Dawn D. Han

1.2k total citations
16 papers, 976 citations indexed

About

Dawn D. Han is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Dawn D. Han has authored 16 papers receiving a total of 976 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 12 papers in Cellular and Molecular Neuroscience and 2 papers in Cell Biology. Recurrent topics in Dawn D. Han's work include Neurotransmitter Receptor Influence on Behavior (12 papers), Receptor Mechanisms and Signaling (9 papers) and Neuroscience and Neuropharmacology Research (7 papers). Dawn D. Han is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (12 papers), Receptor Mechanisms and Signaling (9 papers) and Neuroscience and Neuropharmacology Research (7 papers). Dawn D. Han collaborates with scholars based in United States, China and Italy. Dawn D. Han's co-authors include Howard H. Gu, Rong Chen, Michael R. Tilley, Erik R. Hill, Morgane Thomsen, S. Barak Caine, Hua Wei, Ning Quan, Fu-Ming Zhou and Fu‐Wen Zhou and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Analytical Biochemistry and Biochemical and Biophysical Research Communications.

In The Last Decade

Dawn D. Han

16 papers receiving 967 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Dawn D. Han United States	10	725	460	172	152	90	16	976
Yoko Hagino Japan	16	537 0.7×	309 0.7×	84 0.5×	159 1.0×	77 0.9×	46	779
Sonja J. Stutz United States	20	720 1.0×	442 1.0×	88 0.5×	109 0.7×	121 1.3×	29	1.1k
Marcy J. Bubar United States	16	996 1.4×	590 1.3×	105 0.6×	129 0.8×	148 1.6×	21	1.2k
Marc L. Zeise Chile	20	651 0.9×	289 0.6×	198 1.2×	152 1.0×	64 0.7×	35	1.0k
Brenda Trail United Kingdom	11	716 1.0×	432 0.9×	88 0.5×	123 0.8×	107 1.2×	15	975
Boris Mlinar Italy	20	738 1.0×	669 1.5×	37 0.2×	135 0.9×	84 0.9×	34	1.2k
James R. Shoblock United States	16	539 0.7×	394 0.9×	93 0.5×	301 2.0×	65 0.7×	23	1.1k
Gwenaëlle Le Pen France	18	871 1.2×	386 0.8×	113 0.7×	287 1.9×	225 2.5×	34	1.2k
Charlie Reavill United Kingdom	20	937 1.3×	764 1.7×	171 1.0×	214 1.4×	158 1.8×	28	1.4k

Countries citing papers authored by Dawn D. Han

Since Specialization

Citations

This map shows the geographic impact of Dawn D. Han's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dawn D. Han with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dawn D. Han more than expected).

Fields of papers citing papers by Dawn D. Han

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dawn D. Han. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dawn D. Han. The network helps show where Dawn D. Han may publish in the future.

Co-authorship network of co-authors of Dawn D. Han

This figure shows the co-authorship network connecting the top 25 collaborators of Dawn D. Han. A scholar is included among the top collaborators of Dawn D. Han based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dawn D. Han. Dawn D. Han is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Wu, Hai‐Yin, Brian O’Neill, Dawn D. Han, et al.. (2014). Restoration of cocaine stimulation and reward by reintroducing wild type dopamine transporter in adult knock-in mice with a cocaine-insensitive dopamine transporter. Neuropharmacology. 86. 31–37. 7 indexed citations

O’Neill, Brian, Michael R. Tilley, Dawn D. Han, et al.. (2014). Behavior of knock-in mice with a cocaine-insensitive dopamine transporter after virogenetic restoration of cocaine sensitivity in the striatum. Neuropharmacology. 79. 626–633. 13 indexed citations

Rajamani, Keerthi Thirtamara, Brian O’Neill, Dawn D. Han, et al.. (2014). Inactivation of the Catalytic Phosphatase Domain of PTPRT/RPTPρ Increases Social Interaction in Mice. Autism Research. 8(1). 19–28. 5 indexed citations

Naughton, Bartholomew J., Dawn D. Han, & Howard H. Gu. (2011). Fluorescence-based evaluation of shRNA efficacy. Analytical Biochemistry. 417(1). 162–164. 5 indexed citations

Pinsonneault, Julia K., Dawn D. Han, Katherine E. Burdick, et al.. (2011). Dopamine Transporter Gene Variant Affecting Expression in Human Brain is Associated with Bipolar Disorder. Neuropsychopharmacology. 36(8). 1644–1655. 117 indexed citations

Martin, Bradley J., Bartholomew J. Naughton, Keerthi Thirtamara Rajamani, et al.. (2010). Dopamine transporter inhibition is necessary for cocaine‐induced increases in dendritic spine density in the nucleus accumbens. Synapse. 65(6). 490–496. 27 indexed citations

Thomsen, Morgane, Dawn D. Han, Howard H. Gu, & S. Barak Caine. (2009). Lack of Cocaine Self-Administration in Mice Expressing a Cocaine-Insensitive Dopamine Transporter. Journal of Pharmacology and Experimental Therapeutics. 331(1). 204–211. 74 indexed citations

Tilley, Michael R., Brian O’Neill, Dawn D. Han, & Howard H. Gu. (2008). Cocaine does not produce reward in absence of dopamine transporter inhibition. Neuroreport. 20(1). 9–12. 25 indexed citations

Tilley, Michael R., et al.. (2007). Cocaine reward and locomotion stimulation in mice with reduced dopamine transporter expression. BMC Neuroscience. 8(1). 42–42. 53 indexed citations

10.

Chen, Rong, Michael R. Tilley, Hua Wei, et al.. (2006). Abolished cocaine reward in mice with a cocaine-insensitive dopamine transporter. Proceedings of the National Academy of Sciences. 103(24). 9333–9338. 215 indexed citations

11.

Gu, Howard H., Xiao-Hong Wu, & Dawn D. Han. (2006). Conserved serine residues in serotonin transporter contribute to high-affinity cocaine binding. Biochemical and Biophysical Research Communications. 343(4). 1179–1185. 8 indexed citations

12.

Chen, Rong, Hua Wei, Erik R. Hill, et al.. (2006). Direct evidence that two cysteines in the dopamine transporter form a disulfide bond. Molecular and Cellular Biochemistry. 298(1-2). 41–48. 25 indexed citations

13.

Han, Dawn D. & Howard H. Gu. (2006). Comparison of the monoamine transporters from human and mouse in their sensitivities to psychostimulant drugs. BMC Pharmacology. 6(1). 6–6. 338 indexed citations

14.

Han, Dawn D., Rong Chen, Erik R. Hill, Michael R. Tilley, & Howard H. Gu. (2006). Cause and solutions to the polymerase chain reaction smear problem in genotyping. Analytical Biochemistry. 353(2). 296–298. 2 indexed citations

15.

Chen, Rong, Xiao-Hong Wu, Hua Wei, Dawn D. Han, & Howard H. Gu. (2005). Molecular cloning and functional characterization of the dopamine transporter from Eloria noyesi, a caterpillar pest of cocaine-rich coca plants. Gene. 366(1). 152–160. 9 indexed citations

16.

Chen, Rong, Dawn D. Han, & Howard H. Gu. (2005). A triple mutation in the second transmembrane domain of mouse dopamine transporter markedly decreases sensitivity to cocaine and methylphenidate. Journal of Neurochemistry. 94(2). 352–359. 53 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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