Cris M. Welling

2.2k total citations
11 papers, 1.2k citations indexed

About

Cris M. Welling is a scholar working on Surgery, Molecular Biology and Genetics. According to data from OpenAlex, Cris M. Welling has authored 11 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Surgery, 7 papers in Molecular Biology and 5 papers in Genetics. Recurrent topics in Cris M. Welling's work include Pancreatic function and diabetes (10 papers), Metabolism, Diabetes, and Cancer (5 papers) and Genetics and Neurodevelopmental Disorders (4 papers). Cris M. Welling is often cited by papers focused on Pancreatic function and diabetes (10 papers), Metabolism, Diabetes, and Cancer (5 papers) and Genetics and Neurodevelopmental Disorders (4 papers). Cris M. Welling collaborates with scholars based in United States, Switzerland and Canada. Cris M. Welling's co-authors include M. Alan Permutt, Ernesto Bernal‐Mizrachi, Wen Wu, Yukio Tanizawa, László Korányi, Mitsuru Ohsugi, J. Murray, J. Wasson, Szabolcs Fátrai and Robert E. Schmidt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and PLoS ONE.

In The Last Decade

Cris M. Welling

11 papers receiving 1.2k citations

Peers

Cris M. Welling

SURGE

MB

GENET

EDM

CB

Søren M. Echwald Denmark

Isabelle Briaud United States

Carlo Colombo Italy

Z. Ling Belgium

S Hügl Germany

Xingjun Jing Sweden

Cristina Alarcón United States

Katsuya Tanabe Japan

Mira M. Sachdeva United States

Mary K. Treutelaar United States

Søren M. Echwald Denmark

Cris M. Welling

393 ×0.5

SURGE

616 ×0.9

MB

348 ×0.9

GENET

288 ×0.9

EDM

129 ×0.6

CB

Citations per year, relative to Cris M. Welling Cris M. Welling (= 1×) peers Søren M. Echwald

Countries citing papers authored by Cris M. Welling

Since Specialization

Citations

This map shows the geographic impact of Cris M. Welling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cris M. Welling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cris M. Welling more than expected).

Fields of papers citing papers by Cris M. Welling

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cris M. Welling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cris M. Welling. The network helps show where Cris M. Welling may publish in the future.

Co-authorship network of co-authors of Cris M. Welling

This figure shows the co-authorship network connecting the top 25 collaborators of Cris M. Welling. A scholar is included among the top collaborators of Cris M. Welling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cris M. Welling. Cris M. Welling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Tanabe, Katsuya, Yang Liu, Sara C. Martinez, et al.. (2011). Glucose and Fatty Acids Synergize to Promote B-Cell Apoptosis through Activation of Glycogen Synthase Kinase 3β Independent of JNK Activation. PLoS ONE. 6(4). e18146–e18146. 38 indexed citations

2.

Tanabe, Katsuya, Zhonghao Liu, Satish Patel, et al.. (2008). Genetic Deficiency of Glycogen Synthase Kinase-3β Corrects Diabetes in Mouse Models of Insulin Resistance. PLoS Biology. 6(2). e37–e37. 97 indexed citations

3.

Bernal‐Mizrachi, Ernesto, Mitsuru Ohsugi, J. Wasson, et al.. (2005). Mice conditionally lacking the Wolfram gene in pancreatic islet beta cells exhibit diabetes as a result of enhanced endoplasmic reticulum stress and apoptosis. Diabetologia. 48(11). 2313–2321. 177 indexed citations

4.

Bernal‐Mizrachi, Ernesto, et al.. (2001). Islet β cell expression of constitutively active Akt1/PKBα induces striking hypertrophy, hyperplasia, and hyperinsulinemia. Journal of Clinical Investigation. 108(11). 1631–1638. 324 indexed citations

5.

Bernal‐Mizrachi, Ernesto, et al.. (2001). Islet β cell expression of constitutively active Akt1/PKBα induces striking hypertrophy, hyperplasia, and hyperinsulinemia. Journal of Clinical Investigation. 108(11). 1631–1638. 296 indexed citations

6.

Inoue, Hiroshi, Christopher A. Iannotti, Cris M. Welling, et al.. (1997). Human Cholecystokinin Type A Receptor Gene: Cytogenetic Localization, Physical Mapping, and Identification of Two Missense Variants in Patients with Obesity and Non-Insulin-Dependent Diabetes Mellitus (NIDDM). Genomics. 42(2). 331–335. 40 indexed citations

7.

Inoue, Hiroshi, Jorge Ferrer, Cris M. Welling, et al.. (1996). Sequence Variants in the Sulfonylurea Receptor (SUR) Gene Are Associated With NIDDM in Caucasians. Diabetes. 45(6). 825–831. 110 indexed citations

8.

Olansky, Leann, Cris M. Welling, Stephen J. Giddings, et al.. (1992). A variant insulin promoter in non-insulin-dependent diabetes mellitus.. Journal of Clinical Investigation. 89(5). 1596–1602. 33 indexed citations

9.

Korányi, László, Yukio Tanizawa, Cris M. Welling, Daniel U. Rabin, & M. Alan Permutt. (1992). Human Islet Glucokinase Gene: Isolation and Sequence Analysis of Full-Length cDNA. Diabetes. 41(7). 807–811. 36 indexed citations

10.

Olansky, Leann, Rachel C. Janssen, Cris M. Welling, & M. Alan Permutt. (1992). Variability of the Insulin Gene in American Blacks With NIDDM: Analysis by Single-Strand Conformational Polymorphisms. Diabetes. 41(6). 742–749. 35 indexed citations

11.

Tanizawa, Yukio, László Korányi, Cris M. Welling, & M. Alan Permutt. (1991). Human liver glucokinase gene: cloning and sequence determination of two alternatively spliced cDNAs.. Proceedings of the National Academy of Sciences. 88(16). 7294–7297. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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