This map shows the geographic impact of DC Wheeler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by DC Wheeler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites DC Wheeler more than expected).
This network shows the impact of papers produced by DC Wheeler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by DC Wheeler. The network helps show where DC Wheeler may publish in the future.
Co-authorship network of co-authors of DC Wheeler
This figure shows the co-authorship network connecting the top 25 collaborators of DC Wheeler.
A scholar is included among the top collaborators of DC Wheeler based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with DC Wheeler. DC Wheeler is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
All Works
20 of 20 papers shown
1.
Bjornstad, Petter, et al.. (2021). The potential roles of osmotic and non-osmotic sodium handling in mediating effects of SGLT2 inhibitors on heart failure. UCL Discovery (University College London).1 indexed citations
2.
White, Claire, Sanjay Bhandari, K. Farrington, et al.. (2019). Intravenous Iron in Patients Undergoing Maintenance Hemodialysis [vol 380 (5), p502, 31 Jan 2019]. UCL Discovery (University College London).3 indexed citations
3.
Agarwal, Rupesh, James Baldassarre, G. Bakris, et al.. (2019). Evaluating the Effects of Canagliflozin on Cardiovascular and Renal Events in Patients with Type 2 Diabetes and Chronic Kidney Disease According to Baseline HbA1c, Including Those with HbA1c <7%: Results From the CREDENCE Trial.10 indexed citations
Wheeler, DC, et al.. (2018). Effects of the SGLT2 inhibitor dapagliflozin in patients with type 2 diabetes and stages 3b-4 chronic kidney disease.1 indexed citations
6.
Wheeler, DC, Rajiv Agarwal, G. Bakris, et al.. (2018). The Canagliflozin and Renal Endpoints in Diabetes with Established Nephropathy Clinical Evaluation (CREDENCE) study rationale and design. UCL Discovery (University College London).3 indexed citations
7.
Wheeler, DC, Sangita Basnet, Walter Douthat, et al.. (2017). Complications of CKD: current state, knowledge gaps, and strategy for action. UCL Discovery (University College London).2 indexed citations
8.
Wheeler, DC, et al.. (2013). KDIGO Clinical Practice Guideline for Lipid Management in Chronic Kidney Disease Foreword. UCL Discovery (University College London).19 indexed citations
9.
Wheeler, DC, et al.. (2013). KDIGO Clinical Practice Guideline for the Evaluation and Management of Chronic Kidney Disease Foreword. UCL Discovery (University College London).26 indexed citations
Levey, Andrew S., R. C. Atkins, Josef Coresh, et al.. (2007). Enfermedad renal cronica como problema global en salud publica: Abordajes e iniciativas - propuesta de la Kidney Disease Improving Global Outcomes.5 indexed citations
Jf, Moorhead, et al.. (2003). Dysregulation of LDL receptor by an inflammatory cytokine: A new pathway for foam cell formation in vascular smooth muscle cells.. UCL Discovery (University College London).7 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.