Clement Y. Chow

2.3k total citations
33 papers, 1.6k citations indexed

About

Clement Y. Chow is a scholar working on Cell Biology, Molecular Biology and Genetics. According to data from OpenAlex, Clement Y. Chow has authored 33 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Cell Biology, 17 papers in Molecular Biology and 13 papers in Genetics. Recurrent topics in Clement Y. Chow's work include Endoplasmic Reticulum Stress and Disease (13 papers), Cellular transport and secretion (7 papers) and Lysosomal Storage Disorders Research (5 papers). Clement Y. Chow is often cited by papers focused on Endoplasmic Reticulum Stress and Disease (13 papers), Cellular transport and secretion (7 papers) and Lysosomal Storage Disorders Research (5 papers). Clement Y. Chow collaborates with scholars based in United States, France and Poland. Clement Y. Chow's co-authors include Mariana F. Wolfner, Miriam H. Meisler, Andrew G. Clark, Lois S. Weisman, Yanling Zhang, Natsuko Jin, Xuebao Zhang, Jun Li, Michael E. Shy and James J. Dowling and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The EMBO Journal.

In The Last Decade

Clement Y. Chow

31 papers receiving 1.6k citations

Peers

Clement Y. Chow

CMN

GENET

PHYSI

Chao Tong China

Lei Xue China

Tamás Lukácsovich United States

Zhongyuan Zuo United States

Galit Shohat-Ophir Israel

Paweł Lis United Kingdom

Yohei Ohashi Japan

Diego Peretti United Kingdom

Marie-Christine Fournier France

Karolina Pircs Sweden

Chao Tong China

Clement Y. Chow

661 ×0.9

2.4k ×3.9

371 ×1.1

CMN

592 ×1.8

GENET

88 ×0.3

PHYSI

Citations per year, relative to Clement Y. Chow Clement Y. Chow (= 1×) peers Chao Tong

Countries citing papers authored by Clement Y. Chow

Since Specialization

Citations

This map shows the geographic impact of Clement Y. Chow's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Clement Y. Chow with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Clement Y. Chow more than expected).

Fields of papers citing papers by Clement Y. Chow

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Clement Y. Chow. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Clement Y. Chow. The network helps show where Clement Y. Chow may publish in the future.

Co-authorship network of co-authors of Clement Y. Chow

This figure shows the co-authorship network connecting the top 25 collaborators of Clement Y. Chow. A scholar is included among the top collaborators of Clement Y. Chow based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Clement Y. Chow. Clement Y. Chow is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pedersen, Brent S., Nichole Link, Aaron R. Quinlan, et al.. (2025). Identification of CNTN2 as a genetic modifier of PIGA-CDG in a family with incomplete penetrance and in Drosophila. The American Journal of Human Genetics. 112(3). 572–582.

Chow, Clement Y., et al.. (2024). A drug repurposing screen reveals dopamine signaling as a critical pathway underlying potential therapeutics for the rare disease DPAGT1-CDG. PLoS Genetics. 20(10). e1011458–e1011458. 1 indexed citations

Partha, Raghavendran, et al.. (2024). Evolutionary rate covariation is pervasive between glycosylation pathways and points to potential disease modifiers. PLoS Genetics. 20(9). e1011406–e1011406. 1 indexed citations

Jorde, Lynn B., et al.. (2023). Characterizing genetic variation in the regulation of the ER stress response through computational and cis-eQTL analyses. G3 Genes Genomes Genetics. 13(12). 1 indexed citations

Chow, Clement Y., et al.. (2023). Drosophila models of phosphatidylinositol glycan biosynthesis class A congenital disorder of glycosylation (PIGA-CDG) mirror patient phenotypes. G3 Genes Genomes Genetics. 14(3). 3 indexed citations

Chow, Clement Y., et al.. (2023). A Drosophila screen identifies a role for histone methylation in ER stress preconditioning. G3 Genes Genomes Genetics. 14(2).

Chow, Clement Y., et al.. (2022). The dynamic effect of genetic variation on the in vivo ER stress transcriptional response in different tissues. G3 Genes Genomes Genetics. 12(6). 5 indexed citations

Viswanatha, Raghuvir, et al.. (2022). A genome-wide CRISPR screen identifies DPM1 as a modifier of DPAGT1 deficiency and ER stress. PLoS Genetics. 18(9). e1010430–e1010430. 7 indexed citations

Gorsi, Bushra, Edgar J. Hernández, Clement Y. Chow, et al.. (2021). Causal and Candidate Gene Variants in a Large Cohort of Women With Primary Ovarian Insufficiency. The Journal of Clinical Endocrinology & Metabolism. 107(3). 685–714. 17 indexed citations

10.

Chow, Clement Y., et al.. (2020). Decoupling of Apoptosis from Activation of the ER Stress Response by the Drosophila Metallopeptidase superdeath. Genetics. 214(4). 913–925. 7 indexed citations

11.

Mercenne, Gaëlle, Raghavendran Partha, Kevin A. Hope, et al.. (2020). A Drosophila screen identifies NKCC1 as a modifier of NGLY1 deficiency. eLife. 9. 30 indexed citations

12.

Goodman, Alan G., et al.. (2019). Natural Genetic Variation Screen in Drosophila Identifies Wnt Signaling, Mitochondrial Metabolism, and Redox Homeostasis Genes as Modifiers of Apoptosis. G3 Genes Genomes Genetics. 9(12). 3995–4005. 14 indexed citations

13.

Chow, Clement Y., et al.. (2018). Genetic Modifiers of Neurodegeneration in a Drosophila Model of Parkinson’s Disease. Genetics. 209(4). 1345–1356. 29 indexed citations

14.

Chow, Clement Y. & Lawrence T. Reiter. (2017). Etiology of Human Genetic Disease on the Fly. Trends in Genetics. 33(6). 391–398. 34 indexed citations

15.

Chow, Clement Y., Frank W. Avila, Andrew G. Clark, & Mariana F. Wolfner. (2015). Induction of Excessive Endoplasmic Reticulum Stress in the Drosophila Male Accessory Gland Results in Infertility. PLoS ONE. 10(3). e0119386–e0119386. 33 indexed citations

16.

Chow, Clement Y., et al.. (2015). The Genetic Architecture of the Genome-Wide Transcriptional Response to ER Stress in the Mouse. PLoS Genetics. 11(2). e1004924–e1004924. 29 indexed citations

17.

Chow, Clement Y., et al.. (2015). Candidate genetic modifiers of retinitis pigmentosa identified by exploiting natural variation inDrosophila. Human Molecular Genetics. 25(4). 651–659. 62 indexed citations

18.

Sirot, Laura K., Brooke LaFlamme, Jessica L. Sitnik, et al.. (2009). Molecular Social Interactions. Advances in genetics. 68. 23–56. 56 indexed citations

19.

Zhang, Xuebao, Clement Y. Chow, Zarife Sahenk, et al.. (2008). Mutation of FIG4 causes a rapidly progressive, asymmetric neuronal degeneration. Brain. 131(8). 1990–2001. 113 indexed citations

20.

Zhang, Yanling, Sergey N. Zolov, Clement Y. Chow, et al.. (2007). Loss of Vac14, a regulator of the signaling lipid phosphatidylinositol 3,5-bisphosphate, results in neurodegeneration in mice. Proceedings of the National Academy of Sciences. 104(44). 17518–17523. 176 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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