Doke C.R. Wahrer
- Molecular Biology top 5%
- Cell Biology top 1%
- Oncology top 5%
- Genetics top 5%
- Cancer Research top 5%
- Co-authors
- Daniel A. HaberDaphne W. BellIswar K. HariharanTaryn A. SchiripoNicolas TaponKieran F. HarveyKenneth H. MobergJudy E. Garber
- Topics
- DNA Repair Mechanisms (6 papers)Cancer-related Molecular Pathways (5 papers)Microtubule and mitosis dynamics (5 papers)
- Partner nations
- United StatesAustriaUnited Kingdom
In The Last Decade
Doke C.R. Wahrer
11 papers receiving 2.7k citations
Hit Papers
Peers
Comparison fields: 5 of 84
- Molecular Biology 2.0k
- Cell Biology 1.1k
- Oncology 793
- Genetics 613
- Cancer Research 363
Countries citing papers authored by Doke C.R. Wahrer
This map shows the geographic impact of Doke C.R. Wahrer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Doke C.R. Wahrer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Doke C.R. Wahrer more than expected).
Fields of papers citing papers by Doke C.R. Wahrer
This network shows the impact of papers produced by Doke C.R. Wahrer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Doke C.R. Wahrer. The network helps show where Doke C.R. Wahrer may publish in the future.
Co-authorship network of co-authors of Doke C.R. Wahrer
This figure shows the co-authorship network connecting the top 25 collaborators of Doke C.R. Wahrer. A scholar is included among the top collaborators of Doke C.R. Wahrer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Doke C.R. Wahrer. Doke C.R. Wahrer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 12 | |
| 2 | 59 | |
| 3 | 40 | |
| 4 | 194 | |
| 5 | salvador Promotes Both Cell Cycle Exit and Apoptosis in Drosophila and Is Mutated in Human Cancer Cell Linesbreakdown → | 723 |
| 6 | BACH1, a Novel Helicase-like Protein, Interacts Directly with BRCA1 and Contributes to Its DNA Repair Functionbreakdown → | 541 |
| 7 | 364 | |
| 8 | 47 | |
| 9 | Common nonsense mutations in RAD52. | 29 |
| 10 | Heterozygous Germ Line hCHK2 Mutations in Li-Fraumeni Syndromebreakdown → | 662 |
| 11 | 48 |
About Doke C.R. Wahrer
Doke C.R. Wahrer is a scholar working on Cell Biology, Oncology and Pathology and Forensic Medicine, having authored 11 papers that have together received 2.7k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (6 papers), Cancer-related Molecular Pathways (5 papers) and Microtubule and mitosis dynamics (5 papers). The work is most often cited by research in Cell Biology (1.1k citations), Molecular Biology (2.0k citations) and Oncology (793 citations). Doke C.R. Wahrer has collaborated with scholars based in United States, Austria and United Kingdom. Frequent co-authors include Daniel A. Haber, Daphne W. Bell, Iswar K. Hariharan, Taryn A. Schiripo, Nicolas Tapon, Kieran F. Harvey, Kenneth H. Moberg, Judy E. Garber, Kurt J. Isselbacher and Kristen E. Shannon. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.