Chunlei Cang

2.3k total citations
31 papers, 1.6k citations indexed

About

Chunlei Cang is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Chunlei Cang has authored 31 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Physiology, 14 papers in Molecular Biology and 10 papers in Cellular and Molecular Neuroscience. Recurrent topics in Chunlei Cang's work include Calcium signaling and nucleotide metabolism (17 papers), Cellular transport and secretion (6 papers) and Ion Channels and Receptors (6 papers). Chunlei Cang is often cited by papers focused on Calcium signaling and nucleotide metabolism (17 papers), Cellular transport and secretion (6 papers) and Ion Channels and Receptors (6 papers). Chunlei Cang collaborates with scholars based in China, United States and Poland. Chunlei Cang's co-authors include Dejian Ren, Kimberly Aranda, Zhi‐Qi Zhao, Hua Zhang, Yu‐Qiu Zhang, Young‐Jun Seo, Bruno Gasnier, Itzhak Nissim, Shyue-Fang Battaglia-Hsu and Yandong Zhou and has published in prestigious journals such as Nature, Cell and Nature Communications.

In The Last Decade

Chunlei Cang

28 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Chunlei Cang China 18 717 621 376 327 308 31 1.6k
Katja Rietdorf United Kingdom 18 1.2k 1.6× 1.0k 1.7× 622 1.7× 323 1.0× 212 0.7× 32 2.5k
Eamonn J. Dickson United States 26 224 0.3× 1.3k 2.2× 230 0.6× 534 1.6× 427 1.4× 54 2.4k
Marie‐Jo Moutin France 23 319 0.4× 983 1.6× 171 0.5× 226 0.7× 79 0.3× 47 1.6k
Bruno Gasnier France 25 343 0.5× 1.1k 1.7× 62 0.2× 598 1.8× 406 1.3× 37 2.2k
Shin Jung Kang South Korea 19 109 0.2× 632 1.0× 212 0.6× 247 0.8× 182 0.6× 57 1.4k
David R. Giovannucci United States 26 205 0.3× 1.4k 2.3× 205 0.5× 574 1.8× 219 0.7× 52 2.1k
Rebecca Sitsapesan United Kingdom 27 748 1.0× 2.1k 3.4× 593 1.6× 799 2.4× 120 0.4× 75 2.8k
Lee P. Haynes United Kingdom 22 186 0.3× 953 1.5× 286 0.8× 493 1.5× 144 0.5× 37 1.5k
Nagomi Kurebayashi Japan 26 136 0.2× 1.7k 2.8× 296 0.8× 668 2.0× 300 1.0× 99 2.3k
Ran Zalk Israel 17 141 0.2× 1.6k 2.5× 120 0.3× 451 1.4× 355 1.2× 42 2.2k

Countries citing papers authored by Chunlei Cang

Since Specialization
Citations

This map shows the geographic impact of Chunlei Cang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chunlei Cang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chunlei Cang more than expected).

Fields of papers citing papers by Chunlei Cang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chunlei Cang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chunlei Cang. The network helps show where Chunlei Cang may publish in the future.

Co-authorship network of co-authors of Chunlei Cang

This figure shows the co-authorship network connecting the top 25 collaborators of Chunlei Cang. A scholar is included among the top collaborators of Chunlei Cang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chunlei Cang. Chunlei Cang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lu, Hua, Yao Liu, Jiabei Wang, et al.. (2025). Micropeptide hSPAR regulates glutamine levels and suppresses mammary tumor growth via a TRIM21-P27KIP1-mTOR axis. The EMBO Journal. 44(5). 1414–1441. 3 indexed citations
2.
Zeng, Wenping, Canjun Li, Ruikun Wu, et al.. (2024). Optogenetic manipulation of lysosomal physiology and autophagy-dependent clearance of amyloid beta. PLoS Biology. 22(4). e3002591–e3002591. 5 indexed citations
3.
Wu, Yu, Jiamin Huang, Florence Guivel‐Benhassine, et al.. (2024). Endolysosomal channel TMEM175 mediates antitoxin activity of DABMA. FEBS Journal. 291(18). 4142–4154.
4.
Xu, Na, Lei Qi, Feng Gao, et al.. (2023). Reduced lysosomal density in neuronal dendrites mediates deficits in synaptic plasticity in Huntington’s disease. Cell Reports. 42(12). 113573–113573. 5 indexed citations
5.
Zeng, Wenping, et al.. (2022). Lysosomal K + channel TMEM175 promotes apoptosis and aggravates symptoms of Parkinson's disease. EMBO Reports. 23(9). e53234–e53234. 31 indexed citations
6.
Li, Yawen, Xinru Li, Ping‐Xia Zhao, et al.. (2022). Structure of the Arabidopsis guard cell anion channel SLAC1 suggests activation mechanism by phosphorylation. Nature Communications. 13(1). 2511–2511. 31 indexed citations
7.
8.
Zeng, Wenping, Canjun Li, Wenqi Hu, et al.. (2021). CLN7 is an organellar chloride channel regulating lysosomal function. Science Advances. 7(51). eabj9608–eabj9608. 35 indexed citations
9.
Wie, Jinhong, Zhenjiang Liu, Thomas F. Tropea, et al.. (2021). Author Correction: A growth-factor-activated lysosomal K+ channel regulates Parkinson’s pathology. Nature. 592(7855). E10–E10. 3 indexed citations
10.
Wie, Jinhong, Zhenjiang Liu, Thomas F. Tropea, et al.. (2021). A growth-factor-activated lysosomal K+ channel regulates Parkinson’s pathology. Nature. 591(7850). 431–437. 86 indexed citations
11.
Zhu, Hongying, Qianqian Li, Xiang Yin, et al.. (2021). Metabolomic profiling of single enlarged lysosomes. Nature Methods. 18(7). 788–798. 71 indexed citations
12.
Zhang, Bo, Geng Qin, Yanhong Zhang, et al.. (2021). Wnt8a is one of the candidate genes that play essential roles in the elongation of the seahorse prehensile tail. Marine Life Science & Technology. 3(4). 416–426. 7 indexed citations
13.
14.
Chen, Cheng‐Chang, Chunlei Cang, Stefanie Fenske, et al.. (2017). Patch-clamp technique to characterize ion channels in enlarged individual endolysosomes. Nature Protocols. 12(8). 1639–1658. 72 indexed citations
15.
Stray‐Pedersen, Asbjørg, Jan-Maarten Cobben, Trine Prescott, et al.. (2015). Biallelic Mutations in UNC80 Cause Persistent Hypotonia, Encephalopathy, Growth Retardation, and Severe Intellectual Disability. The American Journal of Human Genetics. 98(1). 202–209. 35 indexed citations
16.
Cang, Chunlei, Kimberly Aranda, Young‐Jun Seo, Bruno Gasnier, & Dejian Ren. (2015). TMEM175 Is an Organelle K+ Channel Regulating Lysosomal Function. Cell. 162(5). 1101–1112. 173 indexed citations
17.
Guo, Jiangtao, Weizhong Zeng, Qingfeng Chen, et al.. (2015). Structure of the voltage-gated two-pore channel TPC1 from Arabidopsis thaliana. Nature. 531(7593). 196–201. 194 indexed citations
18.
Cang, Chunlei, et al.. (2014). The voltage-gated sodium channel TPC1 confers endolysosomal excitability. Nature Chemical Biology. 10(6). 463–469. 138 indexed citations
19.
Cang, Chunlei, Kimberly Aranda, & Dejian Ren. (2014). A non-inactivating high-voltage-activated two-pore Na+ channel that supports ultra-long action potentials and membrane bistability. Nature Communications. 5(1). 5015–5015. 37 indexed citations
20.
Cang, Chunlei, Yandong Zhou, Betsy Navarro, et al.. (2013). mTOR Regulates Lysosomal ATP-Sensitive Two-Pore Na+ Channels to Adapt to Metabolic State. Cell. 152(4). 778–790. 292 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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