David L. Pflugh

557 total citations
9 papers, 457 citations indexed

About

David L. Pflugh is a scholar working on Molecular Biology, Immunology and Epidemiology. According to data from OpenAlex, David L. Pflugh has authored 9 papers receiving a total of 457 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Immunology and 2 papers in Epidemiology. Recurrent topics in David L. Pflugh's work include Immune Cell Function and Interaction (5 papers), T-cell and B-cell Immunology (4 papers) and Bone Metabolism and Diseases (2 papers). David L. Pflugh is often cited by papers focused on Immune Cell Function and Interaction (5 papers), T-cell and B-cell Immunology (4 papers) and Bone Metabolism and Diseases (2 papers). David L. Pflugh collaborates with scholars based in United States, Italy and South Korea. David L. Pflugh's co-authors include Alfred L.M. Bothwell, David G.T. Hesslein, David G. Schatz, Stephen E. Maher, Dipanjan Chowdhury, Ranjan Sen, Mark C. Horowitz, Kathryn Calame, Andrei Thomas‐Tikhonenko and Duonan Yu and has published in prestigious journals such as Genes & Development, Nature Immunology and The Journal of Immunology.

In The Last Decade

David L. Pflugh

9 papers receiving 449 citations

Peers

David L. Pflugh

IMMUN

MB

ONCOL

GENET

SURGE

Akiko Sugahara–Tobinai Japan

L Chen United States

Theresa Podrebarac United States

Hedda H. van Ravenswaay Claasen Netherlands

Jenny A. F. Vermeer Netherlands

A. E. Irvine United Kingdom

Jeanne Naulet France

Velizar Shivarov Bulgaria

Anna Szaflarska Poland

C. Ribbens Belgium

Akiko Sugahara–Tobinai Japan

David L. Pflugh

253 ×0.9

IMMUN

68 ×0.3

MB

52 ×0.6

ONCOL

35 ×0.7

GENET

31 ×0.9

SURGE

Citations per year, relative to David L. Pflugh David L. Pflugh (= 1×) peers Akiko Sugahara–Tobinai

Countries citing papers authored by David L. Pflugh

Since Specialization

Citations

This map shows the geographic impact of David L. Pflugh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David L. Pflugh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David L. Pflugh more than expected).

Fields of papers citing papers by David L. Pflugh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David L. Pflugh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David L. Pflugh. The network helps show where David L. Pflugh may publish in the future.

Co-authorship network of co-authors of David L. Pflugh

This figure shows the co-authorship network connecting the top 25 collaborators of David L. Pflugh. A scholar is included among the top collaborators of David L. Pflugh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David L. Pflugh. David L. Pflugh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Kowalski, S. P., et al.. (2012). Patent Landscape of Influenza A Virus Prophylactic Vaccines and Related Technologies. University of New Hampshire Scholars Repository (University of New Hampshire at Manchester). 1 indexed citations

2.

Giambra, Vincenzo, Sabrina A. Volpi, Alexander Emelyanov, et al.. (2008). Pax5 and Linker Histone H1 Coordinate DNA Methylation and Histone Modifications in the 3′ Regulatory Region of the Immunoglobulin Heavy Chain Locus. Molecular and Cellular Biology. 28(19). 6123–6133. 42 indexed citations

3.

Horowitz, Mark C., Alfred L.M. Bothwell, David G.T. Hesslein, David L. Pflugh, & David G. Schatz. (2005). B cells and osteoblast and osteoclast development. Immunological Reviews. 208(1). 141–153. 59 indexed citations

4.

Johnson, Kristen, David L. Pflugh, Duonan Yu, et al.. (2004). B cell–specific loss of histone 3 lysine 9 methylation in the VH locus depends on Pax5. Nature Immunology. 5(8). 853–861. 91 indexed citations

5.

Horowitz, Mark C., Yougen Xi, David L. Pflugh, et al.. (2004). Pax5-Deficient Mice Exhibit Early Onset Osteopenia with Increased Osteoclast Progenitors. The Journal of Immunology. 173(11). 6583–6591. 48 indexed citations

6.

Hesslein, David G.T., David L. Pflugh, Dipanjan Chowdhury, et al.. (2003). Pax5 is required for recombination of transcribed, acetylated, 5′ IgH V gene segments. Genes & Development. 17(1). 37–42. 132 indexed citations

7.

Pflugh, David L., Stephen E. Maher, & Alfred L.M. Bothwell. (2002). Ly-6 Superfamily Members Ly-6A/E, Ly-6C, and Ly-6I Recognize Two Potential Ligands Expressed by B Lymphocytes. The Journal of Immunology. 169(9). 5130–5136. 43 indexed citations

8.

Pflugh, David L., Stephen E. Maher, & Alfred L.M. Bothwell. (2000). Ly-6I, a New Member of the Murine Ly-6 Superfamily with a Distinct Pattern of Expression. The Journal of Immunology. 165(1). 313–321. 21 indexed citations

9.

Maher, Stephen E., David L. Pflugh, N J Larsen, Max F. Rothschild, & Alfred L.M. Bothwell. (1998). STRUCTURE/FUNCTION CHARACTERIZATION OF PORCINE CD59. Transplantation. 66(8). 1094–1100. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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