Chui‐Se Tham

1.3k citations
16 papers · 1.1k · h-index 16

Impact in

Papers in

    • Neuroscience and Neuropharmacology Research 5
    • Neurotransmitter Receptor Influence on Behavior 4
    • Receptor Mechanisms and Signaling 4
    • Sphingolipid Metabolism and Signaling 3
    • Ion channel regulation and function 2

Chui‐Se Tham

16 papers receiving 1.1k citations

Peers

Chui‐Se Tham
Comparison fields: 5 of 67
  • Cellular and Molecular Neuroscience 562
  • Neurology 124
  • Biological Psychiatry 36
  • Developmental Neuroscience 57
  • Biochemistry 98
Replace Amy D. Sinor with:
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Citations per field
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Citations per year

Countries citing papers authored by Chui‐Se Tham

Since Specialization
Citations

This map shows the geographic impact of Chui‐Se Tham's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chui‐Se Tham with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chui‐Se Tham more than expected).

Fields of papers citing papers by Chui‐Se Tham

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chui‐Se Tham. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chui‐Se Tham. The network helps show where Chui‐Se Tham may publish in the future.

Co-authors

The 25 scholars most cited alongside Chui‐Se Tham, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Chui‐Se Tham Line = papers co-authored together Chui‐Se Tham links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 2004194
2 2001140
3 1991113
4 200399
5 199089
6 200876
7 199564
8 200861
9 201446
10 200745
11 200837
12 199330
13 200429
14 199428
15 199225
16 199524

About Chui‐Se Tham

Chui‐Se Tham is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Pharmacology, Pathology and Forensic Medicine and Endocrine and Autonomic Systems, having authored 16 papers that have together received 1.1k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (5 papers), Receptor Mechanisms and Signaling (4 papers), Cannabis and Cannabinoid Research (4 papers), Neurotransmitter Receptor Influence on Behavior (4 papers), Alcohol Consumption and Health Effects (3 papers), Sphingolipid Metabolism and Signaling (3 papers), Ion channel regulation and function (2 papers) and Neuroscience of respiration and sleep (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (562 citations), Neurology (124 citations), Biological Psychiatry (36 citations), Developmental Neuroscience (57 citations) and Biochemistry (98 citations). Chui‐Se Tham has collaborated with scholars based in United States, Canada and Italy. Frequent co-authors include Michael Webb, H.C. Fibiger, George S. Robertson, G. Damsma, Tadimeti S. Rao, Fen‐Fen Lin, Naichen Yu, Mark A. Klitenick, Lin Luo and Karen Lariosa‐Willingham. Their work appears in journals such as European Journal of Pharmacology, Journal of Pharmacology and Experimental Therapeutics, International Journal of Developmental Neuroscience, Anesthesia & Analgesia and FEBS Letters.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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