David L. Prole

2.1k total citations
30 papers, 1.6k citations indexed

About

David L. Prole is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, David L. Prole has authored 30 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 10 papers in Physiology and 8 papers in Cellular and Molecular Neuroscience. Recurrent topics in David L. Prole's work include Ion channel regulation and function (16 papers), Calcium signaling and nucleotide metabolism (9 papers) and Cellular transport and secretion (7 papers). David L. Prole is often cited by papers focused on Ion channel regulation and function (16 papers), Calcium signaling and nucleotide metabolism (9 papers) and Cellular transport and secretion (7 papers). David L. Prole collaborates with scholars based in United Kingdom, South Sudan and United States. David L. Prole's co-authors include Colin W. Taylor, Stephen C. Tovey, Nagendra Babu Thillaiappan, Taufiq Rahman, Neil V. Marrion, Gary Yellen, Dev Churamani, G. Cristina Brailoiu, Robert Hooper and Sandip Patel and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and The Journal of Cell Biology.

In The Last Decade

David L. Prole

30 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David L. Prole United Kingdom 23 961 478 374 329 280 30 1.6k
Ching‐On Wong United States 21 664 0.7× 361 0.8× 223 0.6× 245 0.7× 537 1.9× 32 1.6k
Eamonn J. Dickson United States 26 1.3k 1.4× 224 0.5× 534 1.4× 665 2.0× 230 0.8× 54 2.4k
Eva C. Schwarz Germany 27 988 1.0× 185 0.4× 348 0.9× 342 1.0× 805 2.9× 49 2.3k
Miyuki Kuno Japan 20 1.1k 1.2× 176 0.4× 599 1.6× 118 0.4× 245 0.9× 59 2.0k
Clotilde Randriamampita France 23 1.3k 1.3× 222 0.5× 512 1.4× 218 0.7× 423 1.5× 35 2.2k
Stephen H. Loukin United States 22 1.1k 1.1× 87 0.2× 321 0.9× 188 0.6× 541 1.9× 32 1.8k
Sylvain Féliciangéli France 20 1.3k 1.4× 72 0.2× 430 1.1× 426 1.3× 156 0.6× 27 1.9k
Paula Nunes Switzerland 24 838 0.9× 136 0.3× 161 0.4× 210 0.6× 218 0.8× 37 1.5k
Judith A. Airey United States 30 2.1k 2.1× 428 0.9× 991 2.6× 257 0.8× 483 1.7× 44 2.8k
Richard H. Ashley United Kingdom 25 1.8k 1.9× 117 0.2× 545 1.5× 339 1.0× 83 0.3× 48 2.5k

Countries citing papers authored by David L. Prole

Since Specialization
Citations

This map shows the geographic impact of David L. Prole's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David L. Prole with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David L. Prole more than expected).

Fields of papers citing papers by David L. Prole

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David L. Prole. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David L. Prole. The network helps show where David L. Prole may publish in the future.

Co-authorship network of co-authors of David L. Prole

This figure shows the co-authorship network connecting the top 25 collaborators of David L. Prole. A scholar is included among the top collaborators of David L. Prole based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David L. Prole. David L. Prole is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wills, E. J., et al.. (2025). Quantitative approaches for studying G protein-coupled receptor signalling and pharmacology. Journal of Cell Science. 138(1). 3 indexed citations
2.
Prole, David L. & Colin W. Taylor. (2019). Structure and Function of IP3Receptors. Cold Spring Harbor Perspectives in Biology. 11(4). a035063–a035063. 129 indexed citations
4.
5.
Thillaiappan, Nagendra Babu, et al.. (2018). IP3 Receptors Preferentially Associate with ER-Lysosome Contact Sites and Selectively Deliver Ca2+ to Lysosomes. Cell Reports. 25(11). 3180–3193.e7. 144 indexed citations
6.
Konieczny, Vera, et al.. (2017). Cyclic AMP Recruits a Discrete Intracellular Ca2+ Store by Unmasking Hypersensitive IP3 Receptors. Cell Reports. 18(3). 711–722. 20 indexed citations
7.
Thillaiappan, Nagendra Babu, et al.. (2017). Ca2+ signals initiate at immobile IP3 receptors adjacent to ER-plasma membrane junctions. Nature Communications. 8(1). 1505–1505. 120 indexed citations
8.
Carrara, Guia, Nuno Saraiva, Maddy Parsons, et al.. (2015). Golgi Anti-apoptotic Proteins Are Highly Conserved Ion Channels That Affect Apoptosis and Cell Migration. Journal of Biological Chemistry. 290(18). 11785–11801. 31 indexed citations
10.
Prole, David L. & Colin W. Taylor. (2013). Identification and Analysis of Putative Homologues of Mechanosensitive Channels in Pathogenic Protozoa. PLoS ONE. 8(6). e66068–e66068. 49 indexed citations
11.
Saraiva, Nuno, David L. Prole, Guia Carrara, et al.. (2013). hGAAP promotes cell adhesion and migration via the stimulation of store-operated Ca2+ entry and calpain 2. The Journal of Cell Biology. 202(4). 699–713. 40 indexed citations
12.
Saraiva, Nuno, David L. Prole, Guia Carrara, et al.. (2013). Human and Viral Golgi Anti-apoptotic Proteins (GAAPs) Oligomerize via Different Mechanisms and Monomeric GAAP Inhibits Apoptosis and Modulates Calcium. Journal of Biological Chemistry. 288(18). 13057–13067. 28 indexed citations
13.
Prole, David L. & Colin W. Taylor. (2012). Identification and Analysis of Cation Channel Homologues in Human Pathogenic Fungi. PLoS ONE. 7(8). e42404–e42404. 29 indexed citations
14.
Taylor, Colin W. & David L. Prole. (2012). Ca2+ Signalling by IP3 Receptors. Sub-cellular biochemistry. 59. 1–34. 11 indexed citations
15.
Prole, David L. & Neil V. Marrion. (2012). Identification of Putative Potassium Channel Homologues in Pathogenic Protozoa. PLoS ONE. 7(2). e32264–e32264. 25 indexed citations
16.
Prole, David L. & Colin W. Taylor. (2011). Identification of Intracellular and Plasma Membrane Calcium Channel Homologues in Pathogenic Parasites. PLoS ONE. 6(10). e26218–e26218. 94 indexed citations
17.
Rossi, Ana M., Stephen C. Tovey, Taufiq Rahman, David L. Prole, & Colin W. Taylor. (2011). Analysis of IP3 receptors in and out of cells. Biochimica et Biophysica Acta (BBA) - General Subjects. 1820(8). 1214–1227. 12 indexed citations
18.
Brailoiu, Eugen, Taufiq Rahman, Dev Churamani, et al.. (2010). An NAADP-gated Two-pore Channel Targeted to the Plasma Membrane Uncouples Triggering from Amplifying Ca2+ Signals. Journal of Biological Chemistry. 285(49). 38511–38516. 149 indexed citations
19.
Prole, David L. & Gary Yellen. (2006). Reversal of HCN Channel Voltage Dependence via Bridging of the S4–S5 Linker and Post-S6. The Journal of General Physiology. 128(3). 273–282. 50 indexed citations
20.
Prole, David L. & Neil V. Marrion. (2004). Ionic Permeation and Conduction Properties of Neuronal KCNQ2/KCNQ3 Potassium Channels. Biophysical Journal. 86(3). 1454–1469. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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