Cornelia M. Spies

822 total citations
17 papers, 597 citations indexed

About

Cornelia M. Spies is a scholar working on Endocrinology, Diabetes and Metabolism, Genetics and Immunology. According to data from OpenAlex, Cornelia M. Spies has authored 17 papers receiving a total of 597 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Endocrinology, Diabetes and Metabolism, 7 papers in Genetics and 5 papers in Immunology. Recurrent topics in Cornelia M. Spies's work include Estrogen and related hormone effects (6 papers), Adrenal Hormones and Disorders (6 papers) and Systemic Lupus Erythematosus Research (4 papers). Cornelia M. Spies is often cited by papers focused on Estrogen and related hormone effects (6 papers), Adrenal Hormones and Disorders (6 papers) and Systemic Lupus Erythematosus Research (4 papers). Cornelia M. Spies collaborates with scholars based in Germany, Netherlands and Australia. Cornelia M. Spies's co-authors include Frank Buttgereit, Timo Gaber, Cindy Strehl, Gerd R Burmester, J. W. J. Bijlsma, Alexander Scheffold, Andreas Radbruch, Rainer H. Straub, Tímea Berki and Roland Lauster and has published in prestigious journals such as The FASEB Journal, Annals of the Rheumatic Diseases and Bone.

In The Last Decade

Cornelia M. Spies

16 papers receiving 581 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Cornelia M. Spies Germany	13	160	155	143	123	113	17	597
Masanori A. Murayama Japan	16	157 1.0×	277 1.8×	335 2.3×	55 0.4×	69 0.6×	61	955
Giorgio Caratti Germany	10	152 0.9×	131 0.8×	101 0.7×	66 0.5×	35 0.3×	14	468
M C Gagnerault France	12	338 2.1×	138 0.9×	351 2.5×	194 1.6×	33 0.3×	17	808
Kazuhiro Ishizaka Japan	16	86 0.5×	309 2.0×	145 1.0×	135 1.1×	38 0.3×	58	1.1k
Gopesh Srivastava Hong Kong	15	132 0.8×	206 1.3×	101 0.7×	37 0.3×	37 0.3×	30	646
Jimena Druker Argentina	13	180 1.1×	298 1.9×	99 0.7×	136 1.1×	22 0.2×	19	706
Laicheng Wang China	19	263 1.6×	323 2.1×	374 2.6×	96 0.8×	93 0.8×	53	1.1k
Ahmad Pazirandeh Sweden	10	175 1.1×	140 0.9×	110 0.8×	103 0.8×	12 0.1×	10	496
A R McDonald United States	9	81 0.5×	206 1.3×	189 1.3×	171 1.4×	25 0.2×	11	575
Sybille D. Reichardt Germany	11	219 1.4×	100 0.6×	65 0.5×	51 0.4×	22 0.2×	20	467

Countries citing papers authored by Cornelia M. Spies

Since Specialization

Citations

This map shows the geographic impact of Cornelia M. Spies's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cornelia M. Spies with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cornelia M. Spies more than expected).

Fields of papers citing papers by Cornelia M. Spies

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cornelia M. Spies. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cornelia M. Spies. The network helps show where Cornelia M. Spies may publish in the future.

Co-authorship network of co-authors of Cornelia M. Spies

This figure shows the co-authorship network connecting the top 25 collaborators of Cornelia M. Spies. A scholar is included among the top collaborators of Cornelia M. Spies based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cornelia M. Spies. Cornelia M. Spies is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Koch, Susanne, et al.. (2016). Motor-nerve excitability changes in critically ill patients. Clinical Neurophysiology. 127(3). e27–e27.

Strehl, Cindy, Timo Gaber, Manuela Jakstadt, et al.. (2013). High-Sensitivity Immunofluorescence Staining: A Comparison of the Liposome Procedure and the FASER Technique on mGR Detection. Journal of Fluorescence. 23(3). 509–518. 8 indexed citations

Jacobs, Johannes W G, Cornelia M. Spies, J. W. J. Bijlsma, & Frank Buttgereit. (2013). Glucocorticoids. Oxford University Press eBooks. 1 indexed citations

Spies, Cornelia M., Rainer H. Straub, & Frank Buttgereit. (2012). Energy metabolism and rheumatic diseases: from cell to organism. Arthritis Research & Therapy. 14(3). 216–216. 32 indexed citations

Spies, Cornelia M., Timo Gaber, Paula Hoff, et al.. (2012). OP0086 Alterations of immune cellular circadian rhythms in rheumatoid arthritis. Annals of the Rheumatic Diseases. 71. 82–82. 1 indexed citations

Spies, Cornelia M., Cindy Strehl, Marlies C. van der Goes, J. W. J. Bijlsma, & Frank Buttgereit. (2011). Glucocorticoids. Best Practice & Research Clinical Rheumatology. 25(6). 891–900. 58 indexed citations

Strehl, Cindy, Timo Gaber, Mark Löwenberg, et al.. (2011). Origin and functional activity of the membrane-bound glucocorticoid receptor. Arthritis & Rheumatism. 63(12). 3779–3788. 57 indexed citations

Spies, Cornelia M., et al.. (2011). Prednisone chronotherapy.. PubMed. 29(5 Suppl 68). S42–5. 15 indexed citations

Strehl, Cindy, Cornelia M. Spies, & Frank Buttgereit. (2011). Pharmacodynamics of glucocorticoids.. PubMed. 29(5 Suppl 68). S13–8. 30 indexed citations

10.

Spies, Cornelia M., J. W. J. Bijlsma, Gerd R Burmester, & Frank Buttgereit. (2010). Pharmacology of glucocorticoids in rheumatoid arthritis. Current Opinion in Pharmacology. 10(3). 302–307. 39 indexed citations

11.

Spies, Cornelia M., Timo Gaber, M. Hahne, et al.. (2010). Rimexolone inhibits proliferation, cytokine expression and signal transduction of human CD4+ T-cells. Immunology Letters. 131(1). 24–32. 15 indexed citations

12.

Dziurla, René, Timo Gaber, Monique Fangradt, et al.. (2010). Effects of hypoxia and/or lack of glucose on cellular energy metabolism and cytokine production in stimulated human CD4+ T lymphocytes. Immunology Letters. 131(1). 97–105. 35 indexed citations

13.

Kalak, Robert, Hong Zhou, Janine Street, et al.. (2009). Endogenous glucocorticoid signalling in osteoblasts is necessary to maintain normal bone structure in mice. Bone. 45(1). 61–67. 57 indexed citations

14.

Buttgereit, Frank, Hong Zhou, Robert Kalak, et al.. (2009). Transgenic disruption of glucocorticoid signaling in mature osteoblasts and osteocytes attenuates K/BxN mouse serum–induced arthritis in vivo. Arthritis & Rheumatism. 60(7). 1998–2007. 43 indexed citations

15.

Spies, Cornelia M., D. Schaumann, Tímea Berki, et al.. (2006). Membrane glucocorticoid receptors are down regulated by glucocorticoids in patients with systemic lupus erythematosus and use a caveolin-1-independent expression pathway. Annals of the Rheumatic Diseases. 65(9). 1139–1146. 35 indexed citations

16.

Tryc, Anita B., Cornelia M. Spies, Udo Schneider, et al.. (2006). Membrane glucocorticoid receptor expression on peripheral blood mononuclear cells in patients with ankylosing spondylitis.. PubMed. 33(11). 2249–53. 17 indexed citations

17.

Spies, Cornelia M., Timo Gaber, Sebastian Schuchmann, et al.. (2004). Membrane glucocorticoid receptors (mGCR) are expressed in normal human peripheral blood mononuclear cells and up‐regulated after in vitro stimulation and in patients with rheumatoid arthritis. The FASEB Journal. 18(1). 70–80. 154 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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