Clare V. Logan

6.3k total citations
21 papers, 1.0k citations indexed

About

Clare V. Logan is a scholar working on Molecular Biology, Genetics and Rheumatology. According to data from OpenAlex, Clare V. Logan has authored 21 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 12 papers in Genetics and 4 papers in Rheumatology. Recurrent topics in Clare V. Logan's work include Genetic and Kidney Cyst Diseases (6 papers), dental development and anomalies (5 papers) and Genetic Syndromes and Imprinting (4 papers). Clare V. Logan is often cited by papers focused on Genetic and Kidney Cyst Diseases (6 papers), dental development and anomalies (5 papers) and Genetic Syndromes and Imprinting (4 papers). Clare V. Logan collaborates with scholars based in United Kingdom, United States and Germany. Clare V. Logan's co-authors include Colin A. Johnson, Matthew Adams, David Parry, Katarzyna Szymańska, Helen R. Dawe, Gabrielle Wheway, Zakia Abdelhamed, Keith Gull, Eamonn Sheridan and Chris F. Inglehearn and has published in prestigious journals such as SHILAP Revista de lepidopterología, Molecular Cell and PLoS ONE.

In The Last Decade

Clare V. Logan

21 papers receiving 1.0k citations

Peers

Clare V. Logan
Clare V. Logan
Citations per year, relative to Clare V. Logan Clare V. Logan (= 1×) peers Marie Legendre

Countries citing papers authored by Clare V. Logan

Since Specialization
Citations

This map shows the geographic impact of Clare V. Logan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Clare V. Logan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Clare V. Logan more than expected).

Fields of papers citing papers by Clare V. Logan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Clare V. Logan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Clare V. Logan. The network helps show where Clare V. Logan may publish in the future.

Co-authorship network of co-authors of Clare V. Logan

This figure shows the co-authorship network connecting the top 25 collaborators of Clare V. Logan. A scholar is included among the top collaborators of Clare V. Logan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Clare V. Logan. Clare V. Logan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Fernández‐Fuentes, Narcís, Martin McKibbin, Nursel Elçioğlu, et al.. (2022). Novel SIX6 mutations cause recessively inherited congenital cataract, microcornea, and corneal opacification with or without coloboma and microphthalmia.. PubMed. 28. 57–69. 2 indexed citations
2.
Tarnauskaitė, Žygimantė, Louise S. Bicknell, Joseph A. Marsh, et al.. (2019). Biallelic variants in DNA2 cause microcephalic primordial dwarfism. Human Mutation. 40(8). 1063–1070. 17 indexed citations
3.
Bellelli, Roberto, Valérie Borel, Clare V. Logan, et al.. (2018). Polε Instability Drives Replication Stress, Abnormal Development, and Tumorigenesis. Molecular Cell. 70(4). 707–721.e7. 59 indexed citations
4.
Parry, David, Claire E. L. Smith, Walid El‐Sayed, et al.. (2016). Mutations in the pH-Sensing G-protein-Coupled Receptor GPR68 Cause Amelogenesis Imperfecta. The American Journal of Human Genetics. 99(4). 984–990. 49 indexed citations
5.
Logan, Clare V., Mike Beadsworth, & Nicholas J. Beeching. (2016). HIV and diarrhoea: what is new?. Current Opinion in Infectious Diseases. 29(5). 486–494. 29 indexed citations
6.
Logan, Clare V., Judith Cossins, Pedro M. Rodríguez Cruz, et al.. (2015). Congenital Myasthenic Syndrome Type 19 Is Caused by Mutations in COL13A1, Encoding the Atypical Non-fibrillar Collagen Type XIII α1 Chain. The American Journal of Human Genetics. 97(6). 878–885. 55 indexed citations
7.
Acevedo, Ana Carolina, James A. Poulter, Caroline Lourenço de Lima, et al.. (2015). Variability of systemic and oro-dental phenotype in two families with non-lethal Raine syndrome with FAM20C mutations. BMC Medical Genetics. 16(1). 8–8. 60 indexed citations
8.
Szymańska, Katarzyna, Gabrielle Wheway, Dan Doherty, et al.. (2015). A high-throughput genome-wide siRNA screen for ciliogenesis identifies new ciliary functional components and ciliopathy genes. ePrints Soton (University of Southampton). 4(S1). 4 indexed citations
9.
Parry, David, Clare V. Logan, Tim M. Strom, et al.. (2015). HACE1 deficiency causes an autosomal recessive neurodevelopmental syndrome. Journal of Medical Genetics. 52(12). 797–803. 34 indexed citations
10.
Watson, Christopher M., Mohammed E. El‐Asrag, David Parry, et al.. (2014). Mutation Screening of Retinal Dystrophy Patients by Targeted Capture from Tagged Pooled DNAs and Next Generation Sequencing. PLoS ONE. 9(8). e104281–e104281. 17 indexed citations
11.
Parry, David, Clare V. Logan, Alexander P.A. Stegmann, et al.. (2013). SAMS, a Syndrome of Short Stature, Auditory-Canal Atresia, Mandibular Hypoplasia, and Skeletal Abnormalities Is a Unique Neurocristopathy Caused by Mutations in Goosecoid. The American Journal of Human Genetics. 93(6). 1135–1142. 27 indexed citations
12.
Parry, David, James A. Poulter, Clare V. Logan, et al.. (2013). Identification of Mutations in SLC24A4, Encoding a Potassium-Dependent Sodium/Calcium Exchanger, as a Cause of Amelogenesis Imperfecta. The American Journal of Human Genetics. 92(2). 307–312. 95 indexed citations
13.
Carr, Ian, Joanne Morgan, Christopher M. Watson, et al.. (2013). Simple and Efficient Identification of Rare Recessive Pathologically Important Sequence Variants from Next Generation Exome Sequence Data. Human Mutation. 34(7). 945–952. 5 indexed citations
14.
Szymańska, Katarzyna, Ian Berry, Clare V. Logan, et al.. (2012). Founder mutations and genotype-phenotype correlations in Meckel-Gruber syndrome and associated ciliopathies. SHILAP Revista de lepidopterología. 1(1). 18–18. 37 indexed citations
15.
Parry, David, Clare V. Logan, Bruce E. Hayward, et al.. (2011). Mutations Causing Familial Biparental Hydatidiform Mole Implicate C6orf221 as a Possible Regulator of Genomic Imprinting in the Human Oocyte. The American Journal of Human Genetics. 89(3). 451–458. 174 indexed citations
16.
Carr, Ian, Joanne Morgan, Christine P. Diggle, et al.. (2011). Illuminator, a desktop program for mutation detection using short-read clonal sequencing. Genomics. 98(4). 302–309. 3 indexed citations
17.
Adams, Matthew, Roslyn Simms, Zakia Abdelhamed, et al.. (2011). A meckelin–filamin A interaction mediates ciliogenesis. Human Molecular Genetics. 21(6). 1272–1286. 85 indexed citations
18.
Logan, Clare V., Zakia Abdelhamed, & Colin A. Johnson. (2010). Molecular Genetics and Pathogenic Mechanisms for the Severe Ciliopathies: Insights into Neurodevelopment and Pathogenesis of Neural Tube Defects. Molecular Neurobiology. 43(1). 12–26. 40 indexed citations
19.
Dawe, Helen R., Matthew Adams, Gabrielle Wheway, et al.. (2009). Nesprin-2 interacts with meckelin and mediates ciliogenesis via remodelling of the actin cytoskeleton. Journal of Cell Science. 122(15). 2716–2726. 110 indexed citations
20.
Adams, Matthew, et al.. (2008). Recent advances in the molecular pathology, cell biology and genetics of ciliopathies. Journal of Medical Genetics. 45(5). 257–267. 109 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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