David I. Weiss

620 total citations
7 papers, 353 citations indexed

About

David I. Weiss is a scholar working on Immunology, Infectious Diseases and Molecular Biology. According to data from OpenAlex, David I. Weiss has authored 7 papers receiving a total of 353 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Immunology, 2 papers in Infectious Diseases and 2 papers in Molecular Biology. Recurrent topics in David I. Weiss's work include Immune Cell Function and Interaction (3 papers), Immunotherapy and Immune Responses (2 papers) and Antimicrobial Peptides and Activities (2 papers). David I. Weiss is often cited by papers focused on Immune Cell Function and Interaction (3 papers), Immunotherapy and Immune Responses (2 papers) and Antimicrobial Peptides and Activities (2 papers). David I. Weiss collaborates with scholars based in United States, Switzerland and South Africa. David I. Weiss's co-authors include Robert L. Modlin, Feiyang Ma, María Teresa Ochoa, Barry R. Bloom, Tudor Hughes, Shuai Shao, Jóhann E. Guðjónsson, Alex K. Shalek, Priscila R. Andrade and Lam C. Tsoi and has published in prestigious journals such as Journal of Clinical Investigation, Immunity and The Journal of Immunology.

In The Last Decade

David I. Weiss

7 papers receiving 345 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David I. Weiss United States	7	184	140	53	49	46	7	353
Franziska Schmidt Germany	7	165 0.9×	109 0.8×	51 1.0×	25 0.5×	38 0.8×	8	270
Nina J. Gao United States	8	128 0.7×	145 1.0×	43 0.8×	45 0.9×	32 0.7×	12	338
Tennille D. Thelen United States	10	135 0.7×	50 0.4×	22 0.4×	66 1.3×	39 0.8×	10	307
Esben Gjerløff Wedebye Schmidt Denmark	8	201 1.1×	168 1.2×	39 0.7×	27 0.6×	49 1.1×	14	386
Paul Engeroff Switzerland	11	211 1.1×	96 0.7×	36 0.7×	50 1.0×	14 0.3×	21	488
Esther W. Taanman-Kueter Netherlands	6	443 2.4×	128 0.9×	54 1.0×	41 0.8×	43 0.9×	9	574
Kalyan C. Nallaparaju United States	7	297 1.6×	102 0.7×	32 0.6×	16 0.3×	67 1.5×	9	423
Christopher Jakobs Germany	9	271 1.5×	227 1.6×	28 0.5×	29 0.6×	35 0.8×	10	389
Sarah Siddiqui Canada	10	236 1.3×	98 0.7×	58 1.1×	51 1.0×	37 0.8×	13	350
Felix M. Behr Netherlands	13	537 2.9×	112 0.8×	91 1.7×	54 1.1×	173 3.8×	17	762

Countries citing papers authored by David I. Weiss

Since Specialization

Citations

This map shows the geographic impact of David I. Weiss's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David I. Weiss with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David I. Weiss more than expected).

Fields of papers citing papers by David I. Weiss

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David I. Weiss. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David I. Weiss. The network helps show where David I. Weiss may publish in the future.

Co-authorship network of co-authors of David I. Weiss

This figure shows the co-authorship network connecting the top 25 collaborators of David I. Weiss. A scholar is included among the top collaborators of David I. Weiss based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David I. Weiss. David I. Weiss is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Meermeier, Erin W., Christina Zheng, Aneta Worley, et al.. (2022). Human lung-resident mucosal-associated invariant T cells are abundant, express antimicrobial proteins, and are cytokine responsive. Communications Biology. 5(1). 942–942. 21 indexed citations

2.

Hughes, Tudor, Marc H. Wadsworth, Todd M. Gierahn, et al.. (2020). Second-Strand Synthesis-Based Massively Parallel scRNA-Seq Reveals Cellular States and Molecular Features of Human Inflammatory Skin Pathologies. Immunity. 53(4). 878–894.e7. 150 indexed citations

3.

Dang, Angeline Tilly, Rosane M. B. Teles, David I. Weiss, et al.. (2019). IL-26 contributes to host defense against intracellular bacteria. Journal of Clinical Investigation. 129(5). 1926–1939. 46 indexed citations

4.

Weiss, David I., Feiyang Ma, Alexander A. Merleev, et al.. (2019). IL-1β Induces the Rapid Secretion of the Antimicrobial Protein IL-26 from Th17 Cells. The Journal of Immunology. 203(4). 911–921. 22 indexed citations

5.

Balin, Samuel J., Matteo Pellegrini, Eynav Klechevsky, et al.. (2018). Human antimicrobial cytotoxic T lymphocytes, defined by NK receptors and antimicrobial proteins, kill intracellular bacteria. Science Immunology. 3(26). 59 indexed citations

6.

Weiss, David I., Brian Young, Antonella Rella, et al.. (2013). A Novel Small Molecule Methyltransferase Is Important for Virulence inCandida albicans. ACS Chemical Biology. 8(12). 2785–2793. 6 indexed citations

7.

Young, Brian D., David I. Weiss, Cecilia I. Zurita‐Lopez, et al.. (2012). Identification of Methylated Proteins in the Yeast Small Ribosomal Subunit: A Role for SPOUT Methyltransferases in Protein Arginine Methylation. Biochemistry. 51(25). 5091–5104. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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