Clarissa A. Henry

1.6k total citations
33 papers, 1.2k citations indexed

About

Clarissa A. Henry is a scholar working on Molecular Biology, Cell Biology and Immunology and Allergy. According to data from OpenAlex, Clarissa A. Henry has authored 33 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 18 papers in Cell Biology and 6 papers in Immunology and Allergy. Recurrent topics in Clarissa A. Henry's work include Muscle Physiology and Disorders (14 papers), Congenital heart defects research (9 papers) and Cellular Mechanics and Interactions (9 papers). Clarissa A. Henry is often cited by papers focused on Muscle Physiology and Disorders (14 papers), Congenital heart defects research (9 papers) and Cellular Mechanics and Interactions (9 papers). Clarissa A. Henry collaborates with scholars based in United States, Canada and Australia. Clarissa A. Henry's co-authors include Michelle F. Goody, Sharon L. Amacher, Bryan D. Crawford, Merrill B. Hille, André Khalil, Chelsi J. Snow, Mark S. Cooper, Leonard D’Amico, Meghan Kelly and Mark S. Cooper and has published in prestigious journals such as Development, Current Biology and Developmental Cell.

In The Last Decade

Clarissa A. Henry

31 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Clarissa A. Henry United States 21 932 497 144 96 74 33 1.2k
Jared C. Talbot United States 16 703 0.8× 263 0.5× 29 0.2× 171 1.8× 104 1.4× 21 1.0k
Kunimasa Ohta Japan 25 992 1.1× 433 0.9× 38 0.3× 142 1.5× 86 1.2× 74 1.7k
Joachim Berger Australia 22 1.2k 1.3× 314 0.6× 50 0.3× 288 3.0× 84 1.1× 44 1.7k
Kevin M. Wright United States 18 929 1.0× 292 0.6× 33 0.2× 87 0.9× 98 1.3× 43 1.3k
Michael J. Jurynec United States 16 1.1k 1.1× 464 0.9× 30 0.2× 185 1.9× 88 1.2× 31 1.7k
Maura McGrail United States 17 1.3k 1.3× 1.1k 2.2× 85 0.6× 135 1.4× 47 0.6× 29 1.6k
Rika Nakayama Japan 16 804 0.9× 172 0.3× 51 0.4× 172 1.8× 55 0.7× 22 1.1k
Ingrid Zwaenepoel France 11 789 0.8× 285 0.6× 92 0.6× 116 1.2× 80 1.1× 12 1.3k
Rolf W. Stottmann United States 24 1.4k 1.5× 235 0.5× 25 0.2× 783 8.2× 70 0.9× 63 1.8k
Juan A. Montero Spain 24 1.2k 1.3× 366 0.7× 67 0.5× 350 3.6× 167 2.3× 69 2.1k

Countries citing papers authored by Clarissa A. Henry

Since Specialization
Citations

This map shows the geographic impact of Clarissa A. Henry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Clarissa A. Henry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Clarissa A. Henry more than expected).

Fields of papers citing papers by Clarissa A. Henry

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Clarissa A. Henry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Clarissa A. Henry. The network helps show where Clarissa A. Henry may publish in the future.

Co-authorship network of co-authors of Clarissa A. Henry

This figure shows the co-authorship network connecting the top 25 collaborators of Clarissa A. Henry. A scholar is included among the top collaborators of Clarissa A. Henry based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Clarissa A. Henry. Clarissa A. Henry is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Farr, Gist H., et al.. (2025). Epigenetic small molecule screening identifies a new HDACi compound for ameliorating Duchenne muscular dystrophy. Molecular Therapy — Nucleic Acids. 36(3). 102683–102683.
2.
Henry, Clarissa A., M. Chiara Manzini, John M. Parant, et al.. (2024). Standardization of zebrafish drug testing parameters for muscle diseases. Disease Models & Mechanisms. 17(1). 13 indexed citations
3.
Henry, Clarissa A., et al.. (2021). Lysosomal Function Impacts the Skeletal Muscle Extracellular Matrix. Journal of Developmental Biology. 9(4). 52–52. 7 indexed citations
5.
Khalil, André, et al.. (2019). NAD+ improves neuromuscular development in a zebrafish model of FKRP-associated dystroglycanopathy. Skeletal Muscle. 9(1). 21–21. 21 indexed citations
6.
Goody, Michelle F. & Clarissa A. Henry. (2018). A need for NAD+ in muscle development, homeostasis, and aging. Skeletal Muscle. 8(1). 9–9. 50 indexed citations
7.
Goody, Michelle F., et al.. (2018). Ethanol Exposure Causes Muscle Degeneration in Zebrafish. Journal of Developmental Biology. 6(1). 7–7. 11 indexed citations
8.
Goody, Michelle F., Denise Jurczyszak, Carol H. Kim, & Clarissa A. Henry. (2017). Influenza A Virus Infection Damages Zebrafish Skeletal Muscle and Exacerbates Disease in Zebrafish Modeling Duchenne Muscular Dystrophy. PLoS Currents. 9. 17 indexed citations
9.
Goody, Michelle F., et al.. (2016). “Muscling” Throughout Life. Current topics in developmental biology. 124. 197–234. 24 indexed citations
10.
Jenkins, Molly H., et al.. (2016). Laminin and Matrix metalloproteinase 11 regulate Fibronectin levels in the zebrafish myotendinous junction. Skeletal Muscle. 6(1). 18–18. 32 indexed citations
11.
Goody, Michelle F. & Clarissa A. Henry. (2010). Dynamic interactions between cells and their extracellular matrix mediate embryonic development. Molecular Reproduction and Development. 77(6). 475–488. 23 indexed citations
12.
Goody, Michelle F., et al.. (2010). Nrk2b-mediated NAD+ production regulates cell adhesion and is required for muscle morphogenesis in vivo. Developmental Biology. 344(2). 809–826. 53 indexed citations
13.
Snow, Chelsi J., Michelle F. Goody, Meghan Kelly, et al.. (2008). Time-Lapse Analysis and Mathematical Characterization Elucidate Novel Mechanisms Underlying Muscle Morphogenesis. PLoS Genetics. 4(10). e1000219–e1000219. 43 indexed citations
14.
Henry, Clarissa A., et al.. (2005). Regionally Autonomous Segmentation Within Zebrafish Presomitic Mesoderm. Zebrafish. 2(1). 7–18. 6 indexed citations
15.
Henry, Clarissa A., et al.. (2005). Interactions between muscle fibers and segment boundaries in zebrafish. Developmental Biology. 287(2). 346–360. 67 indexed citations
16.
Henry, Clarissa A. & Sharon L. Amacher. (2004). Zebrafish Slow Muscle Cell Migration Induces a Wave of Fast Muscle Morphogenesis. Developmental Cell. 7(6). 917–923. 88 indexed citations
17.
Cooper, Mark S., Leonard D’Amico, & Clarissa A. Henry. (2003). Analyzing Morphogenetic Cell Behaviors in Vitally Stained Zebrafish Embryos. Humana Press eBooks. 122. 185–204. 18 indexed citations
18.
Henry, Clarissa A., Michael K. Urban, Kariena K. Dill, et al.. (2002). Two linkedhairy/Enhancer of split-related zebrafish genes,her1andher7, function together to refine alternating somite boundaries. Development. 129(15). 3693–3704. 147 indexed citations
19.
Henry, Clarissa A., et al.. (2000). Somites in zebrafish doubly mutant for knypek and trilobite form without internal mesenchymal cells or compaction. Current Biology. 10(17). 1063–1066. 68 indexed citations
20.
Cooper, Mark S., Leonard D’Amico, & Clarissa A. Henry. (1998). Chapter 11 Confocal Microscopic Analysis of Morphogenetic Movements. Methods in cell biology. 59. 179–204. 68 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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