David J. Barton

3.6k total citations
47 papers, 2.8k citations indexed

About

David J. Barton is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Infectious Diseases. According to data from OpenAlex, David J. Barton has authored 47 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Cardiology and Cardiovascular Medicine, 26 papers in Molecular Biology and 20 papers in Infectious Diseases. Recurrent topics in David J. Barton's work include Viral Infections and Immunology Research (35 papers), Viral gastroenteritis research and epidemiology (17 papers) and RNA Research and Splicing (14 papers). David J. Barton is often cited by papers focused on Viral Infections and Immunology Research (35 papers), Viral gastroenteritis research and epidemiology (17 papers) and RNA Research and Splicing (14 papers). David J. Barton collaborates with scholars based in United States, United Kingdom and Switzerland. David J. Barton's co-authors include James B. Flanegan, Brian J. Kempf, Kenneth E. Murray, Robert H. Silverman, Benjamin J. Morasco, Dorothea L. Sawicki, Stanley G. Sawicki, Babal K. Jha, Rohit K. Jangra and Tetsuro Shimakami and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

David J. Barton

47 papers receiving 2.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David J. Barton United States 30 1.5k 1.4k 982 489 406 47 2.8k
Tim Skern Austria 33 2.4k 1.6× 2.2k 1.6× 1.1k 1.1× 341 0.7× 401 1.0× 102 4.1k
Johan A. den Boon United States 25 575 0.4× 1.3k 0.9× 1.0k 1.0× 697 1.4× 268 0.7× 37 3.5k
Kevin L. McKnight United States 18 522 0.4× 630 0.5× 765 0.8× 148 0.3× 227 0.6× 32 1.8k
G M Duke United States 13 1.2k 0.8× 1.3k 0.9× 709 0.7× 284 0.6× 337 0.8× 15 3.1k
Noemı́ Sevilla Spain 32 667 0.5× 573 0.4× 1.3k 1.3× 218 0.4× 700 1.7× 69 3.1k
Asit K. Pattnaik United States 38 484 0.3× 1.0k 0.8× 2.4k 2.4× 392 0.8× 818 2.0× 88 4.7k
James B. Flanegan United States 29 1.8k 1.3× 1.5k 1.1× 989 1.0× 587 1.2× 82 0.2× 44 2.7k
Dominique Garcin Switzerland 38 380 0.3× 1.0k 0.8× 1.8k 1.8× 321 0.7× 1.6k 3.9× 72 4.3k
Stefan Schwartz Sweden 38 403 0.3× 2.6k 1.9× 714 0.7× 139 0.3× 896 2.2× 94 4.2k
Yorihiro Nishimura Japan 24 901 0.6× 645 0.5× 729 0.7× 50 0.1× 316 0.8× 63 2.0k

Countries citing papers authored by David J. Barton

Since Specialization
Citations

This map shows the geographic impact of David J. Barton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David J. Barton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David J. Barton more than expected).

Fields of papers citing papers by David J. Barton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David J. Barton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David J. Barton. The network helps show where David J. Barton may publish in the future.

Co-authorship network of co-authors of David J. Barton

This figure shows the co-authorship network connecting the top 25 collaborators of David J. Barton. A scholar is included among the top collaborators of David J. Barton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David J. Barton. David J. Barton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Li, Yize, Eveline Kindler, Daphne Cooper, et al.. (2020). Physiologic RNA targets and refined sequence specificity of coronavirus EndoU. RNA. 26(12). 1976–1999. 24 indexed citations
2.
Kempf, Brian J., et al.. (2020). Picornaviral polymerase domain exchanges reveal a modular basis for distinct biochemical activities of viral RNA-dependent RNA polymerases. Journal of Biological Chemistry. 295(31). 10624–10637. 4 indexed citations
3.
Kempf, Brian J., et al.. (2019). Picornavirus RNA Recombination Counteracts Error Catastrophe. Journal of Virology. 93(14). 21 indexed citations
4.
Cooper, Daphne, Babal K. Jha, Robert H. Silverman, Jay R. Hesselberth, & David J. Barton. (2014). Ribonuclease L and metal-ion–independent endoribonuclease cleavage sites in host and viral RNAs. Nucleic Acids Research. 42(8). 5202–5216. 43 indexed citations
5.
Kempf, Brian J., et al.. (2012). A Template RNA Entry Channel in the Fingers Domain of the Poliovirus Polymerase. Journal of Molecular Biology. 417(4). 263–278. 26 indexed citations
6.
Malathi, Krishnamurthy, et al.. (2010). RNase L releases a small RNA from HCV RNA that refolds into a potent PAMP. RNA. 16(11). 2108–2119. 109 indexed citations
7.
Jha, Babal K., et al.. (2008). A viral RNA competitively inhibits the antiviral endoribonuclease domain of RNase L. RNA. 14(6). 1026–1036. 46 indexed citations
8.
Barton, David J., et al.. (2008). Cis-active RNA elements (CREs) and picornavirus RNA replication. Virus Research. 139(2). 240–252. 85 indexed citations
9.
10.
Jha, Babal K., et al.. (2007). A Phylogenetically Conserved RNA Structure in the Poliovirus Open Reading Frame Inhibits the Antiviral Endoribonuclease RNase L. Journal of Virology. 81(11). 5561–5572. 69 indexed citations
11.
Kechris, Katerina, et al.. (2007). Hepatitis C virus RNA: Dinucleotide frequencies and cleavage by RNase L. Virus Research. 130(1-2). 85–95. 50 indexed citations
12.
Barton, David J., et al.. (2005). 2Apro is a multifunctional protein that regulates the stability, translation and replication of poliovirus RNA. Virology. 345(2). 346–357. 28 indexed citations
13.
Xu, Zan, et al.. (2004). Sensitivity of Hepatitis C Virus RNA to the Antiviral Enzyme Ribonuclease L Is Determined by a Subset of Efficient Cleavage Sites. Journal of Interferon & Cytokine Research. 24(11). 664–676. 41 indexed citations
14.
Murray, Kenneth E., et al.. (2004). Replication of Poliovirus RNA with Complete Internal Ribosome Entry Site Deletions. Journal of Virology. 78(3). 1393–1402. 29 indexed citations
15.
16.
Pathak, Harsh B., Saikat Ghosh, Suresh D. Sharma, et al.. (2002). Structure-Function Relationships of the RNA-dependent RNA Polymerase from Poliovirus (3Dpol). Journal of Biological Chemistry. 277(35). 31551–31562. 64 indexed citations
17.
Barton, David J., et al.. (2001). 5′ cloverleaf in poliovirus RNA is a cis-acting replication element required for negative-strand synthesis. The EMBO Journal. 20(6). 1439–1448. 227 indexed citations
18.
Murray, Kenneth E., et al.. (2001). Poly(rC) binding proteins mediate poliovirus mRNA stability. RNA. 7(8). 1126–1141. 66 indexed citations
19.
Barton, David J., et al.. (1999). Fractal clustering of induced seismicity in The Geysers geothermal area, California. Geophysical Journal International. 139(2). 317–324. 43 indexed citations
20.
Barton, David J., et al.. (1996). Poliovirus RNA Polymerase Mutation 3D-M394T Results in a Temperature-Sensitive Defect in RNA Synthesis. Virology. 217(2). 459–469. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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