Donglin Bai

4.8k total citations
89 papers, 3.6k citations indexed

About

Donglin Bai is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Donglin Bai has authored 89 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 77 papers in Molecular Biology, 26 papers in Genetics and 23 papers in Cellular and Molecular Neuroscience. Recurrent topics in Donglin Bai's work include Connexins and lens biology (61 papers), Nicotinic Acetylcholine Receptors Study (32 papers) and Yersinia bacterium, plague, ectoparasites research (26 papers). Donglin Bai is often cited by papers focused on Connexins and lens biology (61 papers), Nicotinic Acetylcholine Receptors Study (32 papers) and Yersinia bacterium, plague, ectoparasites research (26 papers). Donglin Bai collaborates with scholars based in Canada, United Kingdom and Japan. Donglin Bai's co-authors include Xiang‐Qun Gong, Qing Shao, Dale W. Laird, Beverley A. Orser, John F. MacDonald, Peter Pennefather, David B. Sattelle, Michael Jackson, Heinz Breer and Guoyun Zhu and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Donglin Bai

84 papers receiving 3.5k citations

Peers

Donglin Bai
Christian Derst Germany
Sylvie Diochot France
Linus Tsai United States
Noam Zilberberg Israel
William J. Joiner United States
Stephan Kellenberger Switzerland
Vânia F. Prado Canada
Hiroshi Nishino Japan
Aguan Wei United States
Marcel Crest France
Christian Derst Germany
Donglin Bai
Citations per year, relative to Donglin Bai Donglin Bai (= 1×) peers Christian Derst

Countries citing papers authored by Donglin Bai

Since Specialization
Citations

This map shows the geographic impact of Donglin Bai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Donglin Bai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Donglin Bai more than expected).

Fields of papers citing papers by Donglin Bai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Donglin Bai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Donglin Bai. The network helps show where Donglin Bai may publish in the future.

Co-authorship network of co-authors of Donglin Bai

This figure shows the co-authorship network connecting the top 25 collaborators of Donglin Bai. A scholar is included among the top collaborators of Donglin Bai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Donglin Bai. Donglin Bai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chen, Honghong & Donglin Bai. (2024). The rectification of heterotypic Cx46/Cx50 gap junction channels depends on intracellular magnesium. Biophysics Reports. 10(5). 336–336. 1 indexed citations
2.
Li, Yanjie, Juan Wang, Xingyuan Liu, et al.. (2023). Discovery of GJC1 (Cx45) as a New Gene Underlying Congenital Heart Disease and Arrhythmias. Biology. 12(3). 346–346. 10 indexed citations
3.
Neumann, Ruth, et al.. (2023). GJB4 variants linked to skin disease exhibit a trafficking deficiency en route to gap junction formation that can be restored by co-expression of select connexins. Frontiers in Cell and Developmental Biology. 11. 1073805–1073805. 5 indexed citations
4.
Haddad, Bassam G., Honghong Chen, Mena Atalla, et al.. (2021). Connexin 46 and connexin 50 gap junction channel properties are shaped by structural and dynamic features of their N‐terminal domains. The Journal of Physiology. 599(13). 3313–3335. 22 indexed citations
5.
Aoyama, Hiroshi, et al.. (2017). Junctional delay, frequency, and direction-dependent uncoupling of human heterotypic Cx45/Cx43 gap junction channels. Journal of Molecular and Cellular Cardiology. 111. 17–26. 24 indexed citations
6.
Bai, Donglin, et al.. (2017). Crucial motifs and residues in the extracellular loops influence the formation and specificity of connexin docking. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1860(1). 9–21. 41 indexed citations
7.
Kelly, John J., Jessica L. Esseltine, Qing Shao, et al.. (2016). Specific functional pathologies of Cx43 mutations associated with oculodentodigital dysplasia. Molecular Biology of the Cell. 27(14). 2172–2185. 20 indexed citations
8.
9.
Huang, Tao, Qing Shao, Andrew S. MacDonald, et al.. (2013). Autosomal recessiveGJA1(Cx43) gene mutations cause oculodentodigital dysplasia by distinct mechanisms. Journal of Cell Science. 126(Pt 13). 2857–66. 28 indexed citations
10.
Li, Xin, So Nakagawa, Tomitake Tsukihara, & Donglin Bai. (2012). Aspartic Acid Residue D3 Critically Determines Cx50 Gap Junction Channel Transjunctional Voltage-Dependent Gating and Unitary Conductance. Biophysical Journal. 102(5). 1022–1031. 12 indexed citations
11.
Li, Xin, Xiang‐Qun Gong, & Donglin Bai. (2010). The Role of Amino Terminus of Mouse Cx50 in Determining Transjunctional Voltage-Dependent Gating and Unitary Conductance. Biophysical Journal. 99(7). 2077–2086. 25 indexed citations
12.
Jeon, Doosoo & Donglin Bai. (2009). Improving Productivity of Pravastatin, HMG-CoA Reductase Inhibitor. Food Engineering Progress. 13(4). 243–250.
13.
Tong, Dan, Hongxing Wang, Isabelle Plante, et al.. (2009). A Dominant Loss-of-Function GJA1 (Cx43) Mutant Impairs Parturition in the Mouse1. Biology of Reproduction. 80(6). 1099–1106. 42 indexed citations
14.
Gan, Xiaohong Tracey, Xiang‐Qun Gong, Jenny Y. Xue, et al.. (2009). Sodium–hydrogen exchange inhibition attenuates glycoside-induced hypertrophy in rat ventricular myocytes. Cardiovascular Research. 85(1). 79–89. 17 indexed citations
15.
Manias, Janet L., Isabelle Plante, Qing Shao, et al.. (2008). Fate of connexin43 in cardiac tissue harbouring a disease-linked connexin43 mutant. Cardiovascular Research. 80(3). 385–395. 38 indexed citations
16.
Gollob, Michael H., Douglas L. Jones, Andrew D. Krahn, et al.. (2006). Somatic Mutations in the Connexin 40 Gene ( GJA5 ) in Atrial Fibrillation. New England Journal of Medicine. 354(25). 2677–2688. 380 indexed citations
17.
Gong, Xiang‐Qun, et al.. (2005). D‐Aspartate and NMDA, but not L‐aspartate, block AMPA receptors in rat hippocampal neurons. British Journal of Pharmacology. 145(4). 449–459. 33 indexed citations
18.
Kolaj, Miloslav, Donglin Bai, & Leo P. Renaud. (2004). GABAB Receptor Modulation of Rapid Inhibitory and Excitatory Neurotransmission From Subfornical Organ and Other Afferents to Median Preoptic Nucleus Neurons. Journal of Neurophysiology. 92(1). 111–122. 37 indexed citations
19.
Bai, Donglin, Robert U. Muller, & John Roder. (2002). Non‐ionotropic cross‐talk between AMPA and NMDA receptors in rodent hippocampal neurones. The Journal of Physiology. 543(1). 23–33. 16 indexed citations
20.
Bai, Donglin & David B. Sattelle. (1993). Neosurugatoxin blocks an α‐bungarotoxin‐sensitive neuronal nicotinic acetylcholine receptor. Archives of Insect Biochemistry and Physiology. 23(4). 161–167. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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