Diane E. Brattain

1.3k total citations · 1 hit paper
19 papers, 1.1k citations indexed

About

Diane E. Brattain is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Diane E. Brattain has authored 19 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 9 papers in Oncology and 4 papers in Genetics. Recurrent topics in Diane E. Brattain's work include Cancer Cells and Metastasis (5 papers), TGF-β signaling in diseases (4 papers) and Bioactive Compounds and Antitumor Agents (3 papers). Diane E. Brattain is often cited by papers focused on Cancer Cells and Metastasis (5 papers), TGF-β signaling in diseases (4 papers) and Bioactive Compounds and Antitumor Agents (3 papers). Diane E. Brattain collaborates with scholars based in United States, Germany and India. Diane E. Brattain's co-authors include Michael G. Brattain, Jerry N. Thompson, Mary K. McKnight, Alan E. Levine, Naseema M. Hoosein, Kathleen M. Mulder, Michael Marks, Douglas D. Boyd, Subhas Chakrabarty and Lynn C. Yeoman and has published in prestigious journals such as JNCI Journal of the National Cancer Institute, Biochemical and Biophysical Research Communications and British Journal of Cancer.

In The Last Decade

Diane E. Brattain

19 papers receiving 1.1k citations

Hit Papers

Heterogeneity of malignant cells from a human colonic car... 1981 2026 1996 2011 1981 100 200 300 400

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Diane E. Brattain United States 16 731 492 229 171 106 19 1.1k
SHOJI KISHIMOTO Japan 11 798 1.1× 422 0.9× 388 1.7× 67 0.4× 43 0.4× 24 1.3k
HP Koeffler United States 18 975 1.3× 534 1.1× 178 0.8× 185 1.1× 119 1.1× 41 1.5k
Karen M. Hajra United States 7 1.4k 2.0× 679 1.4× 309 1.3× 117 0.7× 120 1.1× 7 1.8k
Alfred M. Rogan Austria 5 607 0.8× 440 0.9× 90 0.4× 72 0.4× 90 0.8× 6 1.0k
Yumi Yokoyama United States 16 729 1.0× 309 0.6× 379 1.7× 90 0.5× 82 0.8× 36 1.1k
Thomas Jascur United States 18 1.0k 1.4× 390 0.8× 188 0.8× 123 0.7× 83 0.8× 25 1.4k
Cynthia Kosinski United States 9 695 1.0× 427 0.9× 156 0.7× 105 0.6× 218 2.1× 9 1.2k
Narmada Shenoy United States 11 532 0.7× 240 0.5× 145 0.6× 67 0.4× 86 0.8× 17 990
Pia Herrmann Germany 18 665 0.9× 443 0.9× 236 1.0× 202 1.2× 41 0.4× 31 1.1k

Countries citing papers authored by Diane E. Brattain

Since Specialization
Citations

This map shows the geographic impact of Diane E. Brattain's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Diane E. Brattain with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Diane E. Brattain more than expected).

Fields of papers citing papers by Diane E. Brattain

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Diane E. Brattain. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Diane E. Brattain. The network helps show where Diane E. Brattain may publish in the future.

Co-authorship network of co-authors of Diane E. Brattain

This figure shows the co-authorship network connecting the top 25 collaborators of Diane E. Brattain. A scholar is included among the top collaborators of Diane E. Brattain based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Diane E. Brattain. Diane E. Brattain is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Hoosein, Naseema M., et al.. (1989). Promotion of differentiation in human colon carcinoma cells by vasoactive intestinal polypeptide. Regulatory Peptides. 24(1). 15–26. 13 indexed citations
2.
Hoosein, Naseema M., Mary K. McKnight, Alan E. Levine, et al.. (1989). Differential sensitivity of subclasses of human colon carcinoma cell lines to the growth inhibitory effects of transforming growth factor-β1. Experimental Cell Research. 181(2). 442–453. 124 indexed citations
3.
Mulder, Kathleen M., et al.. (1988). Modulation of C-myc by transforming growth factor-β in human colon carcinoma cells. Biochemical and Biophysical Research Communications. 150(2). 711–716. 52 indexed citations
5.
McKnight, Mary K., et al.. (1988). Different epidermal growth factor growth responses and receptor levels in human colon carcinoma cell lines. Cancer Letters. 43(1-2). 139–143. 45 indexed citations
6.
Hoosein, Naseema M., Diane E. Brattain, Mary K. McKnight, & Michael G. Brattain. (1988). Comparison of the effects of transforming growth factor β, N,N-dimethylformamide, and retinoic acid on transformed and nontransformed fibroblasts. Experimental Cell Research. 175(1). 125–135. 13 indexed citations
7.
Mulder, Kathleen M., et al.. (1988). Characterization of transforming growth factor-beta-resistant subclones isolated from a transforming growth factor-beta-sensitive human colon carcinoma cell line.. PubMed. 48(24 Pt 1). 7120–5. 35 indexed citations
8.
Boyd, Douglas D., Alan E. Levine, Diane E. Brattain, Mary K. McKnight, & Michael G. Brattain. (1988). Comparison of growth requirements of two human intratumoral colon carcinoma cell lines in monolayer and soft agarose.. PubMed. 48(9). 2469–74. 57 indexed citations
9.
Boyd, Douglas D., et al.. (1988). Alterations of the biological characteristics of a colon carcinoma cell line by colon-derived substrata material.. PubMed. 48(10). 2825–31. 30 indexed citations
10.
Hoosein, Naseema M., Diane E. Brattain, Mary K. McKnight, Alan E. Levine, & Michael G. Brattain. (1987). Characterization of the inhibitory effects of transforming growth factor-beta on a human colon carcinoma cell line.. PubMed. 47(11). 2950–4. 68 indexed citations
11.
Levine, Alan E., et al.. (1986). Retinoic acid restores normal growth control to a transformed mouse embryo fibroblast cell line. Cancer Letters. 33(1). 33–43. 10 indexed citations
12.
Levine, Alan E., et al.. (1985). Identification of endogenous inhibitory growth factors from a human colon carcinoma cell line.. PubMed. 45(5). 2248–54. 24 indexed citations
13.
Willson, James K. V., Byron H. Long, Subhas Chakrabarty, Diane E. Brattain, & Michael G. Brattain. (1985). Effects of BMY 25282, a mitomycin C analogue, in mitomycin C-resistant human colon cancer cells.. PubMed. 45(11 Pt 1). 5281–6. 25 indexed citations
14.
Willson, James K. V., Byron H. Long, Michael Marks, et al.. (1984). Mitomycin C resistance in a human colon carcinoma cell line associated with cell surface protein alterations.. PubMed. 44(12 Pt 1). 5880–5. 25 indexed citations
15.
Long, Byron H., et al.. (1984). Effects of mitomycin on human colon carcinoma cells.. PubMed. 73(4). 787–92. 17 indexed citations
16.
Brattain, Michael G., et al.. (1983). Characterization of human colon carcinoma cell lines isolated from a single primary tumour. British Journal of Cancer. 47(3). 373–381. 55 indexed citations
17.
Brattain, Michael G., et al.. (1982). Enhancement of Growth of Human Colon Tumor Cell Lines by Feeder Layers of Murine Fibroblasts<xref ref-type="fn" rid="FN2">2</xref>. JNCI Journal of the National Cancer Institute. 69(4). 767–71. 24 indexed citations
18.
Brattain, Michael G., et al.. (1981). Initiation and characterization of cultures of human colonic carcinoma with different biological characteristics utilizing feeder layers of confluent fibroblasts.. PubMed. 2(5). 355–66. 71 indexed citations
19.
Brattain, Michael G., et al.. (1981). Heterogeneity of malignant cells from a human colonic carcinoma.. PubMed. 41(5). 1751–6. 409 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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