 3-kinase inhibitor LY294002 and Akt inhibitor SH-5 on cell cycle progression in synchronized HL-60 leukemia cells. Periodicum Biologorum. 113(1). 61–67. 1 indexed citations

15.

Višnjić, Dora & Hrvoje Banfíƈ. (2007). Nuclear phospholipid signaling: phosphatidylinositol-specific phospholipase C and phosphoinositide 3-kinase. Pflügers Archiv - European Journal of Physiology. 455(1). 19–30. 28 indexed citations

16.

Banfíƈ, Hrvoje, et al.. (2007). Two waves of the nuclear phospholipase C activity in serum-stimulated HL-60 cells during G1 phase of the cell cycle. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1771(4). 514–521. 10 indexed citations

17.

Brugnoli, Federica, et al.. (2006). The role of the nuclear Akt activation and Akt inhibitors in all-trans-retinoic acid-differentiated HL-60 cells. Leukemia. 20(6). 941–951. 50 indexed citations

18.

Banfíƈ, Hrvoje, et al.. (2005). Nuclear phospholipase C-β1b activation during G2/M and late G1 phase in nocodazole-synchronized HL-60 cells. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1733(2-3). 148–156. 26 indexed citations

19.

Višnjić, Dora, et al.. (2002). Antiproliferative effect of phosphatidylinositol 3-kinase inhibitor LY294002 on HL-60 and K562 leukemia cell lines. Periodicum Biologorum. 104(4). 405–411. 2 indexed citations

20.

Višnjić, Dora, Drago Batinić, & Hrvoje Banfíƈ. (1997). Arachidonic Acid Mediates Interferon-γ–Induced Sphingomyelin Hydrolysis and Monocytic Marker Expression in HL-60 Cell Line. Blood. 89(1). 81–91. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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