Deborah J. Vestal

2.7k total citations · 1 hit paper
28 papers, 2.2k citations indexed

About

Deborah J. Vestal is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Deborah J. Vestal has authored 28 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 14 papers in Oncology and 14 papers in Immunology. Recurrent topics in Deborah J. Vestal's work include interferon and immune responses (12 papers), Cytokine Signaling Pathways and Interactions (11 papers) and Cell Adhesion Molecules Research (8 papers). Deborah J. Vestal is often cited by papers focused on interferon and immune responses (12 papers), Cytokine Signaling Pathways and Interactions (11 papers) and Cell Adhesion Molecules Research (8 papers). Deborah J. Vestal collaborates with scholars based in United States and Germany. Deborah J. Vestal's co-authors include Scott R. McKercher, Gregory W. Henkel, R. Maki, Hélène Baribault, G E Wu, Bruce E. Torbett, Kenton L. Anderson, Ann J. Feeney, M Klemsz and Christopher J. Paige and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Cell Biology and The EMBO Journal.

In The Last Decade

Deborah J. Vestal

27 papers receiving 2.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Targeted disruption of the PU.1 gene results in multiple hematopoietic abnormalities.

1996 896 citations Scott R. McKercher, Deborah J. Vestal et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Deborah J. Vestal United States	18	1.1k	1.0k	357	356	275	28	2.2k
Nicolas Reymond France	18	1.1k 1.0×	1.3k 1.3×	257 0.7×	902 2.5×	336 1.2×	22	2.9k
Mónica Gordón‐Alonso Spain	20	790 0.7×	918 0.9×	380 1.1×	275 0.8×	139 0.5×	24	1.9k
Michael J. Klemsz United States	24	1.5k 1.3×	1.3k 1.3×	141 0.4×	511 1.4×	269 1.0×	37	2.8k
Dominique Dunon France	24	651 0.6×	1.0k 1.0×	666 1.9×	275 0.8×	110 0.4×	46	2.0k
Alec M. Cheng United States	25	1.7k 1.5×	2.0k 2.0×	339 0.9×	545 1.5×	215 0.8×	28	3.9k
Lindsay Hewlett United Kingdom	18	950 0.9×	668 0.7×	317 0.9×	157 0.4×	121 0.4×	20	2.1k
Tomoya Katakai Japan	28	881 0.8×	2.1k 2.1×	313 0.9×	571 1.6×	134 0.5×	63	3.1k
Scott C. Todd United States	13	684 0.6×	561 0.6×	626 1.8×	168 0.5×	107 0.4×	16	1.7k
Eileen A. Elliott United States	22	741 0.7×	1.6k 1.6×	135 0.4×	374 1.1×	365 1.3×	24	2.8k
Ann J. Feeney United States	34	1.9k 1.7×	3.1k 3.1×	133 0.4×	461 1.3×	284 1.0×	90	4.5k

Countries citing papers authored by Deborah J. Vestal

Since Specialization

Citations

This map shows the geographic impact of Deborah J. Vestal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Deborah J. Vestal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Deborah J. Vestal more than expected).

Fields of papers citing papers by Deborah J. Vestal

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Deborah J. Vestal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Deborah J. Vestal. The network helps show where Deborah J. Vestal may publish in the future.

Co-authorship network of co-authors of Deborah J. Vestal

This figure shows the co-authorship network connecting the top 25 collaborators of Deborah J. Vestal. A scholar is included among the top collaborators of Deborah J. Vestal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Deborah J. Vestal. Deborah J. Vestal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Morran, Michael P., et al.. (2024). The Large GTPase Guanylate-Binding Protein-1 (GBP-1) Promotes Mitochondrial Fission in Glioblastoma. International Journal of Molecular Sciences. 25(20). 11236–11236.

Vestal, Deborah J., et al.. (2022). Unraveling the Role of Guanylate-Binding Proteins (GBPs) in Breast Cancer: A Comprehensive Literature Review and New Data on Prognosis in Breast Cancer Subtypes. Cancers. 14(11). 2794–2794. 7 indexed citations

Wilson, John P., et al.. (2021). The Large GTPase, GBP-2, Regulates Rho Family GTPases to Inhibit Migration and Invadosome Formation in Breast Cancer Cells. Cancers. 13(22). 5632–5632. 6 indexed citations

Tipton, Aaron R., et al.. (2016). Guanylate-Binding Protein-1 protects ovarian cancer cell lines but not breast cancer cell lines from killing by paclitaxel. Biochemical and Biophysical Research Communications. 478(4). 1617–1623. 24 indexed citations

Tipton, Aaron R., et al.. (2016). hGBP-1 Expression Predicts Shorter Progression-Free Survival in Ovarian Cancers, While Contributing to Paclitaxel Resistance. Journal of Cancer Therapy. 7(13). 994–1007. 17 indexed citations

Balasubramanian, Sujata, et al.. (2011). The Interferon-γ-induced GTPase, mGBP-2, Inhibits Tumor Necrosis Factor α (TNF-α) Induction of Matrix Metalloproteinase-9 (MMP-9) by Inhibiting NF-κB and Rac Protein. Journal of Biological Chemistry. 286(22). 20054–20064. 31 indexed citations

Vestal, Deborah J., et al.. (2010). The Guanylate-Binding Proteins: Emerging Insights into the Biochemical Properties and Functions of This Family of Large Interferon-Induced Guanosine Triphosphatase. Journal of Interferon & Cytokine Research. 31(1). 89–97. 131 indexed citations

Balasubramanian, Sujata, et al.. (2010). Role of GTP Binding, Isoprenylation, and the C-Terminal α-Helices in the Inhibition of Cell Spreading by the Interferon-Induced GTPase, Mouse Guanylate-Binding Protein-2. Journal of Interferon & Cytokine Research. 31(3). 291–298. 7 indexed citations

Balasubramanian, Sujata, et al.. (2010). The Interferon-γ–induced Murine Guanylate-Binding Protein-2 Inhibits Rac Activation during Cell Spreading on Fibronectin and after Platelet-derived Growth Factor Treatment: Role for Phosphatidylinositol 3-Kinase. Molecular Biology of the Cell. 21(14). 2514–2528. 30 indexed citations

10.

Gray, John C., et al.. (2006). In Silico Genomic Analysis of the Human and Murine Guanylate-Binding Protein (GBP) Gene Clusters. Journal of Interferon & Cytokine Research. 26(5). 328–352. 126 indexed citations

11.

Vestal, Deborah J.. (2005). Review: The Guanylate-Binding Proteins (GBPs): Proinflammatory Cytokine-Induced Members of the Dynamin Superfamily with Unique GTPase Activity. Journal of Interferon & Cytokine Research. 25(8). 435–443. 43 indexed citations

12.

Gorbacheva, Victoria, et al.. (2005). Inhibition of VSV and EMCV replication by the interferon-induced GTPase, mGBP-2: differential requirement for wild-type GTP binding domain. Archives of Virology. 150(6). 1213–1220. 78 indexed citations

13.

Gorbacheva, Victoria, Daniel J. Lindner, Ganes C. Sen, & Deborah J. Vestal. (2002). The Interferon (IFN)-induced GTPase, mGBP-2. Journal of Biological Chemistry. 277(8). 6080–6087. 72 indexed citations

14.

Vestal, Deborah J., Victoria Gorbacheva, & Ganes C. Sen. (2000). Different Subcellular Localizations for the Related Interferon-Induced GTPases, MuGBP-1 and MuGBP-2: Implications for Different Functions?. Journal of Interferon & Cytokine Research. 20(11). 991–1000. 45 indexed citations

15.

Patel, Rekha C., Deborah J. Vestal, Zan Xu, et al.. (1999). DRBP76, a Double-stranded RNA-binding Nuclear Protein, Is Phosphorylated by the Interferon-induced Protein Kinase, PKR. Journal of Biological Chemistry. 274(29). 20432–20437. 110 indexed citations

16.

Vestal, Deborah J., Janice E. Buss, Scott R. McKercher, et al.. (1998). Murine GBP-2: A New IFN-γ-Induced Member of the GBP Family of GTPases Isolated from Macrophages. Journal of Interferon & Cytokine Research. 18(11). 977–985. 35 indexed citations

17.

Vestal, Deborah J., et al.. (1996). Rat p67 GBP Is Induced by Interferon-γ and Isoprenoid-Modified in Macrophages. Biochemical and Biophysical Research Communications. 224(2). 528–534. 18 indexed citations

18.

Sacristán, María P., Deborah J. Vestal, María T. Dours‐Zimmermann, & Barbara Ranscht. (1993). T‐Cadherin 2: Molecular characterization, function in cell adhesion, and coexpression with T‐cadherin and N‐cadherin. Journal of Neuroscience Research. 34(6). 664–680. 28 indexed citations

19.

Vestal, Deborah J., Bruce H. Davis, & Caroline Enns. (1990). A rapid redistribution of the transferrin receptor to the cell surface of HL-60 cells and K562 cells upon treatment with dimethyl sulfoxide due to slowing of endocytosis. Archives of Biochemistry and Biophysics. 276(1). 278–284. 4 indexed citations

20.

Smith, Jeffrey W., et al.. (1990). Purification and functional characterization of integrin alpha v beta 5. An adhesion receptor for vitronectin.. Journal of Biological Chemistry. 265(19). 11008–11013. 212 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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