Daniel R. Matson

992 total citations
31 papers, 604 citations indexed

About

Daniel R. Matson is a scholar working on Molecular Biology, Hematology and Cell Biology. According to data from OpenAlex, Daniel R. Matson has authored 31 papers receiving a total of 604 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 9 papers in Hematology and 7 papers in Cell Biology. Recurrent topics in Daniel R. Matson's work include Acute Myeloid Leukemia Research (7 papers), Microtubule and mitosis dynamics (7 papers) and Immune Cell Function and Interaction (4 papers). Daniel R. Matson is often cited by papers focused on Acute Myeloid Leukemia Research (7 papers), Microtubule and mitosis dynamics (7 papers) and Immune Cell Function and Interaction (4 papers). Daniel R. Matson collaborates with scholars based in United States, Netherlands and Austria. Daniel R. Matson's co-authors include P. Todd Stukenberg, David T. Yang, John G. Tooley, William M. Rehrauer, Molly A. Accola, Gregory M. Alushin, Eva Nogales, Emery H. Bresnick, Daniel J. Burke and Heather Hardin and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Daniel R. Matson

29 papers receiving 598 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Daniel R. Matson United States 12 368 231 120 84 52 31 604
Eric J. Gapud United States 11 789 2.1× 97 0.4× 269 2.2× 151 1.8× 68 1.3× 21 959
Shui Hao China 13 249 0.7× 101 0.4× 154 1.3× 40 0.5× 71 1.4× 45 566
Ramon U. Jin United States 12 273 0.7× 94 0.4× 123 1.0× 70 0.8× 105 2.0× 26 570
Nina Korzeniewski Germany 15 425 1.2× 201 0.9× 175 1.5× 39 0.5× 117 2.3× 23 679
Yih-Shyong Lai Taiwan 14 205 0.6× 148 0.6× 48 0.4× 24 0.3× 36 0.7× 32 408
Erica Riveiro‐Falkenbach Spain 10 362 1.0× 55 0.2× 192 1.6× 94 1.1× 36 0.7× 25 575
Jeffrey Patterson-Fortin United States 11 578 1.6× 55 0.2× 227 1.9× 63 0.8× 42 0.8× 17 725
Byungho Lim South Korea 14 354 1.0× 69 0.3× 104 0.9× 42 0.5× 85 1.6× 25 563
Jiun‐Sheng Chen United States 11 353 1.0× 95 0.4× 251 2.1× 37 0.4× 46 0.9× 16 565

Countries citing papers authored by Daniel R. Matson

Since Specialization
Citations

This map shows the geographic impact of Daniel R. Matson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel R. Matson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel R. Matson more than expected).

Fields of papers citing papers by Daniel R. Matson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel R. Matson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel R. Matson. The network helps show where Daniel R. Matson may publish in the future.

Co-authorship network of co-authors of Daniel R. Matson

This figure shows the co-authorship network connecting the top 25 collaborators of Daniel R. Matson. A scholar is included among the top collaborators of Daniel R. Matson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniel R. Matson. Daniel R. Matson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Lasarev, Michael, et al.. (2023). Linking GATA2 to myeloid dysplasia and complex cytogenetics in adult myelodysplastic neoplasm and acute myeloid leukemia. Blood Advances. 8(1). 80–92. 4 indexed citations
3.
Ranheim, Erik A., Daniel R. Matson, Yun Zhou, et al.. (2023). A gain-of-function p53 mutant synergizes with oncogenic NRAS to promote acute myeloid leukemia in mice. Journal of Clinical Investigation. 133(24). 10 indexed citations
4.
Bennett, Daniel, et al.. (2023). Spatial transcriptomics of a giant pilomatricoma. Journal of Cutaneous Pathology. 50(11). 963–970. 1 indexed citations
6.
Janczyk, Paweł, et al.. (2022). Aurora A phosphorylates Ndel1 to reduce the levels of Mad1 and NuMA at spindle poles. Molecular Biology of the Cell. 34(1). br1–br1. 1 indexed citations
7.
Matson, Daniel R., et al.. (2022). Discriminating activities of DEAD-Box Helicase 41 from myeloid malignancy-associated germline variants by genetic rescue. Leukemia. 37(1). 235–239. 3 indexed citations
8.
Matson, Daniel R., et al.. (2021). Concurrent Central Diabetes Insipidus and Acute Myeloid Leukemia. SHILAP Revista de lepidopterología. 2021. 1–5. 3 indexed citations
9.
Smeenk, Leonie, Roger Mulet‐Lazaro, Anja Ebert, et al.. (2021). Selective Requirement of MYB for Oncogenic Hyperactivation of a Translocated Enhancer in Leukemia. Cancer Discovery. 11(11). 2868–2883. 29 indexed citations
10.
Matson, Daniel R., Ryan A. Denu, Lauren M. Zasadil, et al.. (2021). High nuclear TPX2 expression correlates with TP53 mutation and poor clinical behavior in a large breast cancer cohort, but is not an independent predictor of chromosomal instability. BMC Cancer. 21(1). 186–186. 21 indexed citations
11.
Chee, Linda, et al.. (2020). Sterile α-motif domain requirement for cellular signaling and survival. Journal of Biological Chemistry. 295(20). 7113–7125. 9 indexed citations
12.
Hewitt, Kyle J., Koichi R. Katsumura, Daniel R. Matson, et al.. (2017). GATA Factor-Regulated Samd14 Enhancer Confers Red Blood Cell Regeneration and Survival in Severe Anemia. Developmental Cell. 42(3). 213–225.e4. 30 indexed citations
13.
Bailey, Adam L., Connor R. Buechler, Daniel R. Matson, et al.. (2017). Pegivirus avoids immune recognition but does not attenuate acute-phase disease in a macaque model of HIV infection. PLoS Pathogens. 13(10). e1006692–e1006692. 11 indexed citations
14.
Matson, Daniel R., Heather Hardin, Darya Buehler, & Ricardo V. Lloyd. (2017). AKT activity is elevated in aggressive thyroid neoplasms where it promotes proliferation and invasion. Experimental and Molecular Pathology. 103(3). 288–293. 18 indexed citations
15.
Janczyk, Paweł, Katarzyna Skorupka, John G. Tooley, et al.. (2017). Mechanism of Ska Recruitment by Ndc80 Complexes to Kinetochores. Developmental Cell. 41(4). 438–449.e4. 39 indexed citations
16.
Alushin, Gregory M., et al.. (2012). Multimodal microtubule binding by the Ndc80 kinetochore complex. Nature Structural & Molecular Biology. 19(11). 1161–1167. 81 indexed citations
17.
Matson, Daniel R., et al.. (2012). A conserved role for COMA/CENP-H/I/N kinetochore proteins in the spindle checkpoint. Genes & Development. 26(6). 542–547. 29 indexed citations
18.
Matson, Daniel R. & P. Todd Stukenberg. (2011). Spindle Poisons and Cell Fate: A Tale of Two Pathways. Molecular Interventions. 11(2). 141–150. 104 indexed citations
19.
Matson, Daniel R., et al.. (2010). Cutaneous Alternariosis in an Adolescent Patient. Pediatric Dermatology. 27(1). 98–100. 6 indexed citations
20.
Zhang, Fan, et al.. (2005). Characteristics and actions of NAD(P)H oxidase on the sarcoplasmic reticulum of coronary artery smooth muscle. American Journal of Physiology-Heart and Circulatory Physiology. 290(3). H1136–H1144. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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