Davide F. Robbiani
- Molecular Biology top 2%
- Immunology top 0.5%
- Infectious Diseases top 1%
- Oncology top 2%
- Cancer Research top 2%
- Co-authors
- Michel C. NussenzweigGwendalyn J. RandolphWilliam A. MüllerAnne BothmerAndré NussenzweigRalph M. SteinmanRafael CasellasKayo Inaba
- Topics
- DNA Repair Mechanisms (17 papers)T-cell and B-cell Immunology (14 papers)Immune Cell Function and Interaction (12 papers)
- Partner nations
- United StatesCubaSwitzerland
In The Last Decade
Davide F. Robbiani
48 papers receiving 7.2k citations
Hit Papers
Peers
Comparison fields: 5 of 127
- Molecular Biology 3.8k
- Immunology 2.7k
- Infectious Diseases 1.4k
- Oncology 1.4k
- Cancer Research 700
Countries citing papers authored by Davide F. Robbiani
This map shows the geographic impact of Davide F. Robbiani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Davide F. Robbiani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Davide F. Robbiani more than expected).
Fields of papers citing papers by Davide F. Robbiani
This network shows the impact of papers produced by Davide F. Robbiani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Davide F. Robbiani. The network helps show where Davide F. Robbiani may publish in the future.
Co-authorship network of co-authors of Davide F. Robbiani
This figure shows the co-authorship network connecting the top 25 collaborators of Davide F. Robbiani. A scholar is included among the top collaborators of Davide F. Robbiani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Davide F. Robbiani. Davide F. Robbiani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 5 | |
| 3 | 157 | |
| 4 | SARS-CoV-2 neutralizing antibody structures inform therapeutic strategiesbreakdown → | 832 |
| 5 | 131 | |
| 6 | 105 | |
| 7 | 49 | |
| 8 | 309 | |
| 9 | 31 | |
| 10 | 325 | |
| 11 | 21 | |
| 12 | 158 | |
| 13 | 222 | |
| 14 | 259 | |
| 15 | 1 | |
| 16 | 117 | |
| 17 | 27 | |
| 18 | 56 | |
| 19 | Differentiation of Phagocytic Monocytes into Lymph Node Dendritic Cells In Vivobreakdown → | 707 |
| 20 | 229 |
About Davide F. Robbiani
Davide F. Robbiani is a scholar working on Immunology, Oncology and Genetics, having authored 48 papers that have together received 7.2k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (17 papers), T-cell and B-cell Immunology (14 papers) and Immune Cell Function and Interaction (12 papers). The work is most often cited by research in Immunology (2.7k citations), Infectious Diseases (1.4k citations) and Molecular Biology (3.8k citations). Davide F. Robbiani has collaborated with scholars based in United States, Cuba and Switzerland. Frequent co-authors include Michel C. Nussenzweig, Gwendalyn J. Randolph, William A. Müller, Anne Bothmer, André Nussenzweig, Ralph M. Steinman, Rafael Casellas, Kayo Inaba, Mila Janković and Anna Gazumyan. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.