David H. Kavanagh

4.4k total citations
9 papers, 362 citations indexed

About

David H. Kavanagh is a scholar working on Genetics, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, David H. Kavanagh has authored 9 papers receiving a total of 362 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Genetics, 5 papers in Molecular Biology and 1 paper in Pathology and Forensic Medicine. Recurrent topics in David H. Kavanagh's work include Genetic Associations and Epidemiology (4 papers), Wnt/β-catenin signaling in development and cancer (3 papers) and Genomic variations and chromosomal abnormalities (3 papers). David H. Kavanagh is often cited by papers focused on Genetic Associations and Epidemiology (4 papers), Wnt/β-catenin signaling in development and cancer (3 papers) and Genomic variations and chromosomal abnormalities (3 papers). David H. Kavanagh collaborates with scholars based in United Kingdom, Ireland and United States. David H. Kavanagh's co-authors include Michael O’Donovan, Katherine E. Tansey, Michael J. Owen, James Walters, Steven A. McCarroll, Andrew Pocklington, Peter Holmans, Elliott Rees, Jennifer L. Moran and Marc P. Forrest and has published in prestigious journals such as Neuron, PLoS ONE and Biochemical Journal.

In The Last Decade

David H. Kavanagh

9 papers receiving 360 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David H. Kavanagh United Kingdom	8	205	179	78	61	30	9	362
Anke Van Dijck Belgium	10	213 1.0×	223 1.2×	112 1.4×	80 1.3×	16 0.5×	18	403
Yanyan Ruan China	13	179 0.9×	179 1.0×	119 1.5×	53 0.9×	12 0.4×	23	387
William S. Ulrich United States	6	272 1.3×	202 1.1×	45 0.6×	58 1.0×	54 1.8×	8	417
Prashanth Rajarajan United States	9	290 1.4×	173 1.0×	34 0.4×	34 0.6×	13 0.4×	18	424
Masayuki Ide Japan	8	133 0.6×	69 0.4×	45 0.6×	80 1.3×	19 0.6×	20	256
Liam Crapper Canada	8	312 1.5×	134 0.7×	62 0.8×	58 1.0×	66 2.2×	9	483
Jue Ji China	12	177 0.9×	148 0.8×	37 0.5×	68 1.1×	41 1.4×	27	378
Jayon Lihm United States	5	186 0.9×	238 1.3×	86 1.1×	26 0.4×	12 0.4×	7	361
Wenjia Song United States	6	228 1.1×	185 1.0×	146 1.9×	73 1.2×	10 0.3×	13	427
Sandra Demars United States	4	245 1.2×	204 1.1×	39 0.5×	128 2.1×	15 0.5×	5	423

Countries citing papers authored by David H. Kavanagh

Since Specialization

Citations

This map shows the geographic impact of David H. Kavanagh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David H. Kavanagh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David H. Kavanagh more than expected).

Fields of papers citing papers by David H. Kavanagh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David H. Kavanagh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David H. Kavanagh. The network helps show where David H. Kavanagh may publish in the future.

Co-authorship network of co-authors of David H. Kavanagh

This figure shows the co-authorship network connecting the top 25 collaborators of David H. Kavanagh. A scholar is included among the top collaborators of David H. Kavanagh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David H. Kavanagh. David H. Kavanagh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

Forrest, Marc P., Matthew Hill, David H. Kavanagh, et al.. (2017). The Psychiatric Risk Gene Transcription Factor 4 (TCF4) Regulates Neurodevelopmental Pathways Associated With Schizophrenia, Autism, and Intellectual Disability. Schizophrenia Bulletin. 44(5). 1100–1110. 69 indexed citations

Richards, Alexander, Ganna Leonenko, James Walters, et al.. (2016). Exome arrays capture polygenic rare variant contributions to schizophrenia. Human Molecular Genetics. 25(5). 1001–1007. 35 indexed citations

Pocklington, Andrew, Elliott Rees, James Walters, et al.. (2015). Novel Findings from CNVs Implicate Inhibitory and Excitatory Signaling Complexes in Schizophrenia. Neuron. 86(5). 1203–1214. 126 indexed citations

McKay, Gareth J., David H. Kavanagh, John Crean, & Alexander P. Maxwell. (2015). Bioinformatic Evaluation of Transcriptional Regulation of WNT Pathway Genes with reference to Diabetic Nephropathy. Journal of Diabetes Research. 2016. 1–9. 7 indexed citations

Kavanagh, David H., Katherine E. Tansey, Michael O’Donovan, & Michael J. Owen. (2014). Schizophrenia genetics: emerging themes for a complex disorder. Molecular Psychiatry. 20(1). 72–76. 66 indexed citations

Kavanagh, David H., Sarah Dwyer, Michael O’Donovan, & Michael J. Owen. (2013). The ENCODE project: implications for psychiatric genetics. Molecular Psychiatry. 18(5). 540–542. 18 indexed citations

Kavanagh, David H., David A. Savage, Christopher Patterson, et al.. (2013). Haplotype association analysis of genes within the WNT signalling pathways in diabetic nephropathy. BMC Nephrology. 14(1). 126–126. 13 indexed citations

Kavanagh, David H., David A. Savage, Christopher Patterson, et al.. (2011). Association Analysis of Canonical Wnt Signalling Genes in Diabetic Nephropathy. PLoS ONE. 6(8). e23904–e23904. 8 indexed citations

Hickey, Fionnuala B., Derek P. Brazil, David H. Kavanagh, et al.. (2011). Connective tissue growth factor antagonizes transforming growth factor-β1/Smad signalling in renal mesangial cells. Biochemical Journal. 441(1). 499–510. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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