Deying Su

424 total citations · 1 hit paper
16 papers, 256 citations indexed

About

Deying Su is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Surgery. According to data from OpenAlex, Deying Su has authored 16 papers receiving a total of 256 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 7 papers in Pathology and Forensic Medicine and 3 papers in Surgery. Recurrent topics in Deying Su's work include Spine and Intervertebral Disc Pathology (5 papers), Hedgehog Signaling Pathway Studies (2 papers) and TGF-β signaling in diseases (2 papers). Deying Su is often cited by papers focused on Spine and Intervertebral Disc Pathology (5 papers), Hedgehog Signaling Pathway Studies (2 papers) and TGF-β signaling in diseases (2 papers). Deying Su collaborates with scholars based in China, United States and Poland. Deying Su's co-authors include Zhiheng Liao, Peiqiang Su, Taifeng Zhou, Yuyu Chen, Hengyu Liu, Xianjian Qiu, Yong Wan, Shun Zhang, Jinna Wu and Taiqiu Chen and has published in prestigious journals such as Advanced Science, Molecular Therapy and Cell Death and Disease.

In The Last Decade

Deying Su

15 papers receiving 255 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Self-amplifying loop of NF-κB and periostin initiated by PIEZO1 accelerates mechano-induced senescence of nucleus pulposus cells and intervertebral disc degeneration

2022 111 citations Jinna Wu, Yuyu Chen et al. Molecular Therapy profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Deying Su China	8	112	87	51	46	46	16	256
Daxue Zhu China	11	100 0.9×	130 1.5×	40 0.8×	72 1.6×	34 0.7×	23	281
Taiqiu Chen China	7	121 1.1×	84 1.0×	29 0.6×	56 1.2×	61 1.3×	10	302
Dongsheng Huang China	12	103 0.9×	254 2.9×	91 1.8×	116 2.5×	76 1.7×	21	430
Wei Shui China	5	73 0.7×	76 0.9×	66 1.3×	29 0.6×	21 0.5×	14	221
Kyung‐Eun Lim South Korea	12	261 2.3×	39 0.4×	16 0.3×	18 0.4×	57 1.2×	21	420
Ryosuke Sato Japan	8	61 0.5×	72 0.8×	108 2.1×	21 0.5×	30 0.7×	19	217
Wenbo Lin China	11	125 1.1×	127 1.5×	118 2.3×	16 0.3×	13 0.3×	32	311
Huiqiao Wu China	14	70 0.6×	243 2.8×	244 4.8×	50 1.1×	44 1.0×	37	413
Rongxin Sun China	12	166 1.5×	33 0.4×	51 1.0×	9 0.2×	25 0.5×	17	324
Rony Sapir‐Koren Israel	10	203 1.8×	33 0.4×	27 0.5×	12 0.3×	61 1.3×	10	401

Countries citing papers authored by Deying Su

Since Specialization

Citations

This map shows the geographic impact of Deying Su's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Deying Su with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Deying Su more than expected).

Fields of papers citing papers by Deying Su

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Deying Su. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Deying Su. The network helps show where Deying Su may publish in the future.

Co-authorship network of co-authors of Deying Su

This figure shows the co-authorship network connecting the top 25 collaborators of Deying Su. A scholar is included among the top collaborators of Deying Su based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Deying Su. Deying Su is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Li, Chuan, Shuqing Chen, Ya Xiao, et al.. (2025). Mapk7 enhances osteogenesis and suppresses adipogenesis by activating Lrp6/β-catenin signaling axis in mesenchymal stem cells. Communications Biology. 8(1). 310–310. 3 indexed citations

Zhang, Baolin, Ya Xiao, Deying Su, et al.. (2024). M13, an anthraquinone compound isolated from Morinda officinalis alleviates the progression of the osteoarthritis via the regulation of STAT3. Phytomedicine. 136. 156329–156329. 3 indexed citations

Zhang, Shun, Baolin Zhang, Zhiheng Liao, et al.. (2024). Hnrnpk protects against osteoarthritis through targeting WWC1 mRNA and inhibiting Hippo signaling pathway. Molecular Therapy. 32(5). 1461–1478. 8 indexed citations

Li, Bin, et al.. (2024). Establishing and Validating a novel Prognostic Model in the Initial Diagnosis of Non-small Cell Lung Cancer with Bone Metastases. Journal of Cancer. 15(14). 4612–4622.

Zhou, Taifeng, Yu Chen, Zhiheng Liao, et al.. (2023). Spatiotemporal Characterization of Human Early Intervertebral Disc Formation at Single‐Cell Resolution. Advanced Science. 10(14). e2206296–e2206296. 31 indexed citations

Wu, Chengzhi, Hengyu Liu, Xiaoming Yang, et al.. (2023). Mapk7 deletion in chondrocytes causes vertebral defects by reducing MEF2C/PTEN/AKT signaling. Genes & Diseases. 11(2). 964–977. 4 indexed citations

Liao, Zhiheng, Deying Su, Caixia Xu, et al.. (2022). Dihydroartemisinin Attenuated Intervertebral Disc Degeneration via Inhibiting PI3K/AKT and NF‐κB Signaling Pathways. Oxidative Medicine and Cellular Longevity. 2022(1). 8672969–8672969. 4 indexed citations

Wu, Jinna, Yuyu Chen, Zhiheng Liao, et al.. (2022). Self-amplifying loop of NF-κB and periostin initiated by PIEZO1 accelerates mechano-induced senescence of nucleus pulposus cells and intervertebral disc degeneration. Molecular Therapy. 30(10). 3241–3256. 111 indexed citations breakdown →

Chen, Yuyu, Jinna Wu, Shun Zhang, et al.. (2022). Hnrnpk maintains chondrocytes survival and function during growth plate development via regulating Hif1α-glycolysis axis. Cell Death and Disease. 13(9). 803–803. 10 indexed citations

10.

Cui, Shangbin, et al.. (2020). Analysis of the morphometric change in the uncinate process of the cervical spondylosis patients: A study of radiological anatomy. Journal of Orthopaedic Translation. 24. 32–38. 7 indexed citations

11.

Zhou, Hang, Chengjie Lian, Tingting Wang, et al.. (2019). MET mutation causes muscular dysplasia and arthrogryposis. EMBO Molecular Medicine. 11(3). 2 indexed citations

12.

Xu, Caixia, Xiaoming Yang, Hang Zhou, et al.. (2019). A novel ZRS variant causes preaxial polydactyly type I by increased sonic hedgehog expression in the developing limb bud. Genetics in Medicine. 22(1). 189–198. 12 indexed citations

13.

Liu, Lei, Tingting Wang, Xiaoming Yang, et al.. (2019). MTNR1B loss promotes chordoma recurrence by abrogating melatonin‐mediated β‐catenin signaling repression. Journal of Pineal Research. 67(2). e12588–e12588. 19 indexed citations

14.

Zheng, Shuhui, Hang Zhou, Bo Gao, et al.. (2018). Estrogen promotes the onset and development of idiopathic scoliosis via disproportionate endochondral ossification of the anterior and posterior column in a bipedal rat model. Experimental & Molecular Medicine. 50(11). 1–11. 18 indexed citations

15.

Zhou, Taifeng, Yongqian Wang, Hang Zhou, et al.. (2018). Dual novel mutations in SLC26A2 in two siblings with multiple epiphyseal dysplasia 4 from a Chinese family: a case report. BMC Medical Genetics. 19(1). 70–70. 4 indexed citations

16.

Zhou, Taifeng, Chong Chen, Hang Zhou, et al.. (2018). Mutant MAPK7-Induced Idiopathic Scoliosis is Linked to Impaired Osteogenesis. Cellular Physiology and Biochemistry. 48(3). 880–890. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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