Craig J. Burd

1.4k total citations
33 papers, 1.0k citations indexed

About

Craig J. Burd is a scholar working on Oncology, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Craig J. Burd has authored 33 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Oncology, 17 papers in Molecular Biology and 10 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Craig J. Burd's work include Estrogen and related hormone effects (10 papers), Cancer-related Molecular Pathways (9 papers) and Prostate Cancer Treatment and Research (8 papers). Craig J. Burd is often cited by papers focused on Estrogen and related hormone effects (10 papers), Cancer-related Molecular Pathways (9 papers) and Prostate Cancer Treatment and Research (8 papers). Craig J. Burd collaborates with scholars based in United States, India and Australia. Craig J. Burd's co-authors include Karen E. Knudsen, Trevor Archer, Lisa M. Morey, Thomas W. Marshall, Erin Williams, Christin E. Petre, Erik S. Knudsen, Erin C. Henry, Gary B. Rosson and Bernard E. Weissman and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Craig J. Burd

33 papers receiving 1.0k citations

Peers

Craig J. Burd
Craig J. Burd
Citations per year, relative to Craig J. Burd Craig J. Burd (= 1×) peers А. И. Карселадзе

Countries citing papers authored by Craig J. Burd

Since Specialization
Citations

This map shows the geographic impact of Craig J. Burd's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Craig J. Burd with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Craig J. Burd more than expected).

Fields of papers citing papers by Craig J. Burd

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Craig J. Burd. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Craig J. Burd. The network helps show where Craig J. Burd may publish in the future.

Co-authorship network of co-authors of Craig J. Burd

This figure shows the co-authorship network connecting the top 25 collaborators of Craig J. Burd. A scholar is included among the top collaborators of Craig J. Burd based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Craig J. Burd. Craig J. Burd is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Aguilar‐Valenzuela, Renan, et al.. (2023). The OSUMMER lines: A series of ultraviolet‐accelerated NRAS‐mutant mouse melanoma cell lines syngeneic to C57BL/6. Pigment Cell & Melanoma Research. 36(5). 365–377. 2 indexed citations
2.
Terrell, Elizabeth M., Venkat R. Chirasani, Min Chen, et al.. (2022). Enhanced BRAF engagement by NRAS mutants capable of promoting melanoma initiation. Nature Communications. 13(1). 3153–3153. 23 indexed citations
3.
Liu, Tong, Lianbo Yu, John A. D’Orazio, et al.. (2022). Cell‐intrinsic melanin fails to protect melanocytes from ultraviolet‐mutagenesis in the absence of epidermal melanin. Pigment Cell & Melanoma Research. 36(1). 6–18. 4 indexed citations
4.
Bowman, Robert L., Amy Webb, Krista M. D. La Perle, et al.. (2021). UVB mutagenesis differs in Nras- and Braf-mutant mouse models of melanoma. Life Science Alliance. 4(9). e202101135–e202101135. 13 indexed citations
5.
Aguilar‐Valenzuela, Renan, et al.. (2020). In utero estrogenic endocrine disruption alters the stroma to increase extracellular matrix density and mammary gland stiffness. Breast Cancer Research. 22(1). 41–41. 21 indexed citations
6.
Mo, Xiaokui, et al.. (2018). GREB1 isoforms regulate proliferation independent of ERα co-regulator activities in breast cancer. Endocrine Related Cancer. 25(7). 735–746. 15 indexed citations
7.
Lavender, Christopher A., Jackson A. Hoffman, Kevin W. Trotter, et al.. (2016). Downstream Antisense Transcription Predicts Genomic Features That Define the Specific Chromatin Environment at Mammalian Promoters. PLoS Genetics. 12(8). e1006224–e1006224. 14 indexed citations
8.
Young, Nicholas A., Lai‐Chu Wu, Craig J. Burd, et al.. (2016). Estrogen-regulated STAT1 activation promotes TLR8 expression to facilitate signaling via microRNA-21 in systemic lupus erythematosus. Clinical Immunology. 176. 12–22. 48 indexed citations
9.
Young, Nicholas A., Lai‐Chu Wu, Craig J. Burd, et al.. (2014). Estrogen modulation of endosome-associated toll-like receptor 8: An IFNα-independent mechanism of sex-bias in systemic lupus erythematosus. Clinical Immunology. 151(1). 66–77. 67 indexed citations
10.
Augello, Michael A., Craig J. Burd, Ruth Birbe, et al.. (2012). Convergence of oncogenic and hormone receptor pathways promotes metastatic phenotypes. Journal of Clinical Investigation. 123(1). 493–508. 35 indexed citations
11.
Burd, Craig J., James M. Ward, Grace E. Kissling, et al.. (2012). Analysis of Chromatin Dynamics during Glucocorticoid Receptor Activation. Molecular and Cellular Biology. 32(10). 1805–1817. 29 indexed citations
12.
Comstock, Clay E.S., Michael A. Augello, Matthew J. Schiewer, et al.. (2011). Cyclin D1 Is a Selective Modifier of Androgen-dependent Signaling and Androgen Receptor Function. Journal of Biological Chemistry. 286(10). 8117–8127. 30 indexed citations
13.
Burd, Craig J., H. Karimi Kinyamu, Frederick W. Miller, & Trevor Archer. (2008). UV Radiation Regulates Mi-2 through Protein Translation and Stability. Journal of Biological Chemistry. 283(50). 34976–34982. 49 indexed citations
14.
Schiewer, Matthew J., Lisa M. Morey, Craig J. Burd, et al.. (2008). Cyclin D1 repressor domain mediates proliferation and survival in prostate cancer. Oncogene. 28(7). 1016–1027. 21 indexed citations
15.
Burd, Craig J., Lisa M. Morey, & Karen E. Knudsen. (2006). Androgen receptor corepressors and prostate cancer. Endocrine Related Cancer. 13(4). 979–994. 53 indexed citations
16.
Burd, Craig J., Christin E. Petre, Lisa M. Morey, et al.. (2006). Cyclin D1b variant influences prostate cancer growth through aberrant androgen receptor regulation. Proceedings of the National Academy of Sciences. 103(7). 2190–2195. 99 indexed citations
17.
Link, Kevin A., Craig J. Burd, Erin Williams, et al.. (2005). BAF57 Governs Androgen Receptor Action and Androgen-Dependent Proliferation through SWI/SNF. Molecular and Cellular Biology. 25(6). 2200–2215. 100 indexed citations
18.
Williams, Erin, Craig J. Burd, Andrew B. Gladden, et al.. (2004). A central domain of cyclin D1 mediates nuclear receptor corepressor activity. Oncogene. 24(3). 431–444. 56 indexed citations
19.
Liby, Karen T., Bonnie Neltner, Lisa Mohamet, Craig J. Burd, & Nira Ben‐Jonathan. (2003). Endostatin Expression by MDA-MB-435 Breast Cancer Cells Effectively Inhibits Tumor Growth. Cancer Biology & Therapy. 2(1). 49–53. 11 indexed citations
20.
Burd, Craig J., et al.. (1997). α and β Phases of 4-Aminoquinoline-2-carboxylic Acid Monohydrate. Acta Crystallographica Section C Crystal Structure Communications. 53(5). 602–605. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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