Cong Ding

626 total citations
46 papers, 460 citations indexed

About

Cong Ding is a scholar working on Molecular Biology, Immunology and Cell Biology. According to data from OpenAlex, Cong Ding has authored 46 papers receiving a total of 460 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 12 papers in Immunology and 8 papers in Cell Biology. Recurrent topics in Cong Ding's work include Immune Response and Inflammation (5 papers), Ubiquitin and proteasome pathways (4 papers) and Immune Cell Function and Interaction (4 papers). Cong Ding is often cited by papers focused on Immune Response and Inflammation (5 papers), Ubiquitin and proteasome pathways (4 papers) and Immune Cell Function and Interaction (4 papers). Cong Ding collaborates with scholars based in China, United States and Japan. Cong Ding's co-authors include Diyi Chen, Xiaoyi Ma, Younghae Do, Yichen Wang, Qiaozhen Kang, Zhenyu Ji, Xin Liu, Yaodong Zhang, Na Liu and Sha Gong and has published in prestigious journals such as Nature Communications, SHILAP Revista de lepidopterología and Scientific Reports.

In The Last Decade

Cong Ding

43 papers receiving 454 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Cong Ding China 9 136 102 78 74 62 46 460
Yufan Wu China 18 183 1.3× 89 0.9× 26 0.3× 48 0.6× 61 1.0× 74 728
Qing Peng China 13 139 1.0× 46 0.5× 28 0.4× 31 0.4× 36 0.6× 55 572
Daisuke Kawashima Japan 17 195 1.4× 167 1.6× 142 1.8× 60 0.8× 42 0.7× 72 874
Baoling Liu China 11 110 0.8× 126 1.2× 36 0.5× 37 0.5× 43 0.7× 53 415
Weijiang Liu China 9 100 0.7× 17 0.2× 42 0.5× 28 0.4× 37 0.6× 20 406
Zhaolun Li China 14 172 1.3× 18 0.2× 19 0.2× 36 0.5× 123 2.0× 36 490
Fengbin Liu China 18 346 2.5× 51 0.5× 129 1.7× 209 2.8× 221 3.6× 49 894
Young Joon Song South Korea 10 79 0.6× 25 0.2× 13 0.2× 118 1.6× 11 0.2× 44 353
Yufei Sun China 13 72 0.5× 79 0.8× 61 0.8× 20 0.3× 21 0.3× 35 468

Countries citing papers authored by Cong Ding

Since Specialization
Citations

This map shows the geographic impact of Cong Ding's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cong Ding with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cong Ding more than expected).

Fields of papers citing papers by Cong Ding

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cong Ding. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cong Ding. The network helps show where Cong Ding may publish in the future.

Co-authorship network of co-authors of Cong Ding

This figure shows the co-authorship network connecting the top 25 collaborators of Cong Ding. A scholar is included among the top collaborators of Cong Ding based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cong Ding. Cong Ding is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ding, Cong, et al.. (2025). The E3 ubiquitin ligase MARCH9 alleviates pyroptosis by regulating NLPR3 ubiquitination following myocardial ischemia reperfusion. Cellular and Molecular Life Sciences. 82(1). 348–348.
2.
Liu, Yuchun, Kang Cheng, Cong Ding, et al.. (2024). UBD participates in neutrophilic asthma by promoting the activation of IL-17 signaling. International Journal of Biological Macromolecules. 264(Pt 1). 130581–130581. 1 indexed citations
3.
Ding, Cong, et al.. (2024). Structural insights into non-hotspot KRAS mutations and their potential as targets for effective cancer therapies. Journal of Biomolecular Structure and Dynamics. 43(15). 8308–8318. 1 indexed citations
4.
5.
Ding, Cong, Hongjun Xiao, Shunxin Wang, et al.. (2024). The circadian gene ARNTL2 promotes nasopharyngeal carcinoma invasiveness and metastasis through suppressing AMOTL2-LATS-YAP pathway. Cell Death and Disease. 15(7). 466–466. 6 indexed citations
6.
Sun, Wenjing, et al.. (2024). Deubiquitinase USP5 regulates RIPK1 driven pyroptosis in response to myocardial ischemic reperfusion injury. Cell Communication and Signaling. 22(1). 466–466. 1 indexed citations
7.
Liu, Jiaojiao, Cong Ding, Xin Liu, & Qiaozhen Kang. (2024). Cytoskeletal Protein 4.1R in Health and Diseases. Biomolecules. 14(2). 214–214. 1 indexed citations
8.
Wu, Ji, et al.. (2024). Study on spontaneous combustion characteristics of coal under thermo mechanical coupling. Scientific Reports. 14(1). 31914–31914. 2 indexed citations
9.
Wang, Zhiqiang, Jinying Peng, Yiying Gu, et al.. (2024). Sirtuin 7 Promotes Alcohol-Associated Liver Injury via Modulating Myeloid Cell Chemokine (C-C Motif) Ligand 2 Secretion through the NF-κB Signaling Pathway. American Journal Of Pathology. 195(3). 575–588.
10.
Ding, Cong, Bohao Liu, Zhiqiang Wang, et al.. (2024). SIRT7 protects against liver fibrosis by suppressing stellate cell activation via TGF-β/SMAD2/3 pathway. Biomedicine & Pharmacotherapy. 180. 117477–117477. 4 indexed citations
11.
Gu, Yiying, Zhiqiang Wang, Jinying Peng, et al.. (2024). SIRT7 stabilizes β-catenin and promotes canonical Wnt activation via upregulating FZD7. Life Sciences. 359. 123240–123240. 4 indexed citations
12.
Liu, Qiang, Liyun Zheng, Yue Wang, et al.. (2023). Primary choledocholithiasis occurrence and recurrence is synergetcally modulated by the bile microbiome and metabolome alternations. Life Sciences. 331. 122073–122073. 5 indexed citations
13.
14.
Ding, Cong, Xinglei Qi, Kaixing Qu, et al.. (2022). Distribution of a missense mutation (rs525805167) within the SLC45A2 gene associated with climatic conditions in Chinese cattle. Gene. 835. 146643–146643. 3 indexed citations
15.
Liu, Jiaojiao, Cong Ding, Xiaolong Wang, et al.. (2022). TLR agonist rMBP-NAP inhibits B16 melanoma tumor growth via induction of DCs maturation and T-cells cytotoxic response. Cytotechnology. 74(4). 459–467. 2 indexed citations
16.
Gu, Ye, Cong Ding, Yifeng Zhou, et al.. (2021). Flavonoid GL-V9 suppresses invasion and migration of human colorectal cancer cells by inhibiting PI3K/Akt and MMP-2/9 signaling. Journal of Cancer. 12(15). 4542–4551. 17 indexed citations
17.
Wang, Chengbo, Cong Ding, Juan Du, et al.. (2019). Protein 4.1B Suppresses Tumor Metastasis by Regulating Epithelial-mesenchymal Transition Progression in Melanoma Cells. International Journal of Medical Sciences. 16(4). 529–536. 5 indexed citations
18.
Chen, Qinchang, Kai Huang, Lingling Li, et al.. (2018). Serum uric acid on admission cannot predict long-term outcome of critically ill patients: a retrospective cohort study. Therapeutics and Clinical Risk Management. Volume 14. 1347–1359. 4 indexed citations
19.
Liu, Xin, Fu Guo, Zhenyu Ji, et al.. (2016). A Recombinant DNA Plasmid Encoding the sIL-4R-NAP Fusion Protein Suppress Airway Inflammation in an OVA-Induced Mouse Model of Asthma. Inflammation. 39(4). 1434–1440. 7 indexed citations
20.
Wang, Ting, Xilong Liu, Zhenyu Ji, et al.. (2015). Antitumor and immunomodulatory effects of recombinant fusion protein rMBP-NAP through TLR-2 dependent mechanism in tumor bearing mice. International Immunopharmacology. 29(2). 876–883. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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