Clare M. Gladding

2.1k total citations
14 papers, 1.7k citations indexed

About

Clare M. Gladding is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Clare M. Gladding has authored 14 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Cellular and Molecular Neuroscience, 10 papers in Molecular Biology and 2 papers in Cognitive Neuroscience. Recurrent topics in Clare M. Gladding's work include Neuroscience and Neuropharmacology Research (13 papers), Genetic Neurodegenerative Diseases (9 papers) and Mitochondrial Function and Pathology (6 papers). Clare M. Gladding is often cited by papers focused on Neuroscience and Neuropharmacology Research (13 papers), Genetic Neurodegenerative Diseases (9 papers) and Mitochondrial Function and Pathology (6 papers). Clare M. Gladding collaborates with scholars based in Canada, United Kingdom and United States. Clare M. Gladding's co-authors include Lynn A. Raymond, Elek Molnár, Austen J. Milnerwood, Stephen M. Fitzjohn, Graham L. Collingridge, Carlos Cepeda, Marc S. Levine, Véronique M. André, Rochelle M. Hines and Timothy H. Murphy and has published in prestigious journals such as Journal of Biological Chemistry, Neuron and Journal of Neuroscience.

In The Last Decade

Clare M. Gladding

14 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Clare M. Gladding Canada	14	1.4k	953	372	274	182	14	1.7k
Lin Pei China	17	1.2k 0.9×	1.2k 1.3×	130 0.3×	192 0.7×	172 0.9×	39	2.0k
Sarah J. Augood United States	24	1.9k 1.4×	1.1k 1.1×	1.1k 2.9×	312 1.1×	265 1.5×	34	2.6k
Joerg Neddens Germany	19	742 0.5×	603 0.6×	156 0.4×	273 1.0×	327 1.8×	46	1.4k
Julika Pitsch Germany	20	841 0.6×	620 0.7×	121 0.3×	219 0.8×	258 1.4×	43	1.4k
Joanne E. Nash Canada	22	1.2k 0.9×	790 0.8×	513 1.4×	204 0.7×	157 0.9×	33	1.9k
Thomas Behnisch China	26	1.3k 0.9×	926 1.0×	99 0.3×	496 1.8×	239 1.3×	69	1.8k
Véronique M. André United States	21	1.8k 1.3×	962 1.0×	804 2.2×	241 0.9×	111 0.6×	26	2.0k
Michael A. Benneyworth United States	18	1.2k 0.9×	850 0.9×	138 0.4×	233 0.9×	206 1.1×	27	1.8k
Jahan Dadgar United States	14	993 0.7×	867 0.9×	282 0.8×	228 0.8×	420 2.3×	15	1.8k
Steven J. Tavalin United States	19	1.3k 1.0×	1.5k 1.6×	105 0.3×	265 1.0×	141 0.8×	25	2.0k

Countries citing papers authored by Clare M. Gladding

Since Specialization

Citations

This map shows the geographic impact of Clare M. Gladding's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Clare M. Gladding with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Clare M. Gladding more than expected).

Fields of papers citing papers by Clare M. Gladding

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Clare M. Gladding. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Clare M. Gladding. The network helps show where Clare M. Gladding may publish in the future.

Co-authorship network of co-authors of Clare M. Gladding

This figure shows the co-authorship network connecting the top 25 collaborators of Clare M. Gladding. A scholar is included among the top collaborators of Clare M. Gladding based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Clare M. Gladding. Clare M. Gladding is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

1.

Gladding, Clare M., Jing Fan, Lily Y. J. Zhang, et al.. (2014). Alterations in STriatal‐Enriched protein tyrosine Phosphatase expression, activation, and downstream signaling in early and late stages of the YAC128 Huntington's disease mouse model. Journal of Neurochemistry. 130(1). 145–159. 28 indexed citations

2.

Gladding, Clare M., et al.. (2013). Chronic blockade of extrasynaptic NMDA receptors ameliorates synaptic dysfunction and pro-death signaling in Huntington disease transgenic mice. Neurobiology of Disease. 62. 533–542. 67 indexed citations

3.

Gladding, Clare M., Marja D. Sepers, Jian Xu, et al.. (2012). Calpain and STriatal-Enriched protein tyrosine Phosphatase (STEP) activation contribute to extrasynaptic NMDA receptor localization in a Huntington's disease mouse model. Human Molecular Genetics. 21(17). 3739–3752. 64 indexed citations

4.

Milnerwood, Austen J., Marja D. Sepers, Clare M. Gladding, et al.. (2012). Mitigation of augmented extrasynaptic NMDAR signaling and apoptosis in cortico-striatal co-cultures from Huntington's disease mice. Neurobiology of Disease. 48(1). 40–51. 55 indexed citations

5.

Raymond, Lynn A., Véronique M. André, Carlos Cepeda, et al.. (2011). Pathophysiology of Huntington's disease: time-dependent alterations in synaptic and receptor function. Neuroscience. 198. 252–273. 238 indexed citations

6.

Gladding, Clare M. & Lynn A. Raymond. (2011). Mechanisms underlying NMDA receptor synaptic/extrasynaptic distribution and function. Molecular and Cellular Neuroscience. 48(4). 308–320. 160 indexed citations

7.

Fan, Jing, Clare M. Gladding, Liang Wang, et al.. (2011). P38 MAPK is involved in enhanced NMDA receptor-dependent excitotoxicity in YAC transgenic mouse model of Huntington disease. Neurobiology of Disease. 45(3). 999–1009. 76 indexed citations

8.

Milnerwood, Austen J., Clare M. Gladding, Mahmoud A. Pouladi, et al.. (2010). Early Increase in Extrasynaptic NMDA Receptor Signaling and Expression Contributes to Phenotype Onset in Huntington's Disease Mice. Neuron. 65(3). 436–436. 16 indexed citations

9.

Milnerwood, Austen J., Clare M. Gladding, Mahmoud A. Pouladi, et al.. (2010). Early Increase in Extrasynaptic NMDA Receptor Signaling and Expression Contributes to Phenotype Onset in Huntington's Disease Mice. Neuron. 65(2). 178–190. 404 indexed citations

10.

Gladding, Clare M., Stephen M. Fitzjohn, & Elek Molnár. (2009). Metabotropic Glutamate Receptor-Mediated Long-Term Depression: Molecular Mechanisms. Pharmacological Reviews. 61(4). 395–412. 175 indexed citations

11.

Zhang, Yang, Deepa V. Venkitaramani, Clare M. Gladding, et al.. (2008). The Tyrosine Phosphatase STEP Mediates AMPA Receptor Endocytosis after Metabotropic Glutamate Receptor Stimulation. Journal of Neuroscience. 28(42). 10561–10566. 154 indexed citations

12.

Gladding, Clare M., Valerie J. Collett, Zhengping Jia, et al.. (2008). Tyrosine dephosphorylation regulates AMPAR internalisation in mGluR-LTD. Molecular and Cellular Neuroscience. 40(2). 267–279. 55 indexed citations

13.

Matsuda, Keiko, Shinji Matsuda, Clare M. Gladding, & Michisuke Yuzaki. (2006). Characterization of the δ2 Glutamate Receptor-binding Protein Delphilin. Journal of Biological Chemistry. 281(35). 25577–25587. 33 indexed citations

14.

Moult, Peter R., Clare M. Gladding, Thomas M. Sanderson, et al.. (2006). Tyrosine Phosphatases Regulate AMPA Receptor Trafficking during Metabotropic Glutamate Receptor-Mediated Long-Term Depression. Journal of Neuroscience. 26(9). 2544–2554. 145 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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