David Öling
Impact in
- Aging top 2%
- Genetics, Aging, and Longevity in Model Organisms
- Cell Biology top 10%
- Endoplasmic Reticulum Stress and Disease
Papers in ⓘ
-
- CRISPR and Genetic Engineering 3
- DNA Repair Mechanisms 2
- Receptor Mechanisms and Signaling 2
- Heat shock proteins research 2
- RNA and protein synthesis mechanisms 1
- Aging 4
- Genetics, Aging, and Longevity in Model Organisms 4
- Co-authors
- Thomas Nyström (4 shared papers)Beidong Liu (2 shared papers)Xinxin Hao (2 shared papers)Antonio Caballero (2 shared papers)Julie Grantham (1 shared paper)Kristian Kvint (4 shared papers)David Bell (1 shared paper)W. M. Shaw (2 shared papers)
- Journals
- ACS Synthetic Biology (2 papers)Cell (2 papers)Molecular Biology of the Cell (1 paper)Molecular Cell (1 paper)The EMBO Journal (1 paper)
- Partner nations
- SwedenUnited KingdomJapan
In The Last Decade
David Öling
9 papers receiving 538 citations
Peers
Comparison fields: 5 of 81
- Aging 120
- Cell Biology 125
- Molecular Biology 464
- Geriatrics and Gerontology 24
- Cellular and Molecular Neuroscience 63
Countries citing papers authored by David Öling
This map shows the geographic impact of David Öling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Öling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Öling more than expected).
Fields of papers citing papers by David Öling
This network shows the impact of papers produced by David Öling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Öling. The network helps show where David Öling may publish in the future.
Co-authors
The 25 scholars most cited alongside David Öling, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2010 | 263 | |
| 2 | 2019 | 129 | |
| 3 | 2011 | 63 | |
| 4 | 2014 | 42 | |
| 5 | 2023 | 20 | |
| 6 | 2021 | 14 | |
| 7 | 2014 | 5 | |
| 8 | 2018 | 4 | |
| 9 | 2022 | 4 |
About David Öling
David Öling is a scholar working on Molecular Biology, Aging, Cell Biology, Cellular and Molecular Neuroscience and Ecology, having authored 9 papers that have together received 544 indexed citations. Recurring topics across this work include Genetics, Aging, and Longevity in Model Organisms (4 papers), CRISPR and Genetic Engineering (3 papers), DNA Repair Mechanisms (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Receptor Mechanisms and Signaling (2 papers), Heat shock proteins research (2 papers), RNA and protein synthesis mechanisms (1 paper) and Reproductive System and Pregnancy (1 paper). The work is most often cited by research in Aging (120 citations), Cell Biology (125 citations), Molecular Biology (464 citations), Geriatrics and Gerontology (24 citations) and Cellular and Molecular Neuroscience (63 citations). David Öling has collaborated with scholars based in Sweden, United Kingdom and Japan. Frequent co-authors include Thomas Nyström, Beidong Liu, Xinxin Hao, Antonio Caballero, Julie Grantham, Kristian Kvint, David Bell, W. M. Shaw, Hitoshi Yamauchi and Tom Ellis. Their work appears in journals such as ACS Synthetic Biology, Cell, Molecular Biology of the Cell, Molecular Cell and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.