Dat P. Ha

1.0k total citations
20 papers, 614 citations indexed

About

Dat P. Ha is a scholar working on Cell Biology, Epidemiology and Molecular Biology. According to data from OpenAlex, Dat P. Ha has authored 20 papers receiving a total of 614 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Cell Biology, 9 papers in Epidemiology and 7 papers in Molecular Biology. Recurrent topics in Dat P. Ha's work include Endoplasmic Reticulum Stress and Disease (15 papers), Autophagy in Disease and Therapy (7 papers) and Pancreatic function and diabetes (6 papers). Dat P. Ha is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (15 papers), Autophagy in Disease and Therapy (7 papers) and Pancreatic function and diabetes (6 papers). Dat P. Ha collaborates with scholars based in United States, Japan and Italy. Dat P. Ha's co-authors include Amy S. Lee, Anthony J. Carlos, Yuan-Li Tsai, Richard Van Krieken, Parkash S. Gill, Ze Liu, Keigo Machida, Min Xiong, Pu Zhang and Chun-Chih Tseng and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Oncogene.

In The Last Decade

Dat P. Ha

20 papers receiving 604 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Dat P. Ha United States 13 265 264 179 123 94 20 614
Yu-Hsuan Chen Taiwan 8 135 0.5× 387 1.5× 314 1.8× 43 0.3× 86 0.9× 13 639
Cheol‐Hee Yoon South Korea 14 67 0.3× 386 1.5× 94 0.5× 85 0.7× 227 2.4× 34 699
Jaewon Kim United States 12 274 1.0× 418 1.6× 56 0.3× 41 0.3× 71 0.8× 18 678
Tiziana Vescovo Italy 11 122 0.5× 274 1.0× 321 1.8× 25 0.2× 81 0.9× 13 603
Wei-Jian Zhang United States 6 86 0.3× 203 0.8× 94 0.5× 88 0.7× 194 2.1× 9 611
Lee Allers United States 11 219 0.8× 389 1.5× 485 2.7× 38 0.3× 207 2.2× 16 867
Darya A. Haas Germany 9 37 0.1× 369 1.4× 160 0.9× 95 0.8× 178 1.9× 12 747
Laura A. Castelli Australia 15 72 0.3× 575 2.2× 264 1.5× 127 1.0× 68 0.7× 24 868
Jutta Müller Germany 12 146 0.6× 267 1.0× 70 0.4× 20 0.2× 43 0.5× 18 549
Sophie Bellanger Singapore 14 96 0.4× 324 1.2× 262 1.5× 20 0.2× 123 1.3× 22 691

Countries citing papers authored by Dat P. Ha

Since Specialization
Citations

This map shows the geographic impact of Dat P. Ha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dat P. Ha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dat P. Ha more than expected).

Fields of papers citing papers by Dat P. Ha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dat P. Ha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dat P. Ha. The network helps show where Dat P. Ha may publish in the future.

Co-authorship network of co-authors of Dat P. Ha

This figure shows the co-authorship network connecting the top 25 collaborators of Dat P. Ha. A scholar is included among the top collaborators of Dat P. Ha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dat P. Ha. Dat P. Ha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Ze, et al.. (2025). Requirements for nuclear GRP78 transcriptional regulatory activities and interaction with nuclear GRP94. Journal of Biological Chemistry. 301(4). 108369–108369. 1 indexed citations
2.
Yamamoto, Vicky, Dat P. Ha, Ze Liu, et al.. (2024). GRP78 inhibitor YUM70 upregulates 4E-BP1 and suppresses c-MYC expression and viability of oncogenic c-MYC tumors. Neoplasia. 55. 101020–101020. 5 indexed citations
3.
Ha, Dat P., Woo‐Jin Shin, Ze Liu, et al.. (2024). Targeting stress induction of GRP78 by cardiac glycoside oleandrin dually suppresses cancer and COVID-19. Cell & Bioscience. 14(1). 115–115. 3 indexed citations
4.
Nguyen, Binh T., et al.. (2023). Microwave-assisted polyol liquefication from bamboo for bio-polyurethane foams fabrication. Journal of environmental chemical engineering. 11(2). 109605–109605. 12 indexed citations
5.
Ha, Dat P., Woo‐Jin Shin, Nouri Neamati, et al.. (2023). GRP78 Inhibitor YUM70 Suppresses SARS-CoV-2 Viral Entry, Spike Protein Production and Ameliorates Lung Damage. Viruses. 15(5). 1118–1118. 12 indexed citations
6.
Liu, Ze, et al.. (2023). ER chaperone GRP78/BiP translocates to the nucleus under stress and acts as a transcriptional regulator. Proceedings of the National Academy of Sciences. 120(31). e2303448120–e2303448120. 75 indexed citations
7.
Ha, Dat P., Bo Huang, Han Wang, et al.. (2022). Targeting GRP78 suppresses oncogenic KRAS protein expression and reduces viability of cancer cells bearing various KRAS mutations. Neoplasia. 33. 100837–100837. 17 indexed citations
8.
Ha, Dat P., Yuan-Li Tsai, & Amy S. Lee. (2021). Suppression of ER-stress induction of GRP78 as an anti-neoplastic mechanism of the cardiac glycoside Lanatoside C in pancreatic cancer. Neoplasia. 23(12). 1213–1226. 16 indexed citations
9.
Krieken, Richard Van, Yuan-Li Tsai, Anthony J. Carlos, Dat P. Ha, & Amy S. Lee. (2021). ER residential chaperone GRP78 unconventionally relocalizes to the cell surface via endosomal transport. Cellular and Molecular Life Sciences. 78(12). 5179–5195. 26 indexed citations
10.
Carlos, Anthony J., Dat P. Ha, Da‐Wei Yeh, et al.. (2021). The chaperone GRP78 is a host auxiliary factor for SARS-CoV-2 and GRP78 depleting antibody blocks viral entry and infection. Journal of Biological Chemistry. 296. 100759–100759. 118 indexed citations
11.
Dubeau, Louis, Ryan S. Park, Priscilla Chan, et al.. (2021). Endoplasmic reticulum chaperone GRP78/BiP is critical for mutant Kras-driven lung tumorigenesis. Oncogene. 40(20). 3624–3632. 23 indexed citations
12.
Ha, Dat P. & Amy S. Lee. (2020). Insulin-like growth factor 1-receptor signaling stimulates GRP78 expression through the PI3K/AKT/mTOR/ATF4 axis. Cellular Signalling. 75. 109736–109736. 17 indexed citations
13.
Tsai, Yuan-Li, Dat P. Ha, He Zhao, et al.. (2018). Endoplasmic reticulum stress activates SRC, relocating chaperones to the cell surface where GRP78/CD109 blocks TGF-β signaling. Proceedings of the National Academy of Sciences. 115(18). E4245–E4254. 100 indexed citations
14.
Bakewell, Suzanne J., Dat P. Ha, Tara Lee Costich, et al.. (2018). Suppression of stress induction of the 78-kilodalton glucose regulated protein (GRP78) in cancer by IT-139, an anti-tumor ruthenium small molecule inhibitor. Oncotarget. 9(51). 29698–29714. 32 indexed citations
15.
Ha, Dat P., Genyuan Zhu, Agnieszka Kobielak, et al.. (2017). GRP78 haploinsufficiency suppresses acinar-to-ductal metaplasia, signaling, and mutant Kras -driven pancreatic tumorigenesis in mice. Proceedings of the National Academy of Sciences. 114(20). E4020–E4029. 39 indexed citations
16.
Ha, Dat P., et al.. (2017). New role of endoplasmic reticulum chaperones in regulating metaplasia during tumorigenesis. Molecular & Cellular Oncology. 4(6). e1345350–e1345350. 1 indexed citations
17.
Nitta, Takayuki, et al.. (2015). Human and murine APOBEC3s restrict replication of koala retrovirus by different mechanisms. Retrovirology. 12(1). 68–68. 5 indexed citations
18.
19.
Stavrou, Spyridon, Takayuki Nitta, Dat P. Ha, et al.. (2013). Murine leukemia virus glycosylated Gag blocks apolipoprotein B editing complex 3 and cytosolic sensor access to the reverse transcription complex. Proceedings of the National Academy of Sciences. 110(22). 9078–9083. 74 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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