Donna R. Trollinger

811 total citations
9 papers, 666 citations indexed

About

Donna R. Trollinger is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Pathology and Forensic Medicine. According to data from OpenAlex, Donna R. Trollinger has authored 9 papers receiving a total of 666 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 3 papers in Cellular and Molecular Neuroscience and 2 papers in Pathology and Forensic Medicine. Recurrent topics in Donna R. Trollinger's work include Mitochondrial Function and Pathology (4 papers), ATP Synthase and ATPases Research (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). Donna R. Trollinger is often cited by papers focused on Mitochondrial Function and Pathology (4 papers), ATP Synthase and ATPases Research (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). Donna R. Trollinger collaborates with scholars based in United States, South Africa and Japan. Donna R. Trollinger's co-authors include John J. Lemasters, Wayne E. Cascio, Hisayuki Ohata, Richard Nuccitelli, R. Rivkah Isseroff, Ian S. Harper, Enrique Chacón, Brian Herman, B. Herman and John M. Bond and has published in prestigious journals such as The FASEB Journal, Biochemical and Biophysical Research Communications and FEBS Letters.

In The Last Decade

Donna R. Trollinger

9 papers receiving 650 citations

Peers

Donna R. Trollinger
Lisa M. Porter United Kingdom
Rachel Davis‐Taber United States
Rafael Mejía-Alvarez United States
Jiejia Xu China
Mickey M. Martin United States
Lisa M. Porter United Kingdom
Donna R. Trollinger
Citations per year, relative to Donna R. Trollinger Donna R. Trollinger (= 1×) peers Lisa M. Porter

Countries citing papers authored by Donna R. Trollinger

Since Specialization
Citations

This map shows the geographic impact of Donna R. Trollinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Donna R. Trollinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Donna R. Trollinger more than expected).

Fields of papers citing papers by Donna R. Trollinger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Donna R. Trollinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Donna R. Trollinger. The network helps show where Donna R. Trollinger may publish in the future.

Co-authorship network of co-authors of Donna R. Trollinger

This figure shows the co-authorship network connecting the top 25 collaborators of Donna R. Trollinger. A scholar is included among the top collaborators of Donna R. Trollinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Donna R. Trollinger. Donna R. Trollinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Trollinger, Donna R., R. Rivkah Isseroff, & Richard Nuccitelli. (2002). Calcium channel blockers inhibit galvanotaxis in human keratinocytes. Journal of Cellular Physiology. 193(1). 1–9. 96 indexed citations
2.
Douglas, Gordon C., et al.. (2001). Chemokine receptor expression by human syncytiotrophoblast. Journal of Reproductive Immunology. 49(2). 97–114. 31 indexed citations
3.
Lanoue, Louise, Donna R. Trollinger, Philip L. Strong, & Carl L. Keen. (2000). Functional impairments in preimplantation mouse embryos following boron deficiency. The FASEB Journal. 14(4). 53915. 7 indexed citations
4.
Trollinger, Donna R., Wayne E. Cascio, & John J. Lemasters. (2000). Mitochondrial Calcium Transients in Adult Rabbit Cardiac Myocytes: Inhibition by Ruthenium Red and Artifacts Caused by Lysosomal Loading of Ca2+-Indicating Fluorophores. Biophysical Journal. 79(1). 39–50. 121 indexed citations
5.
Lemasters, John J., Donna R. Trollinger, Ting Qian, Wayne E. Cascio, & Hisayuki Ohata. (1999). [29] Confocal imaging of Ca2+, pH, electrical potential, and membrane permeability in single living cells. Methods in enzymology on CD-ROM/Methods in enzymology. 302. 341–358. 30 indexed citations
6.
Trollinger, Donna R., Wayne E. Cascio, & John J. Lemasters. (1997). Selective Loading of Rhod 2 into Mitochondria Shows Mitochondrial Ca2+Transients during the Contractile Cycle in Adult Rabbit Cardiac Myocytes. Biochemical and Biophysical Research Communications. 236(3). 738–742. 166 indexed citations
7.
Lemasters, John J., John M. Bond, Enrique Chacón, et al.. (1996). The pH paradox in ischemia-reperfusion injury to cardiac myocytes. Birkhäuser Basel eBooks. 76. 99–114. 118 indexed citations
8.
Chacón, Enrique, John J. Lemasters, Hisayuki Ohata, et al.. (1996). Mitochondrial free calcium transients during excitation‐contraction coupling in rabbit cardiac myocytes. FEBS Letters. 382(1-2). 31–36. 85 indexed citations
9.
Ohata, Hisayuki, Donna R. Trollinger, & John J. Lemasters. (1994). Changes in shape and viability of cultured adult rabbit cardiac myocytes during ischemia/reperfusion injury.. PubMed. 86(3). 259–71. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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