Darren Newton

2.3k total citations
47 papers, 1.6k citations indexed

About

Darren Newton is a scholar working on Immunology, Genetics and Molecular Biology. According to data from OpenAlex, Darren Newton has authored 47 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Immunology, 13 papers in Genetics and 9 papers in Molecular Biology. Recurrent topics in Darren Newton's work include Complement system in diseases (11 papers), Immune Cell Function and Interaction (10 papers) and Chronic Lymphocytic Leukemia Research (8 papers). Darren Newton is often cited by papers focused on Complement system in diseases (11 papers), Immune Cell Function and Interaction (10 papers) and Chronic Lymphocytic Leukemia Research (8 papers). Darren Newton collaborates with scholars based in United Kingdom, United States and Germany. Darren Newton's co-authors include Brian Flanagan, Peter Bundred, John T. Manning, Simon R. Carding, Elizabeth M. Andrew, Andy C. Rawstron, Dennis McGonagle, Charlie Bridgewood, Daniela Tramonti and Jane E. Dalton and has published in prestigious journals such as The Lancet, SHILAP Revista de lepidopterología and Blood.

In The Last Decade

Darren Newton

43 papers receiving 1.5k citations

Peers

Darren Newton
Antony J. Cutler United Kingdom
Stephen R. Brooks United States
Jill F. Wright United States
Ayşe Metìn Türkiye
Wer Ollier United Kingdom
Iván K. Chinn United States
K H Singer United States
Antony J. Cutler United Kingdom
Darren Newton
Citations per year, relative to Darren Newton Darren Newton (= 1×) peers Antony J. Cutler

Countries citing papers authored by Darren Newton

Since Specialization
Citations

This map shows the geographic impact of Darren Newton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Darren Newton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Darren Newton more than expected).

Fields of papers citing papers by Darren Newton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Darren Newton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Darren Newton. The network helps show where Darren Newton may publish in the future.

Co-authorship network of co-authors of Darren Newton

This figure shows the co-authorship network connecting the top 25 collaborators of Darren Newton. A scholar is included among the top collaborators of Darren Newton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Darren Newton. Darren Newton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Bridgewood, Charlie, Miriam Wittmann, Tom Macleod, et al.. (2022). T Helper 2 IL-4/IL-13 Dual Blockade with Dupilumab Is Linked to Some Emergent T Helper 17‒Type Diseases, Including Seronegative Arthritis and Enthesitis/Enthesopathy, but Not to Humoral Autoimmune Diseases. Journal of Investigative Dermatology. 142(10). 2660–2667. 74 indexed citations
3.
Bridgewood, Charlie, Darren Newton, Nicola Luigi Bragazzi, Miriam Wittmann, & Dennis McGonagle. (2021). Unexpected connections of the IL-23/IL-17 and IL-4/IL-13 cytokine axes in inflammatory arthritis and enthesitis. Seminars in Immunology. 58. 101520–101520. 35 indexed citations
4.
Cuthbert, Richard, ‬‬‬‬Abdulla Watad, Evangelos M. Fragkakis, et al.. (2019). Evidence that tissue resident human enthesis γδT-cells can produce IL-17A independently of IL-23R transcript expression. Annals of the Rheumatic Diseases. 78(11). 1559–1565. 120 indexed citations
6.
Richards, Stephen J., Talha Munir, Morag Griffin, et al.. (2017). Extravascular Hemolysis Due to C3-Loading in Patients with PNH Treated with Eculizumab: Defining the Clinical Syndrome. Blood. 130. 3471–3471. 21 indexed citations
7.
Rawstron, Andy C., Aloysius Ssemaganda, Ruth de Tute, et al.. (2017). Monoclonal B-cell lymphocytosis in a hospital-based UK population and a rural Ugandan population: a cross-sectional study. The Lancet Haematology. 4(7). e334–e340. 10 indexed citations
8.
Macrae, Fraser L., et al.. (2017). The prothrombotic state in paroxysmal nocturnal hemoglobinuria: a multifaceted source. Haematologica. 103(1). 9–17. 29 indexed citations
9.
Kwok, Marwan, Andy C. Rawstron, Abraham Varghese, et al.. (2016). Minimal residual disease is an independent predictor for 10-year survival in CLL. Blood. 128(24). 2770–2773. 95 indexed citations
10.
Walker, Catherine, Isabelle Hautefort, Jane E. Dalton, et al.. (2013). Intestinal Intraepithelial Lymphocyte-Enterocyte Crosstalk Regulates Production of Bactericidal Angiogenin 4 by Paneth Cells upon Microbial Challenge. PLoS ONE. 8(12). e84553–e84553. 55 indexed citations
11.
Rhodes, Katherine A., Elizabeth M. Andrew, Darren Newton, Daniela Tramonti, & Simon R. Carding. (2008). A subset of IL‐10‐producing γδ T cells protect the liver from Listeria‐elicited, CD8+ T cell‐mediated injury. European Journal of Immunology. 38(8). 2274–2283. 65 indexed citations
12.
Kirby, Alun C., Darren Newton, Simon R. Carding, & Paul M. Kaye. (2007). Evidence for the involvement of lung‐specific γδ T cell subsets in local responses to Streptococcus pneumoniae infection. European Journal of Immunology. 37(12). 3404–3413. 47 indexed citations
13.
Newton, Darren, et al.. (2006). Identification of Novel [gamma][delta] T-Cell Subsets following Bacterial Infection in the Absence of V[gamma]1⁺ T Cells: Homeostatic Control of [gamma][delta] T-Cell Responses to Pathogen Infection by V[gamma]1⁺ T Cells. Infection and Immunity. 2 indexed citations
14.
Dalton, Jane E., Sheena Cruickshank, Charlotte E. Egan, et al.. (2006). Intraepithelial γδ+ Lymphocytes Maintain the Integrity of Intestinal Epithelial Tight Junctions in Response to Infection. Gastroenterology. 131(3). 818–829. 107 indexed citations
15.
Andrew, Elizabeth M., Darren Newton, Jane E. Dalton, et al.. (2005). Delineation of the Function of a Major γδ T Cell Subset during Infection. The Journal of Immunology. 175(3). 1741–1750. 41 indexed citations
16.
Cruickshank, Sheena, Darren Newton, Anne Graham, et al.. (2004). Toll-like receptor-mediated responses of primary intestinal epithelial cells during the development of colitis. American Journal of Physiology-Gastrointestinal and Liver Physiology. 288(3). G514–G524. 63 indexed citations
17.
Newton, Darren, et al.. (1999). Human Decidualized Endometrial T Lymphocytes Do Not Substantially Down‐Regulate CD3ζ. American Journal of Reproductive Immunology. 41(4). 245–252. 6 indexed citations
18.
Flanagan, Brian, et al.. (1998). A novel isoform of human membrane cofactor protein (CD46) mRNA generated by intron retention. Gene. 212(1). 39–47. 14 indexed citations
19.
Newton, Darren, David Bowen-Jones, Ian T. Crosby, R.M.R. Barnes, & Brian Flanagan. (1998). TNFB gene polymorphism in insulin‐dependent diabetes mellitus: association with HLA‐DR alleles. European Journal of Immunogenetics. 25(1). 39–41. 5 indexed citations
20.
Williams, Cara, et al.. (1997). Conserved structure and tissue expression of rat eotaxin. Immunogenetics. 47(2). 178–180. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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