David Öling

873 total citations
9 papers, 544 citations indexed

About

David Öling is a scholar working on Molecular Biology, Aging and Cell Biology. According to data from OpenAlex, David Öling has authored 9 papers receiving a total of 544 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Aging and 2 papers in Cell Biology. Recurrent topics in David Öling's work include Genetics, Aging, and Longevity in Model Organisms (4 papers), CRISPR and Genetic Engineering (3 papers) and DNA Repair Mechanisms (2 papers). David Öling is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (4 papers), CRISPR and Genetic Engineering (3 papers) and DNA Repair Mechanisms (2 papers). David Öling collaborates with scholars based in Sweden, United Kingdom and Japan. David Öling's co-authors include Thomas Nyström, Beidong Liu, Antonio Caballero, Xinxin Hao, Julie Grantham, Kristian Kvint, W. M. Shaw, Hitoshi Yamauchi, Graham Ladds and Niklas Larsson and has published in prestigious journals such as Cell, The EMBO Journal and Molecular Cell.

In The Last Decade

David Öling

9 papers receiving 538 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David Öling Sweden 6 464 125 120 63 38 9 544
Chuankai Zhou United States 7 626 1.3× 238 1.9× 116 1.0× 77 1.2× 96 2.5× 11 739
Scott Tsuchiyama United States 7 380 0.8× 90 0.7× 224 1.9× 18 0.3× 80 2.1× 7 486
Sarah Hanzén Sweden 7 419 0.9× 141 1.1× 123 1.0× 34 0.5× 41 1.1× 7 472
Triana Amen Israel 12 485 1.0× 209 1.7× 48 0.4× 81 1.3× 96 2.5× 22 712
Jane E. Leadsham United Kingdom 9 327 0.7× 100 0.8× 45 0.4× 22 0.3× 44 1.2× 9 442
Pin‐Chao Liao United States 13 305 0.7× 68 0.5× 46 0.4× 39 0.6× 62 1.6× 26 452
Alice Zuin Spain 9 451 1.0× 88 0.7× 104 0.9× 21 0.3× 24 0.6× 13 518
Ulrike Topf Poland 10 648 1.4× 210 1.7× 58 0.5× 46 0.7× 75 2.0× 17 759
Simon D. Bourque Canada 13 471 1.0× 66 0.5× 258 2.1× 20 0.3× 70 1.8× 16 614
Edith Bogengruber Austria 12 442 1.0× 80 0.6× 50 0.4× 27 0.4× 38 1.0× 14 522

Countries citing papers authored by David Öling

Since Specialization
Citations

This map shows the geographic impact of David Öling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Öling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Öling more than expected).

Fields of papers citing papers by David Öling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Öling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Öling. The network helps show where David Öling may publish in the future.

Co-authorship network of co-authors of David Öling

This figure shows the co-authorship network connecting the top 25 collaborators of David Öling. A scholar is included among the top collaborators of David Öling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Öling. David Öling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Postel, Sandra, Lisa Wissler, Carina Johansson, et al.. (2023). Quaternary glucocorticoid receptor structure highlights allosteric interdomain communication. Nature Structural & Molecular Biology. 30(3). 286–295. 20 indexed citations
2.
Öling, David, Samuel Gilberto, Jordi Chi, et al.. (2022). FRAGLER: A Fragment Recycler Application Enabling Rapid and Scalable Modular DNA Assembly. ACS Synthetic Biology. 11(7). 2229–2237. 4 indexed citations
3.
Hanzén, Sarah, Katarina Vielfort, David Öling, et al.. (2021). Differential role of cytosolic Hsp70s in longevity assurance and protein quality control. PLoS Genetics. 17(1). e1008951–e1008951. 14 indexed citations
4.
Shaw, W. M., Hitoshi Yamauchi, Glen-Oliver F. Gowers, et al.. (2019). Engineering a Model Cell for Rational Tuning of GPCR Signaling. Cell. 177(3). 782–796.e27. 129 indexed citations
5.
Öling, David, W. M. Shaw, Sonya Clark, et al.. (2018). Large Scale Synthetic Site Saturation GPCR Libraries Reveal Novel Mutations That Alter Glucose Signaling. ACS Synthetic Biology. 7(9). 2317–2321. 4 indexed citations
6.
Öling, David, et al.. (2014). Loss of Ubp3 increases silencing, decreases unequal recombination in rDNA, and shortens the replicative life span inSaccharomyces cerevisiae. Molecular Biology of the Cell. 25(12). 1916–1924. 5 indexed citations
7.
Öling, David, Frederik Eisele, Kristian Kvint, & Thomas Nyström. (2014). Opposing roles of Ubp3-dependent deubiquitination regulate replicative life span and heat resistance. The EMBO Journal. 33(7). 747–761. 42 indexed citations
8.
Caballero, Antonio, Beidong Liu, David Öling, et al.. (2011). Absence of Mitochondrial Translation Control Proteins Extends Life Span by Activating Sirtuin-Dependent Silencing. Molecular Cell. 42(3). 390–400. 63 indexed citations
9.
Liu, Beidong, Antonio Caballero, Xinxin Hao, et al.. (2010). The Polarisome Is Required for Segregation and Retrograde Transport of Protein Aggregates. Cell. 140(2). 257–267. 263 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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