David A. Talmage

5.6k total citations · 1 hit paper
79 papers, 4.0k citations indexed

About

David A. Talmage is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, David A. Talmage has authored 79 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 53 papers in Molecular Biology, 28 papers in Cellular and Molecular Neuroscience and 19 papers in Genetics. Recurrent topics in David A. Talmage's work include Neuroscience and Neuropharmacology Research (20 papers), Retinoids in leukemia and cellular processes (16 papers) and Nicotinic Acetylcholine Receptors Study (15 papers). David A. Talmage is often cited by papers focused on Neuroscience and Neuropharmacology Research (20 papers), Retinoids in leukemia and cellular processes (16 papers) and Nicotinic Acetylcholine Receptors Study (15 papers). David A. Talmage collaborates with scholars based in United States, China and New Zealand. David A. Talmage's co-authors include Lorna W. Role, Mala Ananth, Elizabeth Ballinger, Young‐Hwan Jo, Deon Wolpowitz, Robert Freund, Jianxin Bao, Clyde J. Dawe, Thomas L. Benjamin and Melissa Hancock and has published in prestigious journals such as Nature, Cell and Nucleic Acids Research.

In The Last Decade

David A. Talmage

79 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Basal Forebrain Cholinergic Circuits and Signaling in Cognition and Cognitive Decline

2016 519 citations Elizabeth Ballinger, Mala Ananth et al. Neuron profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David A. Talmage United States	31	1.9k	1.3k	686	638	433	79	4.0k
Jan Mulder Sweden	35	1.6k 0.8×	1.4k 1.1×	609 0.9×	406 0.6×	285 0.7×	114	4.6k
Guoyin Feng China	40	2.6k 1.3×	933 0.7×	380 0.6×	430 0.7×	1.3k 3.0×	172	5.0k
Yuji Owada Japan	40	2.8k 1.5×	1.0k 0.8×	332 0.5×	194 0.3×	291 0.7×	178	5.1k
Georgy Bakalkin Sweden	35	2.2k 1.1×	1.6k 1.2×	466 0.7×	337 0.5×	232 0.5×	143	3.8k
Ted B. Usdin United States	42	3.3k 1.7×	2.9k 2.2×	757 1.1×	324 0.5×	268 0.6×	111	6.4k
Stephan von Hörsten Germany	46	2.0k 1.1×	3.1k 2.3×	1.2k 1.7×	383 0.6×	216 0.5×	192	6.8k
Jean‐Louis Nahon France	41	1.5k 0.8×	1.0k 0.8×	376 0.5×	901 1.4×	355 0.8×	104	5.4k
Etsuko Wada Japan	39	3.9k 2.0×	2.9k 2.2×	319 0.5×	296 0.5×	219 0.5×	79	6.4k
Michael F. Miles United States	37	3.0k 1.6×	1.5k 1.1×	237 0.3×	249 0.4×	563 1.3×	133	4.9k
Kyungmin Lee South Korea	27	1.3k 0.7×	1.1k 0.8×	210 0.3×	610 1.0×	509 1.2×	132	3.5k

Countries citing papers authored by David A. Talmage

Since Specialization

Citations

This map shows the geographic impact of David A. Talmage's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David A. Talmage with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David A. Talmage more than expected).

Fields of papers citing papers by David A. Talmage

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David A. Talmage. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David A. Talmage. The network helps show where David A. Talmage may publish in the future.

Co-authorship network of co-authors of David A. Talmage

This figure shows the co-authorship network connecting the top 25 collaborators of David A. Talmage. A scholar is included among the top collaborators of David A. Talmage based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David A. Talmage. David A. Talmage is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Rajebhosale, Prithviraj, Mala Ananth, Ronald Kim, et al.. (2024). Functionally refined encoding of threat memory by distinct populations of basal forebrain cholinergic projection neurons. eLife. 13. 4 indexed citations

Ananth, Mala, Prithviraj Rajebhosale, Ronald Kim, David A. Talmage, & Lorna W. Role. (2023). Basal forebrain cholinergic signalling: development, connectivity and roles in cognition. Nature reviews. Neuroscience. 24(4). 233–251. 61 indexed citations

Ananth, Mala, et al.. (2021). NeuroConstruct: 3D Reconstruction and Visualization of Neurites in Optical Microscopy Brain Images. IEEE Transactions on Visualization and Computer Graphics. 28(12). 4951–4965. 12 indexed citations

Crouse, Richard B., Kristen K.O. Kim, Hannah M. Batchelor, et al.. (2020). Acetylcholine is released in the basolateral amygdala in response to predictors of reward and enhances the learning of cue-reward contingency. eLife. 9. 57 indexed citations

Ballinger, Elizabeth, Christian P. Schaaf, Akash J. Patel, et al.. (2019). Mecp2Deletion from Cholinergic Neurons Selectively Impairs Recognition Memory and Disrupts Cholinergic Modulation of the Perirhinal Cortex. eNeuro. 6(6). ENEURO.0134–19.2019. 16 indexed citations

Záborszky, László, Péter Gombkötő, Matthew R. Gielow, et al.. (2018). Specific Basal Forebrain–Cortical Cholinergic Circuits Coordinate Cognitive Operations. Journal of Neuroscience. 38(44). 9446–9458. 134 indexed citations

Lloyd, David B., David A. Talmage, Cynthia Shannon Weickert, & Tim Karl. (2018). Reduced type III neuregulin 1 expression does not modulate the behavioural sensitivity of mice to acute Δ 9 -tetrahydrocannabinol (D 9 -THC). Pharmacology Biochemistry and Behavior. 170. 64–70. 5 indexed citations

Akmentin, Wendy, et al.. (2017). Axonal Type III Nrg1 Controls Glutamate Synapse Formation and GluA2 Trafficking in Hippocampal-Accumbens Connections. eNeuro. 4(1). ENEURO.0232–16.2017. 11 indexed citations

López‐Hernández, Gretchen Y., Mala Ananth, Li Jiang, et al.. (2017). Electrophysiological properties of basal forebrain cholinergic neurons identified by genetic and optogenetic tagging. Journal of Neurochemistry. 142(S2). 103–110. 14 indexed citations

10.

Fleming, Michael S., Jian J. Li, Tong Li, et al.. (2016). A RET-ER81-NRG1 Signaling Pathway Drives the Development of Pacinian Corpuscles. Journal of Neuroscience. 36(40). 10337–10355. 24 indexed citations

11.

Jiang, Li, Srikanya Kundu, Gretchen Y. López‐Hernández, et al.. (2016). Cholinergic Signaling Controls Conditioned Fear Behaviors and Enhances Plasticity of Cortical-Amygdala Circuits. Neuron. 90(5). 1057–1070. 133 indexed citations

12.

Escobar‐Hoyos, Luisa F., Lucia Roa‐Peña, Elizabeth A. Vanner, et al.. (2015). Keratin-17 Promotes p27KIP1 Nuclear Export and Degradation and Offers Potential Prognostic Utility. Cancer Research. 75(17). 3650–3662. 79 indexed citations

13.

Talmage, David A., et al.. (2013). Type III Neuregulin 1 Is Required for Multiple Forms of Excitatory Synaptic Plasticity of Mouse Cortico-Amygdala Circuits. Journal of Neuroscience. 33(23). 9655–9666. 30 indexed citations

14.

Canetta, Sarah, et al.. (2011). Type III Nrg1 Back Signaling Enhances Functional TRPV1 along Sensory Axons Contributing to Basal and Inflammatory Thermal Pain Sensation. PLoS ONE. 6(9). e25108–e25108. 14 indexed citations

15.

Johnson, Matthew A., Michael D. Lieberman, Rose E. Goodchild, et al.. (2008). Type III Neuregulin-1 Is Required for Normal Sensorimotor Gating, Memory-Related Behaviors, and Corticostriatal Circuit Components. Journal of Neuroscience. 28(27). 6872–6883. 149 indexed citations

16.

Berman, Joshua A., David A. Talmage, & Lorna W. Role. (2007). Cholinergic Circuits and Signaling in the Pathophysiology of Schizophrenia. International review of neurobiology. 78. 193–223. 36 indexed citations

17.

Talmage, David A., et al.. (1997). Decreased Cellular Retinol-Binding Protein Expression Coincides with the Loss of Retinol Responsiveness in Rat Cervical Epithelial Cells. Experimental Cell Research. 230(1). 38–44. 8 indexed citations

18.

Cho, Yunhi, Ann P. Tighe, & David A. Talmage. (1997). Retinoic acid induced growth arrest of human breast carcinoma cells requires protein kinase Cα expression and activity. Journal of Cellular Physiology. 172(3). 306–313. 43 indexed citations

19.

Hillemanns, Peter, et al.. (1992). Localization of cellular retinoid-binding proteins in human cervical intraepithelial neoplasia and invasive carcinoma.. PubMed Central. 141(4). 973–80. 18 indexed citations

20.

Garcea, Robert L., et al.. (1989). Separation of host range from transformation functions of the hr-t gene of polyomavirus. Virology. 168(2). 312–319. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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