Damir Simic

858 total citations
20 papers, 279 citations indexed

About

Damir Simic is a scholar working on Molecular Biology, Ophthalmology and Clinical Biochemistry. According to data from OpenAlex, Damir Simic has authored 20 papers receiving a total of 279 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 3 papers in Ophthalmology and 3 papers in Clinical Biochemistry. Recurrent topics in Damir Simic's work include Mitochondrial Function and Pathology (3 papers), Metabolism and Genetic Disorders (3 papers) and Metabolomics and Mass Spectrometry Studies (2 papers). Damir Simic is often cited by papers focused on Mitochondrial Function and Pathology (3 papers), Metabolism and Genetic Disorders (3 papers) and Metabolomics and Mass Spectrometry Studies (2 papers). Damir Simic collaborates with scholars based in United States, Belgium and United Kingdom. Damir Simic's co-authors include Monicah A. Otieno, Jan Snoeys, Teng Fang, Geraldine A. Hamilton, Katia Karalis, Jacob P. Fraser, Abhishek Jain, Andries D. van der Meer, Mimi Zhou and Riccardo Barrile and has published in prestigious journals such as Clinical Pharmacology & Therapeutics, Investigative Ophthalmology & Visual Science and Toxicology and Applied Pharmacology.

In The Last Decade

Damir Simic

18 papers receiving 271 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Damir Simic United States 9 100 87 47 43 42 20 279
Jenna L. Voellinger United States 9 170 1.7× 157 1.8× 33 0.7× 106 2.5× 33 0.8× 11 408
Pedro Caetano-Pinto Germany 11 168 1.7× 126 1.4× 22 0.5× 90 2.1× 28 0.7× 23 360
Gary L. Engelmann United States 11 182 1.8× 24 0.3× 41 0.9× 59 1.4× 48 1.1× 15 421
Yuanyi Wei China 12 196 2.0× 13 0.1× 26 0.6× 76 1.8× 28 0.7× 29 349
Florian Büttner Germany 13 297 3.0× 35 0.4× 21 0.4× 117 2.7× 36 0.9× 24 474
Sameerah Shaheen Saudi Arabia 10 137 1.4× 26 0.3× 19 0.4× 83 1.9× 28 0.7× 25 314
Zhilin Chen China 11 98 1.0× 21 0.2× 48 1.0× 64 1.5× 52 1.2× 32 319
Bernard Kneip France 10 167 1.7× 41 0.5× 61 1.3× 56 1.3× 80 1.9× 20 387
Ingrid H. Hof Netherlands 10 87 0.9× 52 0.6× 123 2.6× 84 2.0× 134 3.2× 13 380
Shree Bose United States 8 229 2.3× 83 1.0× 6 0.1× 91 2.1× 33 0.8× 11 450

Countries citing papers authored by Damir Simic

Since Specialization
Citations

This map shows the geographic impact of Damir Simic's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Damir Simic with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Damir Simic more than expected).

Fields of papers citing papers by Damir Simic

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Damir Simic. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Damir Simic. The network helps show where Damir Simic may publish in the future.

Co-authorship network of co-authors of Damir Simic

This figure shows the co-authorship network connecting the top 25 collaborators of Damir Simic. A scholar is included among the top collaborators of Damir Simic based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Damir Simic. Damir Simic is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chen, Nancy, David E. Ehmann, Katayoun Derakhchan, et al.. (2023). Gene therapy for cross-correction of somatic organs and the CNS in mucopolysaccharidosis II in rodents and non-human primates. Molecular Therapy — Methods & Clinical Development. 29. 286–302. 4 indexed citations
2.
Kamalian, Laleh, Rosalind E. Jenkins, Alison J. Beckett, et al.. (2020). The utility of a differentiated preclinical liver model, HepaRG cells, in investigating delayed toxicity via inhibition of mitochondrial-replication induced by fialuridine. Toxicology and Applied Pharmacology. 403. 115163–115163. 8 indexed citations
3.
Simic, Damir & Nianli Sang. (2019). Compounds targeting class II histone deacetylases do not cause panHDACI-associated impairment of megakaryocyte differentiation. Experimental Hematology. 72. 36–46. 2 indexed citations
4.
Kamalian, Laleh, Jan Snoeys, Damir Simic, et al.. (2019). Acute Metabolic Switch Assay Using Glucose/Galactose Medium in HepaRG Cells to Detect Mitochondrial Toxicity. Current Protocols in Toxicology. 80(1). e76–e76. 12 indexed citations
5.
Kamalian, Laleh, Jan Snoeys, Damir Simic, et al.. (2018). The utility of HepaRG cells for bioenergetic investigation and detection of drug-induced mitochondrial toxicity. Toxicology in Vitro. 53. 136–147. 35 indexed citations
6.
Barrile, Riccardo, Andries D. van der Meer, Hyoungshin Park, et al.. (2018). Organ‐on‐Chip Recapitulates Thrombosis Induced by an anti‐CD154 Monoclonal Antibody: Translational Potential of Advanced Microengineered Systems. Clinical Pharmacology & Therapeutics. 104(6). 1240–1248. 98 indexed citations
7.
Jang, Kyung‐Jin, Monicah A. Otieno, Janey Ronxhi, et al.. (2018). Liver-chip identifies mitochondrial dysfunction, oxidative stress, and innate immune response as potential pathways of toxicity for the GPR40 agonist TAK-875. Toxicology Letters. 295. S129–S130. 1 indexed citations
8.
Otieno, Monicah A., Jan Snoeys, Wing Lam, et al.. (2017). Fasiglifam (TAK-875): Mechanistic Investigation and Retrospective Identification of Hazards for Drug Induced Liver Injury. Toxicological Sciences. 163(2). 374–384. 66 indexed citations
9.
Guha, Mausumee, James K. Hennan, Damir Simic, et al.. (2017). In vitro and in vivo evaluation of dasatinib and imatinib on physiological parameters of pulmonary arterial hypertension. Cancer Chemotherapy and Pharmacology. 79(4). 711–723. 12 indexed citations
10.
Wang, Faye, Jae Kwagh, Megan K. Fuller, et al.. (2016). Effects of BMS-986094, a Guanosine Nucleotide Analogue, on Mitochondrial DNA Synthesis and Function. Toxicological Sciences. 153(2). 396–408. 10 indexed citations
11.
Simic, Damir, Nancy Bogdan, Teng Fang, & Monicah A. Otieno. (2015). Blocking α5β1 Integrin Attenuates sCD40L-Mediated Platelet Activation. Clinical and Applied Thrombosis/Hemostasis. 23(6). 607–614. 11 indexed citations
12.
13.
Vleet, Terry R. Van, Damir Simic, Theodora W. Salcedo, et al.. (2014). d‐α‐tocopheryl polyethylene glycol 1000 succinate‐containing vehicles provide no detectable chemoprotection from oxidative damage. Journal of Applied Toxicology. 35(7). 791–798. 1 indexed citations
14.
Simic, Damir, Emily R. Haines, Aiqing He, et al.. (2013). MicroRNA changes associated with atypical CYP1A1 inducer BMS-764459. Toxicology. 311(3). 169–177. 3 indexed citations
15.
Simic, Damir, et al.. (2013). Determination of Relative Notch1 and γ-Secretase-Related Gene Expression in Puromycin-Treated Microdissected Rat Kidneys. Gene Expression. 16(1). 39–47. 2 indexed citations
16.
Simic, Damir, et al.. (2012). Assessing Cell Fusion and Cytokinesis Failure as Mechanisms of Clone 9 Hepatocyte Multinucleation In Vitro. Current Protocols in Toxicology. 53(1). Unit 14.9.1–17. 3 indexed citations
17.
18.
Simic, Damir, et al.. (2008). Drug-Induced Lens Toxicity and Differential Expression of miRNA. Investigative Ophthalmology & Visual Science. 49(13). 2780–2780. 1 indexed citations
19.
Simic, Damir, et al.. (2006). Investigating Cytoskeletal Alterations as a Potential Marker of Retinal and Lens Drug-Related Toxicity. Assay and Drug Development Technologies. 4(6). 695–707. 9 indexed citations
20.
Simic, Damir, et al.. (2004). Early MNU–induced retinal toxicity detected by electroretinography and visual acuity in funduscopically normal rats. Investigative Ophthalmology & Visual Science. 45(13). 804–804. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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